Contribution of calcium-dependent facilitation to synaptic plasticity revealed by migraine mutations in the P/Q-type calcium channel.
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Ca2+-dependent modulation of voltage-gated Ca2+ channelsCalmodulin regulation (calmodulation) of voltage-gated calcium channelsTowards a Unified Theory of Calmodulin Regulation (Calmodulation) of Voltage-Gated Calcium and Sodium ChannelsCalcium channels and synaptic transmission in familial hemiplegic migraine type 1 animal models.Activity-dependent calcium, oxygen, and vascular responses in a mouse model of familial hemiplegic migraine type 1.The E1015K variant in the synprint region of the CaV2.1 channel alters channel function and is associated with different migraine phenotypes.Screening of CACNA1A and ATP1A2 genes in hemiplegic migraine: clinical, genetic, and functional studies.In vivo imaging reveals that pregabalin inhibits cortical spreading depression and propagation to subcortical brain structures.Contributions of T-type calcium channel isoforms to neuronal firing.The neuronal splicing factor Nova controls alternative splicing in N-type and P-type CaV2 calcium channels.Calcium binding protein-mediated regulation of voltage-gated calcium channels linked to human diseases.Sense and specificity in neuronal calcium signalling.A rendezvous with the queen of ion channels: Three decades of ion channel research by David T Yue and his Calcium Signals Laboratory.Animal models of monogenic migraine.Pharmacological correction of gating defects in the voltage-gated Ca(v)2.1 Ca²⁺ channel due to a familial hemiplegic migraine mutation.Synaptic gain-of-function effects of mutant Cav2.1 channels in a mouse model of familial hemiplegic migraine are due to increased basal [Ca2+]iAbnormal cortical synaptic transmission in CaV2.1 knockin mice with the S218L missense mutation which causes a severe familial hemiplegic migraine syndrome in humans.Perivascular neurotransmitters: Regulation of cerebral blood flow and role in primary headaches.Large Ca²⁺-dependent facilitation of Ca(V)2.1 channels revealed by Ca²⁺ photo-uncaging.Transcranial direct current stimulation preconditioning modulates the effect of high-frequency repetitive transcranial magnetic stimulation in the human motor cortex.Allosteric regulators selectively prevent Ca-feedback of Ca and Na channelsVoltage-Dependent Calcium Channels, Calcium Binding Proteins, and Their Interaction in the Pathological Process of EpilepsyDrosophila CaV2 channels harboring human migraine mutations cause synapse hyperexcitability that can be suppressed by inhibition of a Ca2+ store release pathway
P2860
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P2860
Contribution of calcium-dependent facilitation to synaptic plasticity revealed by migraine mutations in the P/Q-type calcium channel.
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Contribution of calcium-depend ...... the P/Q-type calcium channel.
@ast
Contribution of calcium-depend ...... the P/Q-type calcium channel.
@en
Contribution of calcium-depend ...... the P/Q-type calcium channel.
@nl
type
label
Contribution of calcium-depend ...... the P/Q-type calcium channel.
@ast
Contribution of calcium-depend ...... the P/Q-type calcium channel.
@en
Contribution of calcium-depend ...... the P/Q-type calcium channel.
@nl
prefLabel
Contribution of calcium-depend ...... the P/Q-type calcium channel.
@ast
Contribution of calcium-depend ...... the P/Q-type calcium channel.
@en
Contribution of calcium-depend ...... the P/Q-type calcium channel.
@nl
P2093
P2860
P356
P1476
Contribution of calcium-depend ...... the P/Q-type calcium channel.
@en
P2093
Brian A MacVicar
Esperanza Garcia
Paul J Adams
Ravi L Rungta
Terrance P Snutch
P2860
P304
18694-18699
P356
10.1073/PNAS.1009500107
P407
P577
2010-10-11T00:00:00Z