Leishmania specific CD4 T cells release IFNγ that limits parasite replication in patients with visceral leishmaniasis.
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The immunology of post-kala-azar dermal leishmaniasis (PKDL)Combined Immune Therapy for the Treatment of Visceral LeishmaniasisSuccessful Treatment of Human Visceral Leishmaniasis Restores Antigen-Specific IFN-γ, but not IL-10 ProductionImmucillins ImmA and ImmH Are Effective and Non-toxic in the Treatment of Experimental Visceral LeishmaniasisVisceral leishmaniasis complicating idiopathic CD4+ T-cell lymphocytopenia: 2 case reports.Alcoholic Fractions F5 and F6 from Withania somnifera Leaves Show a Potent Antileishmanial and Immunomodulatory Activities to Control Experimental Visceral Leishmaniasis.Blood Transcriptional Profiling Reveals Immunological Signatures of Distinct States of Infection of Humans with Leishmania infantum.Transcriptional Profiling in Experimental Visceral Leishmaniasis Reveals a Broad Splenic Inflammatory Environment that Conditions Macrophages toward a Disease-Promoting Phenotype.Leishmania infantum-specific production of IFN-γ and IL-10 in stimulated blood from dogs with clinical leishmaniosis.Optimizing Immunization Strategies for the Induction of Antigen-Specific CD4 and CD8 T Cell Responses for Protection against Intracellular Parasites.Disease severity in patients with visceral leishmaniasis is not altered by co-infection with intestinal parasites.Host resistance to visceral leishmaniasis: prevalence and prevention.A Subset of Neutrophils Expressing Markers of Antigen-Presenting Cells in Human Visceral Leishmaniasis.TNF signalling drives expansion of bone marrow CD4+ T cells responsible for HSC exhaustion in experimental visceral leishmaniasis.Leishmania major large RAB GTPase is highly immunogenic in individuals immune to cutaneous and visceral leishmaniasis.A Non-pathogenic Recombinant Leishmania Expressing Lipophosphoglycan 3 Against Experimental Infection with Leishmania infantum.Tumor necrosis factor alpha neutralization has no direct effect on parasite burden, but causes impaired IFN-γ production by spleen cells from human visceral leishmaniasis patients.Immune Checkpoint Targets for Host-Directed Therapy to Prevent and Treat Leishmaniasis.Combined neutralization of interferon gamma and tumor necrosis factor alpha induces IL-4 production but has no direct additive impact on parasite burden in splenic cultures of human visceral leishmaniasis.Heterologous Immunization With Defined RNA and Subunit Vaccines Enhances T Cell Responses That Protect Against
P2860
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P2860
Leishmania specific CD4 T cells release IFNγ that limits parasite replication in patients with visceral leishmaniasis.
description
2014 nî lūn-bûn
@nan
2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Leishmania specific CD4 T cell ...... s with visceral leishmaniasis.
@ast
Leishmania specific CD4 T cell ...... s with visceral leishmaniasis.
@en
Leishmania specific CD4 T cell ...... s with visceral leishmaniasis.
@nl
type
label
Leishmania specific CD4 T cell ...... s with visceral leishmaniasis.
@ast
Leishmania specific CD4 T cell ...... s with visceral leishmaniasis.
@en
Leishmania specific CD4 T cell ...... s with visceral leishmaniasis.
@nl
prefLabel
Leishmania specific CD4 T cell ...... s with visceral leishmaniasis.
@ast
Leishmania specific CD4 T cell ...... s with visceral leishmaniasis.
@en
Leishmania specific CD4 T cell ...... s with visceral leishmaniasis.
@nl
P2093
P2860
P50
P1476
Leishmania specific CD4 T cell ...... s with visceral leishmaniasis.
@en
P2093
David Sacks
Kamlesh Gidwani
Madhukar Rai
Neetu Singh
Om Prakash Singh
Shalini Gautam
P2860
P356
10.1371/JOURNAL.PNTD.0003198
P407
P577
2014-10-02T00:00:00Z