MiR-194 deregulation contributes to colorectal carcinogenesis via targeting AKT2 pathway.
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MicroRNAs: Clinical Relevance in Colorectal CancerRegulatory Roles of Non-Coding RNAs in Colorectal CancerColorectal Cancer: From the Genetic Model to Posttranscriptional Regulation by Noncoding RNAs.MiR-615 inhibited cell proliferation and cell cycle of human breast cancer cells by suppressing of AKT2 expression.MicroRNA-194 acts as a prognostic marker and inhibits proliferation in hepatocellular carcinoma by targeting MAP4K4.Atypical role of sprouty in colorectal cancer: sprouty repression inhibits epithelial-mesenchymal transition.miR-194 inhibits the proliferation, invasion, migration, and enhances the chemosensitivity of non-small cell lung cancer cells by targeting forkhead box A1 protein.MicroRNA-194 promotes the growth, migration, and invasion of ovarian carcinoma cells by targeting protein tyrosine phosphatase nonreceptor type 12.Melatonin enhances sensitivity to fluorouracil in oesophageal squamous cell carcinoma through inhibition of Erk and Akt pathwayMicroRNAs as potential drug targets for therapeutic intervention in colorectal cancer.Upregulation of nucleostemin in colorectal cancer and its effects on cell malignancyThe roles of microRNAs related with progression and metastasis in human cancers.Targeting IGF1R pathway in cancer with microRNAs: How close are we ?Dysregulation of the miR-194-CUL4B negative feedback loop drives tumorigenesis in non-small-cell lung carcinoma.MiR-194 functions as a tumor suppressor in laryngeal squamous cell carcinoma by targeting Wee1Multidisciplinary efforts driving translational theranostics.MicroRNA-497 inhibits tumor growth through targeting insulin receptor substrate 1 in colorectal cancer.Triangle of AKT2, miRNA, and Tumorigenesis in Different Cancers.Long noncoding RNA H19 contributes to gallbladder cancer cell proliferation by modulated miR-194-5p targeting AKT2.Prognostic value of microRNAs in colorectal cancer: a meta-analysis.The Dual Role of MicroRNAs in Colorectal Cancer ProgressionPM downregulates miR-194-3p and accelerates apoptosis in cigarette-inflamed bronchial epithelium by targeting death-associated protein kinase 1
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MiR-194 deregulation contributes to colorectal carcinogenesis via targeting AKT2 pathway.
description
2014 nî lūn-bûn
@nan
2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
MiR-194 deregulation contributes to colorectal carcinogenesis via targeting AKT2 pathway.
@ast
MiR-194 deregulation contributes to colorectal carcinogenesis via targeting AKT2 pathway.
@en
MiR-194 deregulation contributes to colorectal carcinogenesis via targeting AKT2 pathway.
@nl
type
label
MiR-194 deregulation contributes to colorectal carcinogenesis via targeting AKT2 pathway.
@ast
MiR-194 deregulation contributes to colorectal carcinogenesis via targeting AKT2 pathway.
@en
MiR-194 deregulation contributes to colorectal carcinogenesis via targeting AKT2 pathway.
@nl
prefLabel
MiR-194 deregulation contributes to colorectal carcinogenesis via targeting AKT2 pathway.
@ast
MiR-194 deregulation contributes to colorectal carcinogenesis via targeting AKT2 pathway.
@en
MiR-194 deregulation contributes to colorectal carcinogenesis via targeting AKT2 pathway.
@nl
P2093
P2860
P356
P1433
P1476
MiR-194 deregulation contributes to colorectal carcinogenesis via targeting AKT2 pathway.
@en
P2093
Hui-Jun Zhao
Jing-Yuan Fang
Lin-Lin Ren
Tian-Tian Sun
Ting-Ting Yan
Weiping Zou
Yaou Zhang
P2860
P304
P356
10.7150/THNO.8712
P577
2014-09-19T00:00:00Z