Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease. - Wikidata - wikimedia - TriplyDB

Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

http://www.wikidata.org/entity/Q34287719

about

Chaperone-Mediated Autophagy and Mitochondrial Homeostasis in Parkinson's Disease Reactive oxygen species-activated nanomaterials as theranostic agents New Insights into the Role of Oxidative Stress Mechanisms in the Pathophysiology and Treatment of Multiple Sclerosis Glia Maturation Factor and Mitochondrial Uncoupling Proteins 2 and 4 Expression in the Temporal Cortex of Alzheimer's Disease Brain Promising cannabinoid-based therapies for Parkinson's disease: motor symptoms to neuroprotection Protein 2B of Coxsackievirus B3 Induces Autophagy Relying on Its Transmembrane Hydrophobic Sequences Mitochondrial Retrograde Signaling: Triggers, Pathways, and Outcomes.Time-course and intensity-based classifications of oxidative stresses and their potential application in biomedical, comparative and environmental research.Acetylcholine Attenuates Hypoxia/Reoxygenation Injury by Inducing Mitophagy Through PINK1/Parkin Signal Pathway in H9c2 Cells.Mitochondria as a target for neuroprotection: implications for Alzheimer´s disease.Quantification and Characterization of UVB-Induced Mitochondrial Fragmentation in Normal Primary Human Keratinocytes.NH2-truncated human tau induces deregulated mitophagy in neurons by aberrant recruitment of Parkin and UCHL-1: implications in Alzheimer's disease.Autophagic flux induced by graphene oxide has a neuroprotective effect against human prion protein fragments.Obesity-associated extracellular mtDNA activates central TGFβ pathway to cause blood pressure increase.Piper retrofractum Vahl. Extract, as a PPARδ and AMPK Activator, Suppresses UVB-Induced Photoaging through Mitochondrial Biogenesis and MMPs Inhibition in Human Dermal Fibroblasts and Hairless Mice.Complement regulatory protein CD46 induces autophagy against oxidative stress-mediated apoptosis in normal and asthmatic airway epithelium

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P2860

Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

http://www.wikidata.org/entity/Q34287719

description

2014 nî lūn-bûn

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2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

@ast

Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

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Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

@nl

type

label

Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

@ast

Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

@en

Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

@nl

prefLabel

Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

@ast

Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

@en

Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

@nl

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S12013-014-0006-5

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Cell Biochemistry and Biophysics

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Autophagy of mitochondria: a promising therapeutic target for neurodegenerative disease.

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Anuradha Kalani

http://www.w3.org/2001/XMLSchema#string

Neetu Tyagi

http://www.w3.org/2001/XMLSchema#string

Philip Kyles

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Pradip K Kamat

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Suresh C Tyagi

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P2860

Role of Protein Aggregation in Mitochondrial Dysfunction and Neurodegeneration in Alzheimer's and Parkinson's Diseases

Effects of ALS-related SOD1 mutants on dynein- and KIF5-mediated retrograde and anterograde axonal transport

Parkin is recruited selectively to impaired mitochondria and promotes their autophagy

Loss of PINK1 function promotes mitophagy through effects on oxidative stress and mitochondrial fission

Neuroprotective effect of Nrf2/ARE activators, CDDO ethylamide and CDDO trifluoroethylamide, in a mouse model of amyotrophic lateral sclerosis

Mitochondrial defects in acute multiple sclerosis lesions

Fission and selective fusion govern mitochondrial segregation and elimination by autophagy

Selective degradation of mitochondria by mitophagy

5-aminoimidazole-4-carboxamide-1-beta-4-ribofuranoside (AICAR) attenuates the expression of LPS- and Abeta peptide-induced inflammatory mediators in astroglia.

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neurodegeneration

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