about
HSV-1 ICP27 suppresses NF-kappaB activity by stabilizing IkappaBalphaAn activation domain in the C-terminal subunit of HCF-1 is important for transactivation by VP16 and LZIP.A novel function for human factor C1 (HCF-1), a host protein required for herpes simplex virus infection, in pre-mRNA splicingP-TEFb goes viralHSV-1 tegument protein and the development of its genome editing technologyHSV-1 stimulation-related protein HSRG1 inhibits viral gene transcriptional elongation by interacting with Cyclin T2Flip-Flop HSV-BAC: bacterial artificial chromosome based system for rapid generation of recombinant herpes simplex virus vectors using two independent site-specific recombinases.De novo synthesis of VP16 coordinates the exit from HSV latency in vivo.Distinct effects of knocking down MEK1 and MEK2 on replication of herpes simplex virus type 2.Gamma interferon can block herpes simplex virus type 1 reactivation from latency, even in the presence of late gene expressionHerpes simplex virus type 1 infection leads to loss of serine-2 phosphorylation on the carboxyl-terminal domain of RNA polymerase II.Phosphorylation of transcription factor Sp1 during herpes simplex virus type 1 infectionHerpes simplex virus 1 ICP22 inhibits the transcription of viral gene promoters by binding to and blocking the recruitment of P-TEFbA single amino acid substitution in herpes simplex virus type 1 VP16 inhibits binding to the virion host shutoff protein and is incompatible with virus growth.Herpes simplex virus induces the marked up-regulation of the zinc finger transcriptional factor INSM1, which modulates the expression and localization of the immediate early protein ICP0.L Particles Transmit Viral Proteins from Herpes Simplex Virus 1-Infected Mature Dendritic Cells to Uninfected Bystander Cells, Inducing CD83 DownmodulationGenome-wide analysis of Epstein-Barr virus Rta DNA bindingLatency Entry of Herpes Simplex Virus 1 Is Determined by the Interaction of Its Genome with the Nuclear EnvironmentGenome-wide analysis of G-quadruplexes in herpesvirus genomes.Infection by Herpes Simplex Virus 1 Causes Near-Complete Loss of RNA Polymerase II Occupancy on the Host Cell GenomeThe co-chaperone BAG3 regulates Herpes Simplex Virus replication.The mutated tegument protein UL7 attenuates the virulence of herpes simplex virus 1 by reducing the modulation of α-4 gene transcriptionDuring lytic infection herpes simplex virus type 1 is associated with histones bearing modifications that correlate with active transcription.Role of viral chromatin structure in the regulation of herpes simplex virus 1 gene expression and replication.The ATP-Dependent RNA Helicase DDX3X Modulates Herpes Simplex Virus 1 Gene Expression.P-TEFb goes viral.Replication kinetics of duck enteritis virus UL16 gene in vitro.General and specific alterations in programming of global viral gene expression during infection by VP16 activation-deficient mutants of herpes simplex virus type 1.Therapeutic implications of new insights into the critical role of VP16 in initiating the earliest stages of HSV reactivation from latency.Hepatitis B virus core protein stimulates the proteasome-mediated degradation of viral X protein.VP16-dependent association of chromatin-modifying coactivators and underrepresentation of histones at immediate-early gene promoters during herpes simplex virus infection.Transactivation of a viral target gene by herpes simplex virus ICP27 is posttranscriptional and does not require the endogenous promoter or polyadenylation site.Temporal association of the herpes simplex virus genome with histone proteins during a lytic infection.CDK9 and SPT5 proteins are specifically required for expression of herpes simplex virus 1 replication-dependent late genes.Analysis of herpes simplex virus ICP0 promoter function in sensory neurons during acute infection, establishment of latency, and reactivation in vivo.Chimeric DNA methyltransferases target DNA methylation to specific DNA sequences and repress expression of target genes.Reciprocal activities between herpes simplex virus type 1 regulatory protein ICP0, a ubiquitin E3 ligase, and ubiquitin-specific protease USP7.Human Antiviral Protein IFIX Suppresses Viral Gene Expression during Herpes Simplex Virus 1 (HSV-1) Infection and Is Counteracted by Virus-induced Proteasomal Degradation.Differential cellular requirements for activation of herpes simplex virus type 1 early (tk) and late (gC) promoters by ICP4.Stabilized binding of TBP to the TATA box of herpes simplex virus type 1 early (tk) and late (gC) promoters by TFIIA and ICP4.
P2860
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P2860
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Regulation of herpes simplex virus gene expression.
@ast
Regulation of herpes simplex virus gene expression.
@en
Regulation of herpes simplex virus gene expression.
@nl
type
label
Regulation of herpes simplex virus gene expression.
@ast
Regulation of herpes simplex virus gene expression.
@en
Regulation of herpes simplex virus gene expression.
@nl
prefLabel
Regulation of herpes simplex virus gene expression.
@ast
Regulation of herpes simplex virus gene expression.
@en
Regulation of herpes simplex virus gene expression.
@nl
P1433
P1476
Regulation of herpes simplex virus gene expression.
@en
P2093
P304
P356
10.1016/S0378-1119(01)00512-1
P407
P577
2001-06-01T00:00:00Z