Prenatal hyperandrogenization induces metabolic and endocrine alterations which depend on the levels of testosterone exposure. - Wikidata - wikimedia - TriplyDB

Prenatal hyperandrogenization induces metabolic and endocrine alterations which depend on the levels of testosterone exposure.

Prenatal hyperandrogenization induces metabolic and endocrine alterations which depend on the levels of testosterone exposure.

http://www.wikidata.org/entity/Q34289032

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Early life factors and type 2 diabetes mellitus The Effects of a Single Developmentally Entrained Pulse of Testosterone in Female Neonatal Mice on Reproductive and Metabolic Functions in Adult Life.The time of prenatal androgen exposure affects development of polycystic ovary syndrome-like phenotype in adulthood in female rats Prevalence of Prediabetes Risk in Offspring Born to Mothers with Hyperandrogenism Intergenerational Effects of Endocrine Disorder on Metabolism.Developmental programming: prenatal steroid excess disrupts key members of intraovarian steroidogenic pathway in sheep.Prenatal testosterone exposure worsen the reproductive performance of male rat at adulthood.STMN1 Promotes Progesterone Production Via StAR Up-regulation in Mouse Granulosa Cells.Environmental and developmental origins of ovarian reserve.Expression of the genes for peroxisome proliferator-activated receptor-γ, cyclooxygenase-2, and proinflammatory cytokines in granulosa cells from women with polycystic ovary syndrome.Perinatal testosterone exposure and maternal care effects on the female rat's development and sexual behaviour.Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms.Long-term testosterone treatment during pregnancy does not alter insulin or glucose profile in a sheep model of polycystic ovary syndrome.The impact of prenatal exposure to a single dose of testosterone on insulin resistance, glucose tolerance and lipid profile of female rat's offspring in adulthood.Introducing a rat model of prenatal androgen-induced polycystic ovary syndrome in adulthood.Maternal androgen excess reduces placental and fetal weights, increases placental steroidogenesis, and leads to long-term health effects in their female offspring.Oxidative stress and metabolic markers in pre- and postnatal polycystic ovary syndrome rat protocols.Elevated androstenedione in young adult but not early adolescent prenatally androgenized female rats.Alterations of polyunsaturated fatty acid metabolism in ovarian tissues of polycystic ovary syndrome rats.

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Prenatal hyperandrogenization induces metabolic and endocrine alterations which depend on the levels of testosterone exposure.

http://www.wikidata.org/entity/Q34289032

description

2012 nî lūn-bûn

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2012 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2012年の論文

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2012年論文

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2012年論文

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Prenatal hyperandrogenization ...... vels of testosterone exposure.

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Prenatal hyperandrogenization ...... vels of testosterone exposure.

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Prenatal hyperandrogenization ...... vels of testosterone exposure.

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Adriana Vega Orozco

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Alicia Beatriz Motta

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Omar Pignataro

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Sabrina Amalfi

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Silvana Rocío Ferreira

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Susana Vighi

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P2860

Peroxisome proliferator-activated receptors (PPARs) and ovarian function--implications for regulating steroidogenesis, differentiation, and tissue remodeling.

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Activation of a member of the steroid hormone receptor superfamily by peroxisome proliferators

Anovulation after precocious pubarche: early markers and time course in adolescence

Macrophage polarization: tumor-associated macrophages as a paradigm for polarized M2 mononuclear phagocytes

The origins and sequelae of abnormal neuroendocrine function in polycystic ovary syndrome.

Developmental programming: impact of prenatal testosterone excess and postnatal weight gain on insulin sensitivity index and transfer of traits to offspring of overweight females

Associations of birthweight and gestational age with reproductive and metabolic phenotypes in women with polycystic ovarian syndrome and their first-degree relatives.

Androgen excess fetal programming of female reproduction: a developmental aetiology for polycystic ovary syndrome?

Developmental origin of polycystic ovary syndrome - a hypothesis.

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María Florencia Heber

Leandro Martín Velez

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