Viral particles are required for infection in neurodegenerative Creutzfeldt-Jakob disease.
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PrionsCells infected with scrapie and Creutzfeldt-Jakob disease agents produce intracellular 25-nm virus-like particlesThe kuru infectious agent is a unique geographic isolate distinct from Creutzfeldt-Jakob disease and scrapie agents.Two Creutzfeldt-Jakob disease agents reproduce prion protein-independent identities in cell cultures.The intriguing prion disorders.Agent-specific Shadoo responses in transmissible encephalopathies.Neuroinvasion by a Creutzfeldt-Jakob disease agent in the absence of B cells and follicular dendritic cellsQuantitative recovery of scrapie agent with minimal protein from highly infectious cultures.Unique inflammatory RNA profiles of microglia in Creutzfeldt-Jakob disease.CJD and Scrapie Require Agent-Associated Nucleic Acids for Infection.Virus-like interference in the latency and prevention of Creutzfeldt-Jakob disease.Replication and spread of CJD, kuru and scrapie agents in vivo and in cell cultureHigh CJD infectivity remains after prion protein is destroyed.Scrapie infectivity correlates with converting activity, protease resistance, and aggregation of scrapie-associated prion protein in guanidine denaturation studies.Vaccination with an attenuated Creutzfeldt-Jakob disease strain prevents expression of a virulent agent.Early induction of interferon-responsive mRNAs in Creutzfeldt-Jakob diseaseDiscontools: Identifying gaps in controlling bovine spongiform encephalopathy.Rapid chemical decontamination of infectious CJD and scrapie particles parallels treatments known to disrupt microbes and biofilms.Microglial activation varies in different models of Creutzfeldt-Jakob disease.Follicular dendritic cells and dissemination of Creutzfeldt-Jakob disease.Microglia from Creutzfeldt-Jakob disease-infected brains are infectious and show specific mRNA activation profiles.Retrovirus infection strongly enhances scrapie infectivity release in cell culture.Genesis and variability of [PSI] prion factors in Saccharomyces cerevisiae.Nuclease resistant circular DNAs copurify with infectivity in scrapie and CJD.A Sequence-Dependent DNA Condensation Induced by Prion Protein.A quantitative characterization of interaction between prion protein with nucleic acids.Diet, amyloid enhancing factor (AEF) and amyloidogenesis: An hypothesis
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P2860
Viral particles are required for infection in neurodegenerative Creutzfeldt-Jakob disease.
description
1995 nî lūn-bûn
@nan
1995 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Viral particles are required f ...... ive Creutzfeldt-Jakob disease.
@ast
Viral particles are required f ...... ive Creutzfeldt-Jakob disease.
@en
Viral particles are required f ...... ive Creutzfeldt-Jakob disease.
@nl
type
label
Viral particles are required f ...... ive Creutzfeldt-Jakob disease.
@ast
Viral particles are required f ...... ive Creutzfeldt-Jakob disease.
@en
Viral particles are required f ...... ive Creutzfeldt-Jakob disease.
@nl
prefLabel
Viral particles are required f ...... ive Creutzfeldt-Jakob disease.
@ast
Viral particles are required f ...... ive Creutzfeldt-Jakob disease.
@en
Viral particles are required f ...... ive Creutzfeldt-Jakob disease.
@nl
P2093
P2860
P356
P1476
Viral particles are required for infection in neurodegenerative Creutzfeldt-Jakob disease
@en
P2093
P2860
P304
P356
10.1073/PNAS.92.11.5124
P407
P577
1995-05-01T00:00:00Z