Critical role of an antiviral stress granule containing RIG-I and PKR in viral detection and innate immunity. - Wikidata - wikimedia - TriplyDB

Critical role of an antiviral stress granule containing RIG-I and PKR in viral detection and innate immunity.

Critical role of an antiviral stress granule containing RIG-I and PKR in viral detection and innate immunity.

http://www.wikidata.org/entity/Q34295304

about

A novel function of human Pumilio proteins in cytoplasmic sensing of viral infection Alphavirus Infection: Host Cell Shut-Off and Inhibition of Antiviral Responses Who Regulates Whom? An Overview of RNA Granules and Viral Infections Stress Beyond Translation: Poxviruses and More Enterovirus Control of Translation and RNA Granule Stress Responses Alphaherpesvirus Subversion of Stress-Induced Translational Arrest Regulation of antiviral innate immune signaling by stress-induced RNA granules The Roles of RNase-L in Antimicrobial Immunity and the Cytoskeleton-Associated Innate Response Interplay between Inflammation and Cellular Stress Triggered by Flaviviridae Viruses Modulation of host adaptive immunity by hRSV proteins Diverse Strategies Used by Picornaviruses to Escape Host RNA Decay Pathways Influenza a virus host shutoff disables antiviral stress-induced translation arrest Characterization of a novel mutation in NS1 protein of influenza A virus induced by a chemical substance for the attenuation of pathogenicity RNase L restricts the mobility of engineered retrotransposons in cultured human cells Mumps Virus Induces Protein-Kinase-R-Dependent Stress Granules, Partly Suppressing Type III Interferon Production.Feline Calicivirus Infection Disrupts Assembly of Cytoplasmic Stress Granules and Induces G3BP1 Cleavage Production of a dominant-negative fragment due to G3BP1 cleavage contributes to the disruption of mitochondria-associated protective stress granules during CVB3 infection Encephalomyocarditis virus disrupts stress granules, the critical platform for triggering antiviral innate immune responses.Influenza A induced cellular signal transduction pathways.Innate immune sensing and response to influenza.Andes virus nucleocapsid protein interrupts protein kinase R dimerization to counteract host interference in viral protein synthesis.Activation of stress response pathways promotes formation of antiviral granules and restricts virus replication Induction of stress granules by interferon and down-regulation by the cellular RNA adenosine deaminase ADAR1.The stress granule component TIA-1 binds tick-borne encephalitis virus RNA and is recruited to perinuclear sites of viral replication to inhibit viral translation.Incoming RNA virus nucleocapsids containing a 5'-triphosphorylated genome activate RIG-I and antiviral signaling Innate immunity to influenza virus infection.Quantitative proteomic analysis of the influenza A virus nonstructural proteins NS1 and NS2 during natural cell infection identifies PACT as an NS1 target protein and antiviral host factor.RNase L interacts with Filamin A to regulate actin dynamics and barrier function for viral entry A distinct role of Riplet-mediated K63-Linked polyubiquitination of the RIG-I repressor domain in human antiviral innate immune responses Negative regulation of melanoma differentiation-associated gene 5 (MDA5)-dependent antiviral innate immune responses by Arf-like protein 5B Cell type-specific subcellular localization of phospho-TBK1 in response to cytoplasmic viral DNA The stress granule protein G3BP1 recruits protein kinase R to promote multiple innate immune antiviral responses.DHX36 enhances RIG-I signaling by facilitating PKR-mediated antiviral stress granule formation Hepatitis C Virus. Strategies to Evade Antiviral Responses Stress granules regulate double-stranded RNA-dependent protein kinase activation through a complex containing G3BP1 and Caprin1.Poly(ADP-ribose) polymerase-13 and RNA regulation in immunity and cancer.MDA5-filament, dynamics and disease.Integrative Genomics-Based Discovery of Novel Regulators of the Innate Antiviral Response IFN-β-inducing, unusual viral RNA species produced by paramyxovirus infection accumulated into distinct cytoplasmic structures in an RNA-type-dependent manner Leader-Containing Uncapped Viral Transcript Activates RIG-I in Antiviral Stress Granules

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Critical role of an antiviral stress granule containing RIG-I and PKR in viral detection and innate immunity.

http://www.wikidata.org/entity/Q34295304

description

2012 nî lūn-bûn

@nan

2012 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի օգոստոսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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Critical role of an antiviral ...... detection and innate immunity.

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Critical role of an antiviral ...... detection and innate immunity.

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Critical role of an antiviral ...... detection and innate immunity.

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Critical role of an antiviral ...... detection and innate immunity.

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Critical role of an antiviral ...... detection and innate immunity.

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Critical role of an antiviral ...... detection and innate immunity.

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Critical role of an antiviral ...... detection and innate immunity.

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Critical role of an antiviral ...... detection and innate immunity.

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Critical role of an antiviral ...... detection and innate immunity.

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Atushi Kawaguchi

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Azumi Takemura

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Hideo Namiki

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Ji-Seung Yoo

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Koji Onomoto

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Kyosuke Nagata

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Michihiko Jogi

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Ryo Narita

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Shiho Morimoto

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Suguru Osari

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P2860

Peroxisomes are signaling platforms for antiviral innate immunity

Inhibition of retinoic acid-inducible gene I-mediated induction of beta interferon by the NS1 protein of influenza A virus

Influenza A virus NS1 targets the ubiquitin ligase TRIM25 to evade recognition by the host viral RNA sensor RIG-I

MAVS forms functional prion-like aggregates to activate and propagate antiviral innate immune response

IPS-1, an adaptor triggering RIG-I- and Mda5-mediated type I interferon induction

Cardif is an adaptor protein in the RIG-I antiviral pathway and is targeted by hepatitis C virus

Regulation of innate antiviral defenses through a shared repressor domain in RIG-I and LGP2

Direct triggering of the type I interferon system by virus infection: activation of a transcription factor complex containing IRF-3 and CBP/p300.

Sendai virus trailer RNA binds TIAR, a cellular protein involved in virus-induced apoptosis

Cell proteins TIA-1 and TIAR interact with the 3' stem-loop of the West Nile virus complementary minus-strand RNA and facilitate virus replication.

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8

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Mitsutoshi Yoneyama

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2012-08-13T00:00:00Z

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230716402

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22912779

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3418241

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