MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice
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MicroRNA-98 and let-7 regulate expression of suppressor of cytokine signaling 4 in biliary epithelial cells in response to Cryptosporidium parvum infection.Roles of infection, inflammation, and the immune system in cholesterol gallstone formationPhosphoinositide 3-kinase-dependent inhibition of dendritic cell interleukin-12 production by Giardia lambliaCryptosporidium-malnutrition interactions: mucosal disruption, cytokines, and TLR signaling in a weaned murine model.TLR4 promotes Cryptosporidium parvum clearance in a mouse model of biliary cryptosporidiosis.NF-kappaB p65-dependent transactivation of miRNA genes following Cryptosporidium parvum infection stimulates epithelial cell immune responses.miR-27b targets KSRP to coordinate TLR4-mediated epithelial defense against Cryptosporidium parvum infection.Histone deacetylases and NF-kB signaling coordinate expression of CX3CL1 in epithelial cells in response to microbial challenge by suppressing miR-424 and miR-503.MyD88 signaling is indispensable for primary influenza A virus infection but dispensable for secondary infectionMicroRNA-221 controls expression of intercellular adhesion molecule-1 in epithelial cells in response to Cryptosporidium parvum infection.The cell biology of cryptosporidium infectionInteraction of Cryptosporidium parvum with mouse dendritic cells leads to their activation and parasite transportation to mesenteric lymph nodesCryptosporidiosis-an overview.Experimental and natural infections in MyD88- and IRAK-4-deficient mice and humansThe immunobiology of cholangiocytes.Cryptosporidium parvum induces B7-H1 expression in cholangiocytes by down-regulating microRNA-513.Modulation of dendritic cell responses by parasites: a common strategy to survive.Challenges in understanding the immunopathogenesis of Cryptosporidium infections in humans.Cryptosporidiosis: host immune responses and the prospects for effective immunotherapies.Innate immune responses against Cryptosporidium parvum infection.Apicomplexan infections in the gut.A rapid IL-17 response to Cryptosporidium parvum in the bovine intestine.CD4+ T cells are not essential for control of early acute Cryptosporidium parvum infection in neonatal mice.Trans-suppression of defense DEFB1 gene in intestinal epithelial cells following Cryptosporidium parvum infection is associated with host delivery of parasite Cdg7_FLc_1000 RNA.Nuclear delivery of parasite Cdg2_FLc_0220 RNA transcript to epithelial cells during Cryptosporidium parvum infection modulates host gene transcription.
P2860
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P2860
MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice
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2006 nî lūn-bûn
@nan
2006 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2006年の論文
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2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice
@ast
MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice
@en
MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice
@nl
type
label
MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice
@ast
MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice
@en
MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice
@nl
prefLabel
MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice
@ast
MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice
@en
MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice
@nl
P2093
P2860
P1476
MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice
@en
P2093
A B Rogers
D Golenbock
K A Rogers
P2860
P304
P356
10.1128/IAI.74.1.549-556.2006
P407
P577
2006-01-01T00:00:00Z