Increased expression of fatty-acid and calcium metabolism genes in failing human heart. - Wikidata - wikimedia - TriplyDB

Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

http://www.wikidata.org/entity/Q34305150

about

Metabolic remodeling in moderate synchronous versus dyssynchronous pacing-induced heart failure: integrated metabolomics and proteomics study Diagnostic approaches for diabetic cardiomyopathy.Hypoxia signaling controls postnatal changes in cardiac mitochondrial morphology and function.Endoplasmic reticulum stress induces different molecular structural alterations in human dilated and ischemic cardiomyopathy.Identification of a novel gene for diabetic traits in rats, mice, and humans.Metabolic alterations derived from absence of Two-Pore Channel 1 at cardiac level.Mechanisms of lipotoxicity in the cardiovascular system.The Correlation of PPARα Activity and Cardiomyocyte Metabolism and Structure in Idiopathic Dilated Cardiomyopathy during Heart Failure Progression.Two-pore channels (TPCs): Novel voltage-gated ion channels with pleiotropic functions.Fatty acid metabolism in pulmonary arterial hypertension: role in right ventricular dysfunction and hypertrophy.Lipotoxicity in Obesity: Benefit of Olive Oil.Extracellular vesicles do not contribute to higher circulating levels of soluble LRP1 in idiopathic dilated cardiomyopathy.Tuning the ion selectivity of two-pore channels.Mitochondrial remodeling in mice with cardiomyocyte-specific lipid overload.Endolysosomal two-pore channels regulate autophagy in cardiomyocytes.Mitophagy and Mitochondrial Quality Control Mechanisms in the Heart.Astragaloside IV inhibits ventricular remodeling and improves fatty acid utilization in rats with chronic heart failure.Two-pore channels and disease.24 h nesfatin-1 treatment promotes apoptosis in cardiomyocytes.

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Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

http://www.wikidata.org/entity/Q34305150

description

2012 nî lūn-bûn

@nan

2012 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2012 թվականի հունիսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

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Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

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Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

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type

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Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

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Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

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Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

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prefLabel

Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

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Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

@en

Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

@nl

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JOURNAL.PONE.0037505

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Increased expression of fatty-acid and calcium metabolism genes in failing human heart.

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Ana Mosquera-Leal

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Diego Rodríguez-Penas

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Francisca Lago

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John Parrington

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Josep Brugada

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José Ramón González-Juanatey

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Manuel Calaza

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Manuel Francisco Otero

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Manuel Portolés

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María Álvarez-Barredo

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P2860

NAADP mobilizes calcium from acidic organelles through two-pore channels

Increased InsP3Rs in the junctional sarcoplasmic reticulum augment Ca2+ transients and arrhythmias associated with cardiac hypertrophy

Peroxisome proliferator-activated receptors: nuclear control of metabolism

In obese Zucker rats, lipids accumulate in the heart despite normal mitochondrial content, morphology and long-chain fatty acid oxidation

The two-pore channel TPCN2 mediates NAADP-dependent Ca(2+)-release from lysosomal stores

Dual promoter structure of mouse and human fatty acid translocase/CD36 genes and unique transcriptional activation by peroxisome proliferator-activated receptor alpha and gamma ligands.

Fatty acid transporters in plasma membranes of cardiomyocytes in patients with dilated cardiomyopathy.

Influence of heart failure on nucleocytoplasmic transport in human cardiomyocytes.

Rescue of cardiomyopathy in peroxisome proliferator-activated receptor-alpha transgenic mice by deletion of lipoprotein lipase identifies sources of cardiac lipids and peroxisome proliferator-activated receptor-alpha activators

Altered intracellular Ca2+ handling in heart failure.

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