Initiation of neuronal damage by complex I deficiency and oxidative stress in Parkinson's disease.
about
Role and regulation of ferritin H in rotenone-mediated mitochondrial oxidative stressA constraint-based modelling approach to metabolic dysfunction in Parkinson's diseaseLocal melatoninergic system as the protector of skin integrityMechanisms of altered redox regulation in neurodegenerative diseases--focus on S--glutathionylationReactive oxygen species in inflammation and tissue injuryAge-related decrease in the mitochondrial sirtuin deacetylase Sirt3 expression associated with ROS accumulation in the auditory cortex of the mimetic aging rat model.Structural alterations induced by ten disease-causing mutations of human dihydrolipoamide dehydrogenase analyzed by hydrogen/deuterium-exchange mass spectrometry: Implications for the structural basis of E3 deficiency.Impact of repeated stress on traumatic brain injury-induced mitochondrial electron transport chain expression and behavioral responses in rats.Rapid purification and mass spectrometric characterization of mitochondrial NADH dehydrogenase (Complex I) from rodent brain and a dopaminergic neuronal cell line.Activation of Retinoid X Receptor increases dopamine cell survival in models for Parkinson's disease.Activation of AMPK and inactivation of Akt result in suppression of mTOR-mediated S6K1 and 4E-BP1 pathways leading to neuronal cell death in in vitro models of Parkinson's diseaseThe CREB/CRE transcriptional pathway: protection against oxidative stress-mediated neuronal cell deathDifferentiation of single cell derived human mesenchymal stem cells into cells with a neuronal phenotype: RNA and microRNA expression profile.Reconvene and reconnect the antioxidant hypothesis in human health and disease.Rotenone induction of hydrogen peroxide inhibits mTOR-mediated S6K1 and 4E-BP1/eIF4E pathways, leading to neuronal apoptosis.Activation of TNFR1 ectodomain shedding by mitochondrial Ca2+ determines the severity of inflammation in mouse lung microvessels.Impact of methamphetamine on dopamine neurons in primates is dependent on age: implications for development of Parkinson's disease.Systematic analysis of transcription-level effects of neurodegenerative diseases on human brain metabolism by a newly reconstructed brain-specific metabolic networkNitrite augments tolerance to ischemia/reperfusion injury via the modulation of mitochondrial electron transfer.Calcium homeostasis, selective vulnerability and Parkinson's diseaseCrosstalk between Ca2+ signaling and mitochondrial H2O2 is required for rotenone inhibition of mTOR signaling pathway leading to neuronal apoptosis.Mitochondrial toxins in Basal Ganglia disorders: from animal models to therapeutic strategies.Molecular mechanisms and clinical implications of reversible protein S-glutathionylation.A novel derivative of the natural agent deguelin for cancer chemoprevention and therapy.Calcium, cellular aging, and selective neuronal vulnerability in Parkinson's disease.Rifampicin and Parkinson's disease.Oxidative stress and metabolic syndrome: cause or consequence of Alzheimer's disease?Does PGC1α/FNDC5/BDNF Elicit the Beneficial Effects of Exercise on Neurodegenerative Disorders?Redox Signaling Mediated by Thioredoxin and Glutathione Systems in the Central Nervous System.Mechanisms of MPP⁺-induced PC12 cell apoptosis via reactive oxygen species.Preparation, characterization and efficacy evaluation of synthetic biocompatible polymers linking natural antioxidants.Differential impact of environmental stresses on the pea mitochondrial proteome.mt-Nd2a suppresses reactive oxygen species production by mitochondrial complexes I and III.Paraquat-induced apoptosis in human neuroblastoma SH-SY5Y cells: involvement of p53 and mitochondria.Potentiation of in vivo neuroprotection by BclX(L) and GDNF co-expression depends on post-lesion time in deafferentiated CNS neurons.Induction of reactive oxygen species by bisphenol A and abrogation of bisphenol A-induced cell injury by DJ-1.Genetic and environmental factors influencing glutathione homeostasis in Saccharomyces cerevisiae.Mitochondrial impairment and melatonin protection in parkinsonian mice do not depend of inducible or neuronal nitric oxide synthasesRedox-optimized ROS balance: a unifying hypothesisKinetic Modeling of the Mitochondrial Energy Metabolism of Neuronal Cells: The Impact of Reduced α-Ketoglutarate Dehydrogenase Activities on ATP Production and Generation of Reactive Oxygen Species.
P2860
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P2860
Initiation of neuronal damage by complex I deficiency and oxidative stress in Parkinson's disease.
description
2004 nî lūn-bûn
@nan
2004 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի մարտին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Initiation of neuronal damage ...... stress in Parkinson's disease.
@ast
Initiation of neuronal damage ...... stress in Parkinson's disease.
@en
Initiation of neuronal damage ...... stress in Parkinson's disease.
@nl
type
label
Initiation of neuronal damage ...... stress in Parkinson's disease.
@ast
Initiation of neuronal damage ...... stress in Parkinson's disease.
@en
Initiation of neuronal damage ...... stress in Parkinson's disease.
@nl
prefLabel
Initiation of neuronal damage ...... stress in Parkinson's disease.
@ast
Initiation of neuronal damage ...... stress in Parkinson's disease.
@en
Initiation of neuronal damage ...... stress in Parkinson's disease.
@nl
P1476
Initiation of neuronal damage ...... stress in Parkinson's disease
@en
P2093
Ildiko Sipos
Laszlo Tretter
P304
P356
10.1023/B:NERE.0000014827.94562.4B
P577
2004-03-01T00:00:00Z
P6179
1044554912