DNase Sda1 allows invasive M1T1 Group A Streptococcus to prevent TLR9-dependent recognition
about
Inflammasome/IL-1β Responses to Streptococcal PathogensVibrio cholerae evades neutrophil extracellular traps by the activity of two extracellular nucleasesAn emm5 Group A Streptococcal Outbreak Among Workers in a Factory Manufacturing Telephone Accessories.Increased neutrophil extracellular trap-mediated Staphylococcus aureus clearance through inhibition of nuclease activity by clindamycin and immunoglobulinDisease manifestations and pathogenic mechanisms of Group A Streptococcus.Determining antibody-binding site of streptococcal pyrogenic exotoxin B to protect mice from group a streptococcus infectionTranscriptional regulation of a bacteriophage encoded extracellular DNase (Spd-3) by Rgg in Streptococcus pyogenes.Peroxide responsive regulator PerR of group A Streptococcus is required for the expression of phage-associated DNase Sda1 under oxidative stress.Mechanisms of group A Streptococcus resistance to reactive oxygen species.Targeted Curing of All Lysogenic Bacteriophage from Streptococcus pyogenes Using a Novel Counter-selection TechniqueContribution of nuclease to the pathogenesis of Aeromonas hydrophila.Tamoxifen augments the innate immune function of neutrophils through modulation of intracellular ceramide.Structural characterization of the virulence factor Sda1 nuclease from Streptococcus pyogenes.Molecular mechanisms regulating NETosis in infection and disease.Rapidly fatal hemorrhagic pneumonia and group A Streptococcus serotype M1.Streptococcus suis Serotype 2 Biofilms Inhibit the Formation of Neutrophil Extracellular TrapsSubterfuge and sabotage: evasion of host innate defenses by invasive gram-positive bacterial pathogens.Interferon α-Enhanced Clearance of Group A Streptococcus Despite Neutropenia.Genomic Comparison among Lethal Invasive Strains of Streptococcus pyogenes Serotype M1.Group A Streptococcus encounters with host macrophages.Nuclease A (Gbs0661), an extracellular nuclease of Streptococcus agalactiae, attacks the neutrophil extracellular traps and is needed for full virulence.Human IgG Increases Virulence of Streptococcus pyogenes through Complement Evasion.A novel extracellular vesicle-associated endodeoxyribonuclease helps Streptococcus pneumoniae evade neutrophil extracellular traps and is required for full virulence.
P2860
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P2860
DNase Sda1 allows invasive M1T1 Group A Streptococcus to prevent TLR9-dependent recognition
description
2012 nî lūn-bûn
@nan
2012 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
DNase Sda1 allows invasive M1T ...... ent TLR9-dependent recognition
@ast
DNase Sda1 allows invasive M1T ...... ent TLR9-dependent recognition
@en
DNase Sda1 allows invasive M1T ...... ent TLR9-dependent recognition
@nl
type
label
DNase Sda1 allows invasive M1T ...... ent TLR9-dependent recognition
@ast
DNase Sda1 allows invasive M1T ...... ent TLR9-dependent recognition
@en
DNase Sda1 allows invasive M1T ...... ent TLR9-dependent recognition
@nl
prefLabel
DNase Sda1 allows invasive M1T ...... ent TLR9-dependent recognition
@ast
DNase Sda1 allows invasive M1T ...... ent TLR9-dependent recognition
@en
DNase Sda1 allows invasive M1T ...... ent TLR9-dependent recognition
@nl
P2860
P50
P1433
P1476
DNase Sda1 allows invasive M1T ...... ent TLR9-dependent recognition
@en
P2093
Federica Andreoni
Satoshi Uchiyama
P2860
P304
P356
10.1371/JOURNAL.PPAT.1002736
P577
2012-06-14T00:00:00Z