Uterine epithelial estrogen receptor α is dispensable for proliferation but essential for complete biological and biochemical responses. - Wikidata - wikimedia - TriplyDB

Uterine epithelial estrogen receptor α is dispensable for proliferation but essential for complete biological and biochemical responses.

Uterine epithelial estrogen receptor α is dispensable for proliferation but essential for complete biological and biochemical responses.

http://www.wikidata.org/entity/Q34320366

about

Mechanisms of uterine estrogen signaling during early pregnancy in mice: an update What's new in estrogen receptor action in the female reproductive tract Estrogen receptor β: the guardian of the endometrium Estrogen receptor signaling during vertebrate development ARID1A Is Essential for Endometrial Function during Early Pregnancy STAT3 regulates uterine epithelial remodeling and epithelial-stromal crosstalk during implantation Reconstruction of endometrium from human endometrial side population cell lines.The role of genetics in estrogen responses: a critical piece of an intricate puzzle.Crucial role of estrogen for the mammalian female in regulating semen coagulation and liquefaction in vivo.Minireview: Tipping the balance: ligand-independent activation of steroid receptors.Membrane-localized estrogen receptor α is required for normal organ development and function.Progesterone: the ultimate endometrial tumor suppressor.Minireview: Steroid-regulated paracrine mechanisms controlling implantation Cadmium-induced effects on cellular signaling pathways in the liver of transgenic estrogen reporter mice.Physiological and molecular determinants of embryo implantation Progesterone action in endometrial cancer, endometriosis, uterine fibroids, and breast cancer.Stromal-to-epithelial transition during postpartum endometrial regeneration.Grhl2 determines the epithelial phenotype of breast cancers and promotes tumor progression.Activation of protein synthesis in mouse uterine epithelial cells by estradiol-17β is mediated by a PKC-ERK1/2-mTOR signaling pathway Activated mutant p110α causes endometrial carcinoma in the setting of biallelic Pten deletion.Development of phenotypic and transcriptional biomarkers to evaluate relative activity of potentially estrogenic chemicals in ovariectomized mice Uterine gland formation in mice is a continuous process, requiring the ovary after puberty, but not after parturition.Uterine epithelial cell estrogen receptor alpha-dependent and -independent genomic profiles that underlie estrogen responses in mice.The p160/steroid receptor coactivator family: potent arbiters of uterine physiology and dysfunction Influence of AKT on progesterone action in endometrial diseases.The regulation of embryo implantation and endometrial decidualization by progesterone receptor signaling.Kruppel-like factor 5 (KLF5) is critical for conferring uterine receptivity to implantation.Epithelial progesterone receptor exhibits pleiotropic roles in uterine development and function.KLF15 negatively regulates estrogen-induced epithelial cell proliferation by inhibition of DNA replication licensing Uterine Epithelial Estrogen Receptor-α Controls Decidualization via a Paracrine Mechanism.Endometrial tumorigenesis in Pten(+/-) mice is independent of coexistence of estrogen and estrogen receptor α.Epithelial estrogen receptor 1 intrinsically mediates squamous differentiation in the mouse vagina.The progesterone receptor regulates implantation, decidualization, and glandular development via a complex paracrine signaling network.Endometrial stromal beta-catenin is required for steroid-dependent mesenchymal-epithelial cross talk and decidualization Krüppel-Like Factor 13 Deficiency in Uterine Endometrial Cells Contributes to Defective Steroid Hormone Receptor Signaling but Not Lesion Establishment in a Mouse Model of Endometriosis Nuclear receptor coactivator-6 attenuates uterine estrogen sensitivity to permit embryo implantation.Neonatal diethylstilbestrol exposure alters the metabolic profile of uterine epithelial cells.Deletion of Lysophosphatidic Acid Receptor 3 (Lpar3) Disrupts Fine Local Balance of Progesterone and Estrogen Signaling in Mouse Uterus During Implantation.Oviductal estrogen receptor α signaling prevents protease-mediated embryo death Requirement for stromal estrogen receptor alpha in cervical neoplasia

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Uterine epithelial estrogen receptor α is dispensable for proliferation but essential for complete biological and biochemical responses.

http://www.wikidata.org/entity/Q34320366

description

2010 nî lūn-bûn

@nan

2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Uterine epithelial estrogen re ...... cal and biochemical responses.

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Uterine epithelial estrogen re ...... cal and biochemical responses.

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Uterine epithelial estrogen re ...... cal and biochemical responses.

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Uterine epithelial estrogen re ...... cal and biochemical responses.

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Uterine epithelial estrogen re ...... cal and biochemical responses.

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Uterine epithelial estrogen re ...... cal and biochemical responses.

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Uterine epithelial estrogen re ...... cal and biochemical responses.

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Uterine epithelial estrogen re ...... cal and biochemical responses.

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Uterine epithelial estrogen re ...... cal and biochemical responses.

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PNAS.1013226107

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Uterine epithelial estrogen re ...... cal and biochemical responses.

@en

P2093

Grant D Orvis

http://www.w3.org/2001/XMLSchema#string

Richard R Behringer

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P2860

Estrogen receptor alpha rapidly activates the IGF-1 receptor pathway

X-linked IAP is a direct inhibitor of cell-death proteases

Blastocyst implantation depends on maternal expression of leukaemia inhibitory factor

Effect of single and compound knockouts of estrogen receptors alpha (ERalpha) and beta (ERbeta) on mouse reproductive phenotypes

Estrogen suppresses uterine epithelial apoptosis by inducing birc1 expression

Estrogen Receptor Null Mice: What Have We Learned and Where Will They Lead Us?

Apoptosis: controlled demolition at the cellular level

Apoptotic action of estrogen.

Apoptosis at the time of embryo implantation in mouse and rat.

Lack of CCAAT enhancer binding protein beta (C/EBPbeta) in uterine epithelial cells impairs estrogen-induced DNA replication, induces DNA damage response pathways, and promotes apoptosis.

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19272-19277

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10.1073/PNAS.1013226107

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45

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107

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Kenneth S. Korach

Wipawee Winuthayanon

Sylvia C Hewitt

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2010-10-25T00:00:00Z

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47544359

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