Matrix metalloproteinase inhibition after myocardial infarction: a new approach to prevent heart failure?
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Elevated levels of matrix metalloprotein-3 in patients with coronary aneurysm: A case control studyTranslating Koch's postulates to identify matrix metalloproteinase roles in postmyocardial infarction remodeling: cardiac metalloproteinase actions (CarMA) postulatesGH and the cardiovascular system: an update on a topic at heartHMGB1 attenuates cardiac remodelling in the failing heart via enhanced cardiac regeneration and miR-206-mediated inhibition of TIMP-3Physiological Implications of Myocardial Scar StructureDonepezil, anti-Alzheimer's disease drug, prevents cardiac rupture during acute phase of myocardial infarction in miceIncreased systemic and myocardial expression of neutrophil gelatinase-associated lipocalin in clinical and experimental heart failureComputational modeling of cardiac fibroblasts and fibrosisMyocardial dysfunction in the periinfarct and remote regions following anterior infarction in rats quantified by 2D radial strain echocardiography: an observational cohort studyIntramyocardial fibroblast myocyte communication.Adenovirus-mediated stromal cell-derived factor-1 alpha gene transfer improves cardiac structure and function after experimental myocardial infarction through angiogenic and antifibrotic actionsPeriostin in Mature Stage Localized Scleroderma.Effects of novel semiselective matrix metalloproteinase inhibitors on ex vivo cardiac structure-function.Zinc-binding groups modulate selective inhibition of MMPs.Expanded network of inflammatory markers of atherogenesis: where are we now?Thoracic aortic dissection: are matrix metalloproteinases involved?AP-1 (Fra-1/c-Jun)-mediated induction of expression of matrix metalloproteinase-2 is required for 15S-hydroxyeicosatetraenoic acid-induced angiogenesis.Caspase inhibition modulates left ventricular remodeling following myocardial infarction through cellular and extracellular mechanisms.Coexistence of cardiac troponin T variants reduces heart efficiency.Study of the correlations among some parameters of the oxidative status, gelatinases, and their inhibitors in a group of subjects with metabolic syndrome.Long-term localized high-frequency electric stimulation within the myocardial infarct: effects on matrix metalloproteinases and regional remodelingSpatiotemporal induction of matrix metalloproteinase-9 transcription after discrete myocardial injury.Discordant activation of gene promoters for matrix metalloproteinases and tissue inhibitors of the metalloproteinases following myocardial infarction.Regulation of MMP-9 by a WIN-binding site in the monocyte-macrophage system independent from cannabinoid receptors.Myocardial remodelling and matrix metalloproteinases in heart failure: turmoil within the interstitium.Toll-like receptor 9 prevents cardiac rupture after myocardial infarction in mice independently of inflammation.Genetic variants associated with circulating MMP1 levels near matrix metalloproteinase genes on chromosome 11q21-22 in Taiwanese: interaction with obesity.Novel association between plasma matrix metalloproteinase-9 and risk of incident atrial fibrillation in a case-cohort study: the Atherosclerosis Risk in Communities studyPreventive effect of yuzu and hesperidin on left ventricular remodeling and dysfunction in rat permanent left anterior descending coronary artery occlusion model.Endogenous matrix metalloproteinases 2 and 9 regulate activation of CD4+ and CD8+ T cells.Expression levels of matrix metalloproteinase-9 in human gastric carcinoma.Ataxia telangiectasia mutated kinase plays a protective role in β-adrenergic receptor-stimulated cardiac myocyte apoptosis and myocardial remodeling.Deficiency of ataxia telangiectasia mutated kinase modulates cardiac remodeling following myocardial infarction: involvement in fibrosis and apoptosis.Increased expression of plasminogen activator inhibitor-1 in cardiomyocytes contributes to cardiac fibrosis after myocardial infarction.Predictive utility of the changes in matrix metalloproteinase-2 in the early phase for left ventricular reverse remodeling after an acute myocardial infarction.β-Adrenergic Receptor-Stimulated Cardiac Myocyte Apoptosis: Role of β1 Integrins.Targeted deletion of CC chemokine receptor 2 attenuates left ventricular remodeling after experimental myocardial infarction.Impact of ischemia-reperfusion on extracellular matrix processing and structure of the basement membrane of the heartDiabetes influences cardiac extracellular matrix remodelling after myocardial infarction and subsequent development of cardiac dysfunction.Association between plasma ADAMTS-7 levels and ventricular remodeling in patients with acute myocardial infarction.
P2860
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P2860
Matrix metalloproteinase inhibition after myocardial infarction: a new approach to prevent heart failure?
description
2001 nî lūn-bûn
@nan
2001 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Matrix metalloproteinase inhib ...... oach to prevent heart failure?
@ast
Matrix metalloproteinase inhib ...... oach to prevent heart failure?
@en
Matrix metalloproteinase inhib ...... oach to prevent heart failure?
@nl
type
label
Matrix metalloproteinase inhib ...... oach to prevent heart failure?
@ast
Matrix metalloproteinase inhib ...... oach to prevent heart failure?
@en
Matrix metalloproteinase inhib ...... oach to prevent heart failure?
@nl
prefLabel
Matrix metalloproteinase inhib ...... oach to prevent heart failure?
@ast
Matrix metalloproteinase inhib ...... oach to prevent heart failure?
@en
Matrix metalloproteinase inhib ...... oach to prevent heart failure?
@nl
P2093
P356
P1433
P1476
Matrix metalloproteinase inhib ...... oach to prevent heart failure?
@en
P2093
Cleutjens JP
Creemers EE
P304
P356
10.1161/HH1501.094396
P577
2001-08-01T00:00:00Z