Peripheral tolerance to allogeneic class II histocompatibility antigens expressed in transgenic mice: evidence against a clonal-deletion mechanism
about
Organ injury associated with extrathymic induction of immune tolerance in doubly transgenic mice.Peripheral tolerance in mice expressing a liver-specific class I molecule: inactivation/deletion of a T-cell subpopulationDeterminants of the B-cell response against a transgenic autoantigenT-cell receptor V beta expression in normal human skinMechanism of self-tolerance of gamma/delta T cells in epithelial tissue.An evaluation of the potential to use tumor-associated antigens as targets for antitumor T cell therapy using transgenic mice expressing a retroviral tumor antigen in normal lymphoid tissues.Propagation of dendritic cell progenitors from normal mouse liver using granulocyte/macrophage colony-stimulating factor and their maturational development in the presence of type-1 collagenHeterogeneity of single cell cytokine gene expression in clonal T cell populations.Endogenous myelin basic protein inactivates the high avidity T cell repertoireICOS costimulation expands Th2 immunity by augmenting migration of lymphocytes to draining lymph nodesT-cell responsiveness to an oncogenic peripheral protein and spontaneous autoimmunity in transgenic mice.Cellular and peptide requirements for in vitro clonal deletion of immature thymocytes.Expression of myosin-class II major histocompatibility complexes in the normal myocardium occurs before induction of autoimmune myocarditis.Insulin dependent diabetes mellitus hypothesis of autoimmunity.Autoantibody production in hepatitis B e antigen transgenic mice elicited with a self T-cell peptide and inhibited with nonself peptides.A nondeletional mechanism of peripheral tolerance in T-cell receptor transgenic mice.Balancing immunity and tolerance: deleting and tuning lymphocyte repertoires.Selective targeting of the immune response in autoimmune demyelination.The thymus and T-cell tolerance.Developmental regulation of thymocyte susceptibility to deletion by "self"-peptide.T cell contact with Ia antigens on nonhemopoietic cells in vivo can lead to immunity rather than tolerance.Nonmitogenic anti-CD3 monoclonal antibodies deliver a partial T cell receptor signal and induce clonal anergyHeterogeneity in the clonal T cell response. Implications for models of T cell activation and cytokine phenotype development.
P2860
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P2860
Peripheral tolerance to allogeneic class II histocompatibility antigens expressed in transgenic mice: evidence against a clonal-deletion mechanism
description
1989 nî lūn-bûn
@nan
1989 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Peripheral tolerance to alloge ...... st a clonal-deletion mechanism
@ast
Peripheral tolerance to alloge ...... st a clonal-deletion mechanism
@en
Peripheral tolerance to alloge ...... st a clonal-deletion mechanism
@nl
type
label
Peripheral tolerance to alloge ...... st a clonal-deletion mechanism
@ast
Peripheral tolerance to alloge ...... st a clonal-deletion mechanism
@en
Peripheral tolerance to alloge ...... st a clonal-deletion mechanism
@nl
prefLabel
Peripheral tolerance to alloge ...... st a clonal-deletion mechanism
@ast
Peripheral tolerance to alloge ...... st a clonal-deletion mechanism
@en
Peripheral tolerance to alloge ...... st a clonal-deletion mechanism
@nl
P2093
P2860
P356
P1476
Peripheral tolerance to alloge ...... st a clonal-deletion mechanism
@en
P2093
P2860
P304
10034-10038
P356
10.1073/PNAS.86.24.10034
P407
P577
1989-12-01T00:00:00Z