NANOG-dependent function of TET1 and TET2 in establishment of pluripotency. - Wikidata - wikimedia - TriplyDB

NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

http://www.wikidata.org/entity/Q34327015

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Overexpression of Cancer-Associated Genes via Epigenetic Derepression Mechanisms in Gynecologic Cancer Modifiers and Readers of DNA Modifications and Their Impact on Genome Structure, Expression, and Stability in Disease Induced Pluripotent Stem Cells: Generation Strategy and Epigenetic Mystery behind Reprogramming Hypoxia, Epithelial-Mesenchymal Transition, and TET-Mediated Epigenetic Changes Potential Gene Interactions in the Cell Cycles of Gametes, Zygotes, Embryonic Stem Cells and the Development of Cancer Insights into Chemoresistance of Prostate Cancer Induction of pluripotency by defined factors Cytosine modifications in neurodevelopment and diseases Unlocking epigenetic codes in neurogenesis Mechanism and function of oxidative reversal of DNA and RNA methylation The extended pluripotency protein interactome and its links to reprogramming Chromatin dynamics during cellular reprogramming The role of pluripotency gene regulatory network components in mediating transitions between pluripotent cell states Reprogramming of cell fate: epigenetic memory and the erasure of memories past Role of TET enzymes in DNA methylation, development, and cancer Insights into the Nanog gene: A propeller for stemness in primitive stem cells DNA methylation and hydroxymethylation in stem cells Concise Review: NANOG in Cancer Stem Cells and Tumor Development: An Update and Outstanding Questions WT1 recruits TET2 to regulate its target gene expression and suppress leukemia cell proliferation A direct physical interaction between Nanog and Sox2 regulates embryonic stem cell self-renewal Toward pluripotency by reprogramming: mechanisms and application Different reprogramming propensities in plants and mammals: Are small variations in the core network wirings responsible?Zeb2 Regulates Cell Fate at the Exit from Epiblast State in Mouse Embryonic Stem Cells Potential functional roles of DNA demethylation intermediates Context-Dependent Functions of NANOG Phosphorylation in Pluripotency and Reprogramming.TET enzymes, TDG and the dynamics of DNA demethylation.Reprogramming the methylome: erasing memory and creating diversity.Molecular control of induced pluripotency.Roles of small molecules in somatic cell reprogramming A genome-wide RNAi screen identifies opposing functions of Snai1 and Snai2 on the Nanog dependency in reprogramming.Mechanisms and models of somatic cell reprogramming.NANOG amplifies STAT3 activation and they synergistically induce the naive pluripotent program.Predicting the human epigenome from DNA motifs.The combination of Tet1 with Oct4 generates high-quality mouse-induced pluripotent stem cells.TET1 regulates hypoxia-induced epithelial-mesenchymal transition by acting as a co-activator.Molecules and mechanisms controlling the active DNA demethylation of the mammalian zygotic genome.Effects of Tet-mediated oxidation products of 5-methylcytosine on DNA transcription in vitro and in mammalian cells.CRL4(VprBP) E3 ligase promotes monoubiquitylation and chromatin binding of TET dioxygenases.Epigenetic mechanisms of induced pluripotency.Dynamic switching of active promoter and enhancer domains regulates Tet1 and Tet2 expression during cell state transitions between pluripotency and differentiation.

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P2860

NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

http://www.wikidata.org/entity/Q34327015

description

2013 nî lūn-bûn

@nan

2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի փետրվարին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

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2013年论文

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name

NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

@ast

NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

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NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

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type

label

NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

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NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

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NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

@nl

prefLabel

NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

@ast

NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

@en

NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

@nl

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NANOG-dependent function of TET1 and TET2 in establishment of pluripotency.

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Arven Saunders

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Dana N Levasseur

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Jianlong Wang

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Junjun Ding

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Mingjiang Xu

http://www.w3.org/2001/XMLSchema#string

Pavel V Shliaha

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Sabine Dietmann

http://www.w3.org/2001/XMLSchema#string

Satyabrata Das

http://www.w3.org/2001/XMLSchema#string

Zhe Li

http://www.w3.org/2001/XMLSchema#string

P2860

Conversion of 5-methylcytosine to 5-hydroxymethylcytosine in mammalian DNA by MLL partner TET1

Direct cell reprogramming is a stochastic process amenable to acceleration

Induction of pluripotent stem cells from mouse embryonic and adult fibroblast cultures by defined factors

The homeoprotein Nanog is required for maintenance of pluripotency in mouse epiblast and ES cells

Functional expression cloning of Nanog, a pluripotency sustaining factor in embryonic stem cells

A protein interaction network for pluripotency of embryonic stem cells

Promotion of reprogramming to ground state pluripotency by signal inhibition

Role of Tet proteins in 5mC to 5hmC conversion, ES-cell self-renewal and inner cell mass specification

TET1 and hydroxymethylcytosine in transcription and DNA methylation fidelity

Integration of external signaling pathways with the core transcriptional network in embryonic stem cells

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370-374

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scholarly article

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10.1038/NATURE11925

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7441

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495

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Timothy Alexander Hore

Thorold Theunissen

Francesco Faiola

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2013-02-10T00:00:00Z

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235441402

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1034824135

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23395962

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3606645

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