Functional analysis of a retroviral host-range mutant: altered long terminal repeat sequences allow expression in embryonal carcinoma cells.
about
Nuclear and nucleolar targeting of human ribosomal protein S6Transplantable myeloproliferative disease induced in mice by an interleukin 6 retrovirus.Influence of vector design and host cell on the mechanism of recombination and emergence of mutant subpopulations of replicating retroviral vectors.Embryonic stem cell virus, a recombinant murine retrovirus with expression in embryonic stem cellsRegulation of RNA polymerase III transcription in response to Simian virus 40 transformationConstruction of retroviral vectors with improved safety, gene expression, and versatility.Silencing of gene expression: implications for design of retrovirus vectors.Regulation and patterns of endogenous and exogenous gene expression during differentiation of embryonal carcinoma cells.Strategies to insulate lentiviral vector-expressed transgenesThe Moloney murine leukemia virus repressor binding site represses expression in murine and human hematopoietic stem cellsMultiple modifications in cis elements of the long terminal repeat of retroviral vectors lead to increased expression and decreased DNA methylation in embryonic carcinoma cells.Functional heterogeneity of a large family of human LTR-like promoters and enhancers.Novel retroviral vectors for efficient expression of the multidrug resistance (mdr-1) gene in early hematopoietic cells.Abrogation of the requirement for feeder cell interaction and T cell receptor stimulation of lymphocytes infected with retroviral vectors.Primer binding site-dependent restriction of murine leukemia virus requires HP1 binding by TRIM28.Increased probability of expression from modified retroviral vectors in embryonal stem cells and embryonal carcinoma cells.An internal deletion enhances the transcriptional activity of a recombinant retrovirus in hematopoietic cells in vivo.Derivatives of Moloney murine sarcoma virus capable of being transcribed in embryonal carcinoma stem cells have gained a functional Sp1 binding site.Host restriction factors blocking retroviral replicationSequences and factors required for the F9 embryonal carcinoma stem cell E1a-like activity.Distinct segments within the enhancer region collaborate to specify the type of leukemia induced by nondefective Friend and Moloney virusesDevelopment of a retroviral vector for inducible expression of transforming growth factor beta 1.Two blocks in Moloney murine leukemia virus expression in undifferentiated F9 embryonal carcinoma cells as determined by transient expression assaysEmbryonal long terminal repeat-binding protein is a murine homolog of FTZ-F1, a member of the steroid receptor superfamily.Genetically modified skin fibroblasts persist long after transplantation but gradually inactivate introduced genes.Transcriptional Silencing of Moloney Murine Leukemia Virus in Human Embryonic Carcinoma Cells.EBP1, a novel host factor involved in primer binding site-dependent restriction of moloney murine leukemia virus in embryonic cells.Host cis-mediated extinction of a retrovirus permissive for expression in embryonal stem cells during differentiation.Lack of shielding of primer binding site silencer-mediated repression of an internal promoter in a retrovirus vector by the putative insulators scs, BEAD-1, and HS4.Amelioration of retroviral vector silencing in locus control region beta-globin-transgenic mice and transduced F9 embryonic cells.Inactivation of the Moloney murine leukemia virus long terminal repeat in murine fibroblast cell lines is associated with methylation and dependent on its chromosomal position.Determinants of retrovirus gene expression in embryonal carcinoma cells.Analysis of the binding proteins and activity of the long terminal repeat of Moloney murine leukemia virus during differentiation of mouse embryonal carcinoma cells.A single point mutation activates the Moloney murine leukemia virus long terminal repeat in embryonal stem cells.NF-kappa B-independent activation of beta-interferon expression in mouse F9 embryonal carcinoma cellsMechanism of suppression of the long terminal repeat of Moloney leukemia virus in mouse embryonal carcinoma cells.Distinct classes of factor-independent mutants can be isolated after retroviral mutagenesis of a human myeloid stem cell line.Gammaretroviral vector encoding a fluorescent marker to facilitate detection of reprogrammed human fibroblasts during iPSC generation.High expression of transgenes mediated by hybrid retroviral vectors in hepatocytes: comparison of promoters from murine retroviruses in vitro and in vivo.
P2860
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P2860
Functional analysis of a retroviral host-range mutant: altered long terminal repeat sequences allow expression in embryonal carcinoma cells.
description
1987 nî lūn-bûn
@nan
1987 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1987 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
name
Functional analysis of a retro ...... in embryonal carcinoma cells.
@ast
Functional analysis of a retro ...... in embryonal carcinoma cells.
@en
Functional analysis of a retro ...... in embryonal carcinoma cells.
@nl
type
label
Functional analysis of a retro ...... in embryonal carcinoma cells.
@ast
Functional analysis of a retro ...... in embryonal carcinoma cells.
@en
Functional analysis of a retro ...... in embryonal carcinoma cells.
@nl
prefLabel
Functional analysis of a retro ...... in embryonal carcinoma cells.
@ast
Functional analysis of a retro ...... in embryonal carcinoma cells.
@en
Functional analysis of a retro ...... in embryonal carcinoma cells.
@nl
P2093
P2860
P356
P1476
Functional analysis of a retro ...... in embryonal carcinoma cells.
@en
P2093
P2860
P304
P356
10.1073/PNAS.84.15.5232
P407
P577
1987-08-01T00:00:00Z