Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1.
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Endothelin-1 and its role in the pathogenesis of infectious diseasesThe pathobiology of vascular dementiaCerebral hypoperfusion: a new pathophysiologic concept in multiple sclerosis?Perfusion by Arterial Spin labelling following Single dose Tadalafil In Small vessel disease (PASTIS): study protocol for a randomised controlled trial.Influence of Circulating Endothelin-1 and Asymmetric Dimethylarginine on Whole Brain Circulation Time in Multiple Sclerosis.Circulating endothelin-1 alters critical mechanisms regulating cerebral microcirculationInterleukin-1 and acute brain injury.Cerebral circulation time is prolonged and not correlated with EDSS in multiple sclerosis patients: a study using digital subtracted angiography.Systemic inflammation impairs tissue reperfusion through endothelin-dependent mechanisms in cerebral ischemia.Significant Contribution of Mouse Mast Cell Protease 4 in Early Phases of Experimental Autoimmune EncephalomyelitisFluoxetine in progressive multiple sclerosis (FLUOX-PMS): study protocol for a randomized controlled trial.Cerebral hypoperfusion and glucose hypometabolism: Key pathophysiological modulators promote neurodegeneration, cognitive impairment, and Alzheimer's disease.Flavonol-rich dark cocoa significantly decreases plasma endothelin-1 and improves cognition in urban children.Vascular hypoperfusion in acute optic neuritis is a potentially new neurovascular model for demyelinating diseases.Hemodynamic evidence linking cognitive deficits in clinically isolated syndrome to regional brain inflammation.What Have We Learned from Perfusion MRI in Multiple Sclerosis?Brain perfusion by arterial spin labeling MRI in multiple sclerosis.Blockade of sustained tumor necrosis factor in a transgenic model of progressive autoimmune encephalomyelitis limits oligodendrocyte apoptosis and promotes oligodendrocyte maturation.Cerebral Blood Flow Changes in Multiple Sclerosis and Neuromyelitis Optica and Their Correlations With Clinical Disability.Impaired Cerebral Perfusion in Multiple Sclerosis: Relevance of Endothelial Factors.
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P2860
Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1.
description
2013 nî lūn-bûn
@nan
2013 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի մարտին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1.
@ast
Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1.
@en
Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1.
@nl
type
label
Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1.
@ast
Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1.
@en
Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1.
@nl
prefLabel
Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1.
@ast
Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1.
@en
Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1.
@nl
P2093
P2860
P356
P1476
Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1.
@en
P2093
Anne-Marie Vanbinst
Christian Verborgh
Jacques De Keyser
Johan Demey
Melissa Cambron
Miguel D'haeseleer
Roel Beelen
Yves Fierens
P2860
P304
P356
10.1073/PNAS.1222560110
P407
P577
2013-03-18T00:00:00Z