Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice.
about
Pathogenic mechanisms in type 1 diabetes: the islet is both target and driver of disease.Perinatal tolerance to proinsulin is sufficient to prevent autoimmune diabetes.Association of increased eomesodermin, BCL6, and granzyme B expression with major clinical manifestations of Hashimoto's thyroiditis - an observational study.Genetic deletion of granzyme B does not confer resistance to the development of spontaneous diabetes in non-obese diabetic mice.
P2860
Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice.
description
2012 nî lūn-bûn
@nan
2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice.
@ast
Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice.
@en
Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice.
@nl
type
label
Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice.
@ast
Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice.
@en
Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice.
@nl
prefLabel
Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice.
@ast
Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice.
@en
Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice.
@nl
P2093
P2860
P1433
P1476
Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice.
@en
P2093
Balasubramanian Krishnamurthy
Helen E Thomas
Kate L Graham
Prerak Trivedi
Thomas W Kay
Zia U Mollah
P2860
P304
P356
10.1371/JOURNAL.PONE.0040357
P407
P577
2012-07-09T00:00:00Z