Phenotypic alterations in fos-transgenic mice correlate with changes in Fos/Jun-dependent collagenase type I expression. Regulation of mouse metalloproteinases by carcinogens, tumor promoters, cAMP, and Fos oncoprotein.

Phenotypic alterations in fos-transgenic mice correlate with changes in Fos/Jun-dependent collagenase type I expression. Regulation of mouse metalloproteinases by carcinogens, tumor promoters, cAMP, and Fos oncoprotein.

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http://www.wikidata.org/entity/Q34338492
CHOP enhancement of gene transcription by interactions with Jun/Fos AP-1 complex proteinsNeoplastic transformation of rat thyroid cells requires the junB and fra-1 gene induction which is dependent on the HMGI-C gene productThe stromal proteinase MMP3/stromelysin-1 promotes mammary carcinogenesis.The matrix metalloproteinase stromelysin-1 acts as a natural mammary tumor promoter.Factor recruitment and TIF2/GRIP1 corepressor activity at a collagenase-3 response element that mediates regulation by phorbol esters and hormones.Two distinct phases of apoptosis in mammary gland involution: proteinase-independent and -dependent pathways.Matrix metalloproteinases are expressed during ductal and alveolar mammary morphogenesis, and misregulation of stromelysin-1 in transgenic mice induces unscheduled alveolar developmentc-Fos-dependent induction of the small ras-related GTPase Rab11a in skin carcinogenesisDefective trophoblast function in mice with a targeted mutation of Ets2.Early events of metastasis in the microcirculation involve changes in gene expression of cancer cells. Tracking mRNA levels of metastasizing cancer cells in the chick embryo chorioallantoic membrane.Red Light Modulates Ultraviolet-Induced Gene Expression in the Epidermis of Hairless MiceThe DNA binding-independent function of the glucocorticoid receptor mediates repression of AP-1-dependent genes in skin.Structural organization and chromosomal localization of the mouse collagenase type I gene.c-Jun-dependent CD95-L expression is a rate-limiting step in the induction of apoptosis by alkylating agentsCollagenase expression in transgenic mouse skin causes hyperkeratosis and acanthosis and increases susceptibility to tumorigenesis.Fos is an essential component of the mammalian UV response.
P2860

Phenotypic alterations in fos-transgenic mice correlate with changes in Fos/Jun-dependent collagenase type I expression. Regulation of mouse metalloproteinases by carcinogens, tumor promoters, cAMP, and Fos oncoprotein.

Item
http://www.wikidata.org/entity/Q34338492
description
1994 nî lūn-bûn
@nan
1994 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1994 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
name
Phenotypic alterations in fos- ...... rs, cAMP, and Fos oncoprotein.
@ast
Phenotypic alterations in fos- ...... rs, cAMP, and Fos oncoprotein.
@en
Phenotypic alterations in fos- ...... rs, cAMP, and Fos oncoprotein.
@nl
type
label
Phenotypic alterations in fos- ...... rs, cAMP, and Fos oncoprotein.
@ast
Phenotypic alterations in fos- ...... rs, cAMP, and Fos oncoprotein.
@en
Phenotypic alterations in fos- ...... rs, cAMP, and Fos oncoprotein.
@nl
prefLabel
Phenotypic alterations in fos- ...... rs, cAMP, and Fos oncoprotein.
@ast
Phenotypic alterations in fos- ...... rs, cAMP, and Fos oncoprotein.
@en
Phenotypic alterations in fos- ...... rs, cAMP, and Fos oncoprotein.
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P1433
P1476
P2093
P304
P31
P407
P433
P478
P577
P698
P1433
P1476
Phenotypic alterations in fos- ...... ers, cAMP, and Fos oncoprotein
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P2093
P304
P31
P407
P433
P478
P577
P698