Presence of six different lesion types suggests diverse mechanisms of tissue injury in neuromyelitis optica - Wikidata - wikimedia - TriplyDB

Presence of six different lesion types suggests diverse mechanisms of tissue injury in neuromyelitis optica

Presence of six different lesion types suggests diverse mechanisms of tissue injury in neuromyelitis optica

http://www.wikidata.org/entity/Q34338762

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Antibodies as Mediators of Brain Pathology Aquaporins in the Spinal Cord Histopathology and clinical course of MOG-antibody-associated encephalomyelitis Fulminant demyelinating encephalomyelitis: Insights from antibody studies and neuropathology Anti-aquaporin-1 autoantibodies in patients with neuromyelitis optica spectrum disorders.Clinical spectrum and treatment of neuromyelitis optica spectrum disorders: evolution and current status.Neuromyelitis optica: clinical features, immunopathogenesis and treatment.Neuromyelitis optica spectrum disorders associated with other autoimmune diseases.Myelin Oligodendrocyte Glycoprotein: Deciphering a Target in Inflammatory Demyelinating Diseases.Aquaporin-4 antibody-positive myelitis initially biopsied for suspected spinal cord tumors: diagnostic considerations.Experimental models of neuromyelitis optica.Differential diagnosis of neuromyelitis optica spectrum disorders.Longitudinally extensive NMO spinal cord pathology produced by passive transfer of NMO-IgG in mice lacking complement inhibitor CD59.The pathology of an autoimmune astrocytopathy: lessons learned from neuromyelitis optica.The treatment of neuromyelitis optica.Early disruption of glial communication via connexin gap junction in multiple sclerosis, Baló's disease and neuromyelitis optica.Effectiveness of mycophenolate mofetil as first-line therapy in AQP4-IgG, MOG-IgG, and seronegative neuromyelitis optica spectrum disorders.Diagnostic utility of aquaporin-4 in the analysis of active demyelinating lesions.The intrinsic pathogenic role of autoantibodies to aquaporin 4 mediating spinal cord disease in a rat passive-transfer model Frontal white matter hyperintensities, clasmatodendrosis and gliovascular abnormalities in ageing and post-stroke dementia Antibodies to aquaporin-1 are not present in neuromyelitis optica.Severely exacerbated neuromyelitis optica rat model with extensive astrocytopathy by high affinity anti-aquaporin-4 monoclonal antibody.Multiple sclerosis and neuromyelitis optica spectrum disorders: some similarities in two distinct diseases.Inflammatory demyelination without astrocyte loss in MOG antibody-positive NMOSD In vivo imaging reveals rapid astrocyte depletion and axon damage in a model of neuromyelitis optica-related pathology.Complement-independent retinal pathology produced by intravitreal injection of neuromyelitis optica immunoglobulin G.Variable sensitivity to complement-dependent cytotoxicity in murine models of neuromyelitis optica.Finding NMO: The Evolving Diagnostic Criteria of Neuromyelitis Optica.Evidence of aquaporin involvement in human central pontine myelinolysis.Effects of neuromyelitis optica-IgG at the blood-brain barrier in vitro Experimental mouse model of optic neuritis with inflammatory demyelination produced by passive transfer of neuromyelitis optica-immunoglobulin G.Complement C3 on microglial clusters in multiple sclerosis occur in chronic but not acute disease: Implication for disease pathogenesis.Endogenous reactive oxygen species cause astrocyte defects and neuronal dysfunctions in the hippocampus: a new model for aging brain.Unique neuromyelitis optica pathology produced in naïve rats by intracerebral administration of NMO-IgG Mechanisms of white matter damage in multiple sclerosis.B cells in MS and NMO: pathogenesis and therapy.Pain in neuromyelitis optica--prevalence, pathogenesis and therapy.Neuromyelitis optica: a positive appraisal of seronegative cases.Experimental models of neuromyelitis optica: current status, challenges and future directions.Inherited and acquired disorders of myelin: The underlying myelin pathology.

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Presence of six different lesion types suggests diverse mechanisms of tissue injury in neuromyelitis optica

http://www.wikidata.org/entity/Q34338762

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2013 nî lūn-bûn

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2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2013 թվականի ապրիլին հրատարակված գիտական հոդված

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2013年の論文

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Presence of six different lesi ...... injury in neuromyelitis optica

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Presence of six different lesi ...... injury in neuromyelitis optica

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Presence of six different lesi ...... injury in neuromyelitis optica

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Presence of six different lesi ...... injury in neuromyelitis optica

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Presence of six different lesi ...... injury in neuromyelitis optica

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Presence of six different lesi ...... injury in neuromyelitis optica

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Presence of six different lesi ...... injury in neuromyelitis optica

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Presence of six different lesi ...... injury in neuromyelitis optica

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Acta Neuropathologica

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Ferenc Garzuly

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Hans Lassmann

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Hidehiko Konno

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Ichiro Nakashima

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Isabella Wimmer

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Kazuo Fujihara

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Masashi Aoki

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Shuhei Nishiyama

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P2860

Human astrocytes express aquaporin-1 and aquaporin-4 in vitro and in vivo

IgG marker of optic-spinal multiple sclerosis binds to the aquaporin-4 water channel

Mitochondrial defects in acute multiple sclerosis lesions

Potassium channel KIR4.1 as an immune target in multiple sclerosis

Neuromyelitis optica lesions may inform multiple sclerosis heterogeneity debate

The immunopathology of multiple sclerosis: an overview

Astrocytic clasmatodendrosis in hippocampal organ culture

Aquaporin-4 astrocytopathy in Baló’s disease

Heterogeneity of multiple sclerosis lesions: implications for the pathogenesis of demyelination

A serum autoantibody marker of neuromyelitis optica: distinction from multiple sclerosis

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