Biological and biochemical consequences of global deletion of exon 3 from the ER alpha gene - Wikidata - wikimedia - TriplyDB

Biological and biochemical consequences of global deletion of exon 3 from the ER alpha gene

Biological and biochemical consequences of global deletion of exon 3 from the ER alpha gene

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What's new in estrogen receptor action in the female reproductive tract Serine 216 phosphorylation of estrogen receptor α in neutrophils: migration and infiltration into the mouse uterus Nuclear Receptor Signaling Atlas: Opening Access to the Biology of Nuclear Receptor Signaling Pathways Expression of Human NSAID Activated Gene 1 in Mice Leads to Altered Mammary Gland Differentiation and Impaired Lactation Design of pathway preferential estrogens that provide beneficial metabolic and vascular effects without stimulating reproductive tissues Generation of Esr1-knockout rats using zinc finger nuclease-mediated genome editing.Estrogen response element-independent signaling partially restores post-ovariectomy body weight gain but is not sufficient for 17β-estradiol's control of energy homeostasis Novel DNA motif binding activity observed in vivo with an estrogen receptor α mutant mouse.Estrogen responsiveness of the TFIID subunit TAF4B in the normal mouse ovary and in ovarian tumors An animal model with a cardiomyocyte-specific deletion of estrogen receptor alpha: functional, metabolic, and differential network analysis Ex3αERKO male infertility phenotype recapitulates the αERKO male phenotype Sex steroid actions in male bone Estrogenic activity of bisphenol A and 2,2-bis(p-hydroxyphenyl)-1,1,1-trichloroethane (HPTE) demonstrated in mouse uterine gene profiles.What have we learned about GPER function in physiology and disease from knockout mice?The absence of ER-β results in altered gene expression in ovarian granulosa cells isolated from in vivo preovulatory follicles Estrogen receptor (ER)α-regulated lipocalin 2 expression in adipose tissue links obesity with breast cancer progression Estrogen receptor α AF-2 mutation results in antagonist reversal and reveals tissue selective function of estrogen receptor modulators.Myeloid-specific estrogen receptor alpha deficiency impairs metabolic homeostasis and accelerates atherosclerotic lesion development.Predicting the future: opportunities and challenges for the chemical industry to apply 21st-century toxicity testing.Development of phenotypic and transcriptional biomarkers to evaluate relative activity of potentially estrogenic chemicals in ovariectomized mice Uterine gland formation in mice is a continuous process, requiring the ovary after puberty, but not after parturition.Uterine epithelial cell estrogen receptor alpha-dependent and -independent genomic profiles that underlie estrogen responses in mice.Uterine Epithelial Estrogen Receptor-α Controls Decidualization via a Paracrine Mechanism.Uterine Expression of NDRG4 Is Induced by Estrogen and Up-Regulated during Embryo Implantation Process in Mice.Endometrial tumorigenesis in Pten(+/-) mice is independent of coexistence of estrogen and estrogen receptor α.Neonatal Progesterone Programs Adult Uterine Responses to Progesterone and Susceptibility to Uterine Dysfunction Role of estrogen receptor signaling required for endometriosis-like lesion establishment in a mouse model.Oviductal estrogen receptor α signaling prevents protease-mediated embryo death Transactivating function (AF) 2-mediated AF-1 activity of estrogen receptor α is crucial to maintain male reproductive tract function.Requirement for stromal estrogen receptor alpha in cervical neoplasia Roles of Estrogen Receptor-α and the Coactivator MED1 During Human Endometrial Decidualization.Estrogen hormone physiology: reproductive findings from estrogen receptor mutant mice.Male risk taking, female odors, and the role of estrogen receptors.Dual suppression of estrogenic and inflammatory activities for targeting of endometriosis LXRβ/estrogen receptor-α signaling in lipid rafts preserves endothelial integrity.Skeletal muscle action of estrogen receptor α is critical for the maintenance of mitochondrial function and metabolic homeostasis in females Estrogen receptor α L543A,L544A mutation changes antagonists to agonists, correlating with the ligand binding domain dimerization associated with DNA binding activity.aP2-Cre-mediated inactivation of estrogen receptor alpha causes hydrometra.SCA-1 Labels a Subset of Estrogen-Responsive Bipotential Repopulating Cells within the CD24+ CD49fhi Mammary Stem Cell-Enriched Compartment.Loss of ERα partially reverses the effects of maternal high-fat diet on energy homeostasis in female mice.

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P2860

Biological and biochemical consequences of global deletion of exon 3 from the ER alpha gene

http://www.wikidata.org/entity/Q34352842

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2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年学术文章

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2010年学术文章

@zh-cn

2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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name

Biological and biochemical con ...... exon 3 from the ER alpha gene

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Biological and biochemical con ...... exon 3 from the ER alpha gene

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Biological and biochemical con ...... exon 3 from the ER alpha gene

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type

label

Biological and biochemical con ...... exon 3 from the ER alpha gene

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Biological and biochemical con ...... exon 3 from the ER alpha gene

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Biological and biochemical con ...... exon 3 from the ER alpha gene

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prefLabel

Biological and biochemical con ...... exon 3 from the ER alpha gene

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Biological and biochemical con ...... exon 3 from the ER alpha gene

@en

Biological and biochemical con ...... exon 3 from the ER alpha gene

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Biological and biochemical con ...... exon 3 from the ER alpha gene

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Grace E Kissling

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Karen E Fieselman

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Kevin E Gerrish

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P2860

Effect of single and compound knockouts of estrogen receptors alpha (ERalpha) and beta (ERbeta) on mouse reproductive phenotypes

Estrogen Receptor Null Mice: What Have We Learned and Where Will They Lead Us?

Additional weight bearing during exercise and estrogen in the rat: the effect on bone mass, turnover, and structure.

Obesity and disturbed lipoprotein profile in estrogen receptor-alpha-deficient male mice.

Increased adipose tissue in male and female estrogen receptor-alpha knockout mice.

Generation and characterization of a complete null estrogen receptor alpha mouse using Cre/LoxP technology.

Alteration of reproductive function but not prenatal sexual development after insertional disruption of the mouse estrogen receptor gene.

Estrogen receptor specificity in the regulation of skeletal growth and maturation in male mice.

Physiological coupling of growth factor and steroid receptor signaling pathways: estrogen receptor knockout mice lack estrogen-like response to epidermal growth factor.

Selective disruption of ER{alpha} DNA-binding activity alters uterine responsiveness to estradiol.

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12

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Kenneth S. Korach

Sylvia C Hewitt

Friederike L Jayes

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2010-07-28T00:00:00Z

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45406356

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