Passage through the mammalian gut triggers a phenotypic switch that promotes Candida albicans commensalism - Wikidata - wikimedia - TriplyDB

Passage through the mammalian gut triggers a phenotypic switch that promotes Candida albicans commensalism

Passage through the mammalian gut triggers a phenotypic switch that promotes Candida albicans commensalism

http://www.wikidata.org/entity/Q34360240

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Genetic and phenotypic intra-species variation in Candida albicans Systems Level Dissection of Candida Recognition by Dectins: A Matter of Fungal Morphology and Site of Infection Candida albicans the chameleon: transitions and interactions between multiple phenotypic states confer phenotypic plasticity N-acetylglucosamine Regulates Virulence Properties in Microbial Pathogens The role of phenotypic switching in the basic biology and pathogenesis of Candida albicans The cryptic sexual strategies of human fungal pathogens Structure of a new DNA-binding domain which regulates pathogenesis in a wide variety of fungi Crystal structure of the WOPR-DNA complex and implications for Wor1 function in white-opaque switching of Candida albicans Recent advances on Candida albicans biology and virulence Cell biology of Candida albicans-host interactions Experimental Models of Vaginal Candidiasis and Their Relevance to Human Candidiasis The non-receptor tyrosine kinase Tec controls assembly and activity of the noncanonical caspase-8 inflammasome Amplification of TLO Mediator Subunit Genes Facilitate Filamentous Growth in Candida Spp Phenotypic Profiling Reveals that Candida albicans Opaque Cells Represent a Metabolically Specialized Cell State Compared to Default White Cells Discovery of the gray phenotype and white-gray-opaque tristable phenotypic transitions in Candida dubliniensis Phenotypic switch: The enigmatic white-gray-opaque transition system of Candida albicans.How duplicated transcription regulators can diversify to govern the expression of nonoverlapping sets of genes Iron-responsive chromatin remodelling and MAPK signalling enhance adhesion in Candida albicans.Overview of vertebrate animal models of fungal infection.A relay network of extracellular heme-binding proteins drives C. albicans iron acquisition from hemoglobin.Discovery of a "white-gray-opaque" tristable phenotypic switching system in candida albicans: roles of non-genetic diversity in host adaptation.Candida albicans commensalism in the gastrointestinal tract.Candida albicans cell-type switching and functional plasticity in the mammalian host.Anaerobic bacteria grow within Candida albicans biofilms and induce biofilm formation in suspension cultures The mycobiota: interactions between commensal fungi and the host immune system.Functional profiling of human fungal pathogen genomes.Richness and diversity of mammalian fungal communities shape innate and adaptive immunity in health and disease.Candida albicans colonization and dissemination from the murine gastrointestinal tract: the influence of morphology and Th17 immunity Innate Defense against Fungal Pathogens Finding a Missing Gene: EFG1 Regulates Morphogenesis in Candida tropicalis.Hypoxia and Temperature Regulated Morphogenesis in Candida albicans RNA Enrichment Method for Quantitative Transcriptional Analysis of Pathogens In Vivo Applied to the Fungus Candida albicans Immune Interactions with Pathogenic and Commensal Fungi: A Two-Way Street Distinct stages during colonization of the mouse gastrointestinal tract by Candida albicans The WOR1 5' untranslated region regulates white-opaque switching in Candida albicans by reducing translational efficiency N-Acetylglucosamine-Induced Cell Death in Candida albicans and Its Implications for Adaptive Mechanisms of Nutrient Sensing in Yeasts.A Multistate Toggle Switch Defines Fungal Cell Fates and Is Regulated by Synergistic Genetic Cues Candida albicans ISW2 Regulates Chlamydospore Suspensor Cell Formation and Virulence In Vivo in a Mouse Model of Disseminated Candidiasis.Function and Regulation of Cph2 in Candida albicans.Transcriptional Profiling of Candida albicans in the Host

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P2860

Passage through the mammalian gut triggers a phenotypic switch that promotes Candida albicans commensalism

http://www.wikidata.org/entity/Q34360240

description

2013 nî lūn-bûn

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2013 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2013 թվականի հուլիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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Passage through the mammalian ...... Candida albicans commensalism

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Passage through the mammalian ...... Candida albicans commensalism

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Nature Genetics

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Passage through the mammalian ...... Candida albicans commensalism

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Changbin Chen

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Kalyan Pande

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Suzanne M Noble

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P2860

Interlocking transcriptional feedback loops control white-opaque switching in Candida albicans

The Fungi: 1, 2, 3 ... 5.1 million species?

Epigenetic properties of white-opaque switching in Candida albicans are based on a self-sustaining transcriptional feedback loop

Bayesian analysis of gene expression levels: statistical quantification of relative mRNA level across multiple strains or treatments

Accidental virulence, cryptic pathogenesis, martians, lost hosts, and the pathogenicity of environmental microbes

Goulphar: rapid access and expertise for standard two-color microarray normalization methods.

Systematic screens of a Candida albicans homozygous deletion library decouple morphogenetic switching and pathogenicity.

The transcriptomes of two heritable cell types illuminate the circuit governing their differentiation

Strains and strategies for large-scale gene deletion studies of the diploid human fungal pathogen Candida albicans

White-opaque switching in Candida albicans is controlled by mating-type locus homeodomain proteins and allows efficient mating.

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10.1038/NG.2710

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9

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23892606

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3758371

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