STAT3 mutations indicate the presence of subclinical T-cell clones in a subset of aplastic anemia and myelodysplastic syndrome patients.
about
Large granular lymphocytosis during dasatinib therapyClinical impact of recurrently mutated genes on lymphoma diagnostics: state-of-the-art and beyondEarly-onset lymphoproliferation and autoimmunity caused by germline STAT3 gain-of-function mutationsThe analysis of clonal diversity and therapy responses using STAT3 mutations as a molecular marker in large granular lymphocytic leukemia.The pathogenesis and treatment of large granular lymphocyte leukemiaSTAT3 gene mutations and their association with pure red cell aplasia in large granular lymphocyte leukemiaThe Ying and Yang of STAT3 in Human DiseaseMutations in the linker domain affect phospho-STAT3 function and suggest targets for interrupting STAT3 activity.Anemia of Central Origin.The genetic basis of myelodysplasia and its clinical relevanceUncovering the pathogenesis of large granular lymphocytic leukemia-novel STAT3 and STAT5b mutations.Disease-specific mutations in mature lymphoid neoplasms: recent advances.STAT3 inhibitors: finding a home in lymphoma and leukemia.Aplastic anemia: possible associations with lymphoproliferative neoplasms.The JAK-STAT pathway: impact on human disease and therapeutic interventionClinical significance of acquired somatic mutations in aplastic anaemia.Emerging biological therapies for the treatment of myelodysplastic syndromes.Convergent mutations and kinase fusions lead to oncogenic STAT3 activation in anaplastic large cell lymphoma.Asymptomatic circulating T-cell clone cause renal polymorphic inflammatory fibrosis.Persistent cytotoxic T lymphocyte expansions after allogeneic haematopoietic stem cell transplantation: kinetics, clinical impact and absence of STAT3 mutations.Long-term outcome of patients with acquired chronic pure red cell aplasia (PRCA) following immunosuppressive therapy: a final report of the nationwide cohort study in 2004/2006 by the Japan PRCA collaborative study group.Clinical Features, Pathogenesis, and Treatment of Large Granular Lymphocyte Leukemias.Cytotoxic T cells in chronic idiopathic neutropenia express restricted antigen receptors.Diagnosis and Treatment of Aplastic Anemia.Concurrent STAT3, DNMT3A, and TET2 mutations in T-LGL leukemia with molecularly distinct clonal hematopoiesis of indeterminate potential.Spectrum of Clonal Large Granular Lymphocytes (LGLs) of αβ T Cells: T-Cell Clones of Undetermined Significance, T-Cell LGL Leukemias, and T-Cell Immunoclones.Frequent mutations in CD8 T cells from patients with pure red cell aplasia
P2860
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P2860
STAT3 mutations indicate the presence of subclinical T-cell clones in a subset of aplastic anemia and myelodysplastic syndrome patients.
description
2013 nî lūn-bûn
@nan
2013 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
STAT3 mutations indicate the p ...... odysplastic syndrome patients.
@ast
STAT3 mutations indicate the p ...... odysplastic syndrome patients.
@en
STAT3 mutations indicate the p ...... odysplastic syndrome patients.
@nl
type
label
STAT3 mutations indicate the p ...... odysplastic syndrome patients.
@ast
STAT3 mutations indicate the p ...... odysplastic syndrome patients.
@en
STAT3 mutations indicate the p ...... odysplastic syndrome patients.
@nl
prefLabel
STAT3 mutations indicate the p ...... odysplastic syndrome patients.
@ast
STAT3 mutations indicate the p ...... odysplastic syndrome patients.
@en
STAT3 mutations indicate the p ...... odysplastic syndrome patients.
@nl
P2093
P2860
P50
P1433
P1476
STAT3 mutations indicate the p ...... odysplastic syndrome patients.
@en
P2093
Alan E Lichtin
Aleksandra Wodnar-Filipowicz
Andres Jerez
André Tichelli
Austin Kulasekararaj
Bartlomiej Przychodzen
Catherine Nissen
Francis LeBlanc
Ghulam J Mufti
P2860
P304
P356
10.1182/BLOOD-2013-04-494930
P407
P577
2013-08-07T00:00:00Z