Exogenous antigens are processed through the endoplasmic reticulum-associated degradation (ERAD) in cross-presentation by dendritic cells.
about
DEC-205 receptor on dendritic cells mediates presentation of HIV gag protein to CD8+ T cells in a spectrum of human MHC I haplotypesThe Role of FcRn in Antigen PresentationMHC class I endosomal and lysosomal trafficking coincides with exogenous antigen loading in dendritic cellsMHC class I antigen presentation of DRiP-derived peptides from a model antigen is not dependent on the AAA ATPase p97The interaction between the ER membrane protein UNC93B and TLR3, 7, and 9 is crucial for TLR signalingMannose receptor polyubiquitination regulates endosomal recruitment of p97 and cytosolic antigen translocation for cross-presentation.Cross-presentation of synthetic long peptides by human dendritic cells: a process dependent on ERAD component p97/VCP but Not sec61 and/or Derlin-1.Mechanisms of MHC class I-restricted antigen processing and cross-presentation.Cross-presentation: underlying mechanisms and role in immune surveillance.Ubiquitin ligases, critical mediators of endoplasmic reticulum-associated degradationThe cell biology of cross-presentation and the role of dendritic cell subsets.Receptor-mediated phagocytosis elicits cross-presentation in nonprofessional antigen-presenting cells.Immuno-potentiating pathway of HBsAg-HBIG immunogenic complex visualized.MHC class I assembly: out and about.Enhanced endoplasmic reticulum entry of tumor antigen is crucial for cross-presentation induced by dendritic cell-targeted vaccination.Heat shock proteins in antigen trafficking--implications on antigen presentation to T cells.Surrogate in vitro activation of innate immunity synergizes with interleukin-7 to unleash rapid antigen-driven outgrowth of CD4+ and CD8+ human peripheral blood T-cells naturally recognizing MUC1, HER2/neu and other tumor-associated antigens.Cross-presentation: how to get there - or how to get the ER.Murine and Human Model Systems for the Study of Dendritic Cell Immunobiology.Lipid peroxidation causes endosomal antigen release for cross-presentation.Antigen Cross-Presentation and Heat Shock Protein-Based Vaccines.Sec61 blockade by mycolactone inhibits antigen cross-presentation independently of endosome-to-cytosol export.The ongoing saga of the mechanism(s) of MHC class I-restricted cross-presentation.Spatial separation of the processing and MHC class I loading compartments for cross-presentation of the tumor-associated antigen HER2/neu by human dendritic cells.The Biology and Underlying Mechanisms of Cross-Presentation of Exogenous Antigens on MHC-I Molecules.Chemokine programming dendritic cell antigen response: part I - select chemokine programming of antigen uptake even after maturation.Extracellular heat shock protein 90 plays a role in translocating chaperoned antigen from endosome to proteasome for generating antigenic peptide to be cross-presented by dendritic cells.TLR and NLRP3 inflammasome-dependent innate immune responses to tumor-derived autophagosomes (DRibbles).Exposure to Melan-A/MART-126-35 tumor epitope specific CD8(+)T cells reveals immune escape by affecting the ubiquitin-proteasome system (UPS)Purification of the Membrane Compartment for Endoplasmic Reticulum-associated Degradation of Exogenous Antigens in Cross-presentation.Purification of the subcellular compartment in which exogenous antigens undergo endoplasmic reticulum-associated degradation from dendritic cells.Carbohydrate mimetic peptides augment carbohydrate-reactive immune responses in the absence of immune pathology.Intracellular events regulating cross-presentation.Stripy nanoparticles revisited.Augmented induction of CD8+ cytotoxic T-cell response and antitumour resistance by T helper type 1-inducing peptide.Establishment and Characterization of a Functionally Competent Type 2 Conventional Dendritic Cell LineA novel probe to assess cytosolic entry of exogenous proteins
P2860
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P2860
Exogenous antigens are processed through the endoplasmic reticulum-associated degradation (ERAD) in cross-presentation by dendritic cells.
description
2004 nî lūn-bûn
@nan
2004 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Exogenous antigens are process ...... esentation by dendritic cells.
@ast
Exogenous antigens are process ...... esentation by dendritic cells.
@en
Exogenous antigens are processed through the endoplasmic reticulum-associated degradation
@nl
type
label
Exogenous antigens are process ...... esentation by dendritic cells.
@ast
Exogenous antigens are process ...... esentation by dendritic cells.
@en
Exogenous antigens are processed through the endoplasmic reticulum-associated degradation
@nl
prefLabel
Exogenous antigens are process ...... esentation by dendritic cells.
@ast
Exogenous antigens are process ...... esentation by dendritic cells.
@en
Exogenous antigens are processed through the endoplasmic reticulum-associated degradation
@nl
P2093
P356
P1476
Exogenous antigens are process ...... resentation by dendritic cells
@en
P2093
Hironori Hasegawa
Ichiro Yahara
Mikako Maruya
P356
10.1093/INTIMM/DXH184
P577
2004-11-16T00:00:00Z