Progressive replacement of embryo-derived cardiac macrophages with age - Wikidata - wikimedia - TriplyDB

Progressive replacement of embryo-derived cardiac macrophages with age

Progressive replacement of embryo-derived cardiac macrophages with age

http://www.wikidata.org/entity/Q34375144

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Monocyte Heterogeneity: Consequences for Monocyte-Derived Immune Cells Parallel Aspects of the Microenvironment in Cancer and Autoimmune Disease Histone deacetylases in monocyte/macrophage development, activation and metabolism: refining HDAC targets for inflammatory and infectious diseases The Interplay Between Monocytes/Macrophages and CD4(+) T Cell Subsets in Rheumatoid Arthritis The development and maintenance of resident macrophages Transcriptional Regulation of Mononuclear Phagocyte Development Ontogeny of Tissue-Resident Macrophages New Insights Into Tissue Macrophages: From Their Origin to the Development of Memory A Hitchhiker's Guide to Myeloid Cell Subsets: Practical Implementation of a Novel Mononuclear Phagocyte Classification System Monocytes in myocardial infarction The interaction of anticancer therapies with tumor-associated macrophages Origin, fate and dynamics of macrophages at central nervous system interfaces Immune Modulation of Cardiac Repair and Regeneration: The Art of Mending Broken Hearts Tissue macrophages: heterogeneity and functions.Cre/lox Studies Identify Resident Macrophages as the Major Source of Circulating Coagulation Factor XIII-A.Cardiac resident macrophages are involved in hypoxia‑induced postnatal cardiomyocyte proliferation Monocytes find a new place to dwell in the niche of heartbreak hotel.The pancreas anatomy conditions the origin and properties of resident macrophages.Chasing the recipe for a pro-regenerative immune system Regulation of macrophage development and function in peripheral tissues Abandoning M1/M2 for a Network Model of Macrophage Function.The type of injury dictates the mode of repair in neonatal and adult heart.Tissue-resident macrophages: then and now.Myocardial infarction: a critical role of macrophages in cardiac remodeling.The adult murine heart has a sparse, phagocytically active macrophage population that expands through monocyte recruitment and adopts an 'M2' phenotype in response to Th2 immunologic challenge.Understanding the Mysterious M2 Macrophage through Activation Markers and Effector Mechanisms.Defining Mononuclear Phagocyte Subset Homology Across Several Distant Warm-Blooded Vertebrates Through Comparative Transcriptomics.Bone marrow-derived monocytes give rise to self-renewing and fully differentiated Kupffer cells Fate mapping reveals that microglia and recruited monocyte-derived macrophages are definitively distinguishable by phenotype in the retina.Innate immune cells in ischaemic heart disease: does myocardial infarction beget myocardial infarction?Monocyte and macrophage contributions to cardiac remodeling.Long-lived self-renewing bone marrow-derived macrophages displace embryo-derived cells to inhabit adult serous cavities Pivotal role for skin transendothelial radio-resistant anti-inflammatory macrophages in tissue repair GM-CSF primes cardiac inflammation in a mouse model of Kawasaki disease.MHC II+ resident peritoneal and pleural macrophages rely on IRF4 for development from circulating monocytes Macrophagic control of the response to uropathogenic E. coli infection by regulation of iron retention in an IL-6-dependent manner.Aging dysregulates D- and E-series resolvins to modulate cardiosplenic and cardiorenal network following myocardial infarction.Macrophages as Key Drivers of Cancer Progression and Metastasis.Macrophage activation and polarization in post-infarction cardiac remodeling.Myocardial aging as a T-cell-mediated phenomenon.

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Progressive replacement of embryo-derived cardiac macrophages with age

http://www.wikidata.org/entity/Q34375144

description

2014 nî lūn-bûn

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2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Progressive replacement of embryo-derived cardiac macrophages with age

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Progressive replacement of embryo-derived cardiac macrophages with age

@en

Progressive replacement of embryo-derived cardiac macrophages with age

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type

label

Progressive replacement of embryo-derived cardiac macrophages with age

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Progressive replacement of embryo-derived cardiac macrophages with age

@en

Progressive replacement of embryo-derived cardiac macrophages with age

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prefLabel

Progressive replacement of embryo-derived cardiac macrophages with age

@ast

Progressive replacement of embryo-derived cardiac macrophages with age

@en

Progressive replacement of embryo-derived cardiac macrophages with age

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Journal of Experimental Medicine

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Progressive replacement of embryo-derived cardiac macrophages with age

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Alexander R Pinto

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Hans-Reimer Rodewald

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Jeremy Favret

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Kaaweh Molawi

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Kathrin Frenzel

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Kay Klapproth

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Marc Bajenoff

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Marco Prinz

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Nora Hagemeyer

http://www.w3.org/2001/XMLSchema#string

Prashanth K Kandalla

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Identification of splenic reservoir monocytes and their deployment to inflammatory sites

Tissue-resident macrophages

The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

Fate mapping analysis reveals that adult microglia derive from primitive macrophages

Development of monocytes, macrophages, and dendritic cells

Analysis of fractalkine receptor CX(3)CR1 function by targeted deletion and green fluorescent protein reporter gene insertion

Gene-expression profiles and transcriptional regulatory pathways that underlie the identity and diversity of mouse tissue macrophages

A lineage of myeloid cells independent of Myb and hematopoietic stem cells

Transient regenerative potential of the neonatal mouse heart

Monocyte emigration from bone marrow during bacterial infection requires signals mediated by chemokine receptor CCR2

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2151-2158

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10.1084/JEM.20140639

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11

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211

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Bernard Malissen

Nadia Rosenthal

Michael H Sieweke

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2014-09-22T00:00:00Z

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266026945

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25245760

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4203946

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