Scavenger receptor class B type I (SR-BI) regulates perivascular macrophages and modifies amyloid pathology in an Alzheimer mouse model - Wikidata - wikimedia - TriplyDB

Scavenger receptor class B type I (SR-BI) regulates perivascular macrophages and modifies amyloid pathology in an Alzheimer mouse model

Scavenger receptor class B type I (SR-BI) regulates perivascular macrophages and modifies amyloid pathology in an Alzheimer mouse model

http://www.wikidata.org/entity/Q34375724

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Intracerebral haemorrhage in Down syndrome: protected or predisposed?Microglial cell dysregulation in brain aging and neurodegeneration Inflammatory events at blood-brain barrier in neuroinflammatory and neurodegenerative disorders: implications for clinical disease Microglia development and function Involvement of formyl peptide receptors in receptor for advanced glycation end products (RAGE)--and amyloid beta 1-42-induced signal transduction in glial cells Lymphatics in Neurological Disorders: A Neuro-Lympho-Vascular Component of Multiple Sclerosis and Alzheimer's Disease?Perivascular macrophages mediate the neurovascular and cognitive dysfunction associated with hypertension.Neuroinflammatory challenges compromise neuronal function in the aging brain: Postoperative cognitive delirium and Alzheimer's disease.Lack of LDL receptor enhances amyloid deposition and decreases glial response in an Alzheimer's disease mouse model.New comparative genomics approach reveals a conserved health span signature across species.Microglial scavenger receptors and their roles in the pathogenesis of Alzheimer's disease.Clearance of amyloid-β peptides by microglia and macrophages: the issue of what, when and where.Role of scavenger receptors in glia-mediated neuroinflammatory response associated with Alzheimer's disease.Innate immunity and neuroinflammation.Microglia, neuroinflammation, and beta-amyloid protein in Alzheimer's disease.Modulation of inflammation in transgenic models of Alzheimer's disease Imaging of cerebrovascular pathology in animal models of Alzheimer's disease The role of cholesterol metabolism in Alzheimer's disease.HDL and cholesterol handling in the brain.Microglial Aβ receptors in Alzheimer's disease.Recruiting specialized macrophages across the borders to restore brain functions.Scavenger receptor A deficiency accelerates cerebrovascular amyloidosis in an animal model.Brain perivascular macrophages: characterization and functional roles in health and disease.Treatment with D3 removes amyloid deposits, reduces inflammation, and improves cognition in aged AβPP/PS1 double transgenic mice.High-density lipoproteins suppress Aβ-induced PBMC adhesion to human endothelial cells in bioengineered vessels and in monoculture.Characterization of ApoJ-reconstituted high-density lipoprotein (rHDL) nanodisc for the potential treatment of cerebral β-amyloidosis.The Neurovascular Unit Coming of Age: A Journey through Neurovascular Coupling in Health and Disease.Alzheimer’s Disease, Brain Injury, and C.N.S. Nanotherapy in Humans: Sonoporation Augmenting Drug Targeting.ABCA7 Mediates Phagocytic Clearance of Amyloid-β in the Brain.Failure of perivascular drainage of β-amyloid in cerebral amyloid angiopathy.Insulin differentially affects the distribution kinetics of amyloid beta 40 and 42 in plasma and brain.Growth Arrest Specific 6 Concentration is Increased in the Cerebrospinal Fluid of Patients with Alzheimer's Disease.Enriched Environment Significantly Reduced Senile Plaques in a Transgenic Mice Model of Alzheimer's Disease, Improving Memory Caspase-1 inhibition alleviates cognitive impairment and neuropathology in an Alzheimer's disease mouse model CNS-border associated macrophages respond to acute ischemic stroke attracting granulocytes and promoting vascular leakage

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Scavenger receptor class B type I (SR-BI) regulates perivascular macrophages and modifies amyloid pathology in an Alzheimer mouse model

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description

2010 nî lūn-bûn

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2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年学术文章

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2010年學術文章

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Scavenger receptor class B typ ...... gy in an Alzheimer mouse model

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Scavenger receptor class B typ ...... gy in an Alzheimer mouse model

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P2093

Apostolia Fragkouli

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Fotini Stylianopoulou

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Kalliopi Thanopoulou

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Spiros Georgopoulos

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P2860

A targeted mutation in the murine gene encoding the high density lipoprotein (HDL) receptor scavenger receptor class B type I reveals its key role in HDL metabolism

Expression of scavenger receptor class B, type I, by astrocytes and vascular smooth muscle cells in normal adult mouse and human brain and in Alzheimer's disease brain

Abnormal lipoprotein metabolism and reversible female infertility in HDL receptor (SR-BI)-deficient mice

Loss of forebrain cholinergic neurons and impairment in spatial learning and memory in LHX7-deficient mice

Place navigation impaired in rats with hippocampal lesions

High-level neuronal expression of abeta 1-42 in wild-type human amyloid protein precursor transgenic mice: synaptotoxicity without plaque formation

Scavenger receptor class B type I (SR-BI) mediates adhesion of neonatal murine microglia to fibrillar beta-amyloid.

Targeted mutation reveals a central role for SR-BI in hepatic selective uptake of high density lipoprotein cholesterol.

Hepatic and extrahepatic scavenger receptors: function in relation to disease.

Perivascular microglial cells of the CNS are bone marrow-derived and present antigen in vivo.

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21076037

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Alzheimer's disease

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2996412

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