Imbalance of ionic conductances contributes to diverse symptoms of demyelination.
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Physiological Dynamics in Demyelinating Diseases: Unraveling Complex Relationships through Computer Modeling.The Hodgkin-Huxley heritage: from channels to circuitsSpontaneous excitation patterns computed for axons with injury-like impairments of sodium channels and Na/K pumpsMyelin Loss and Axonal Ion Channel Adaptations Associated with Gray Matter Neuronal HyperexcitabilityNeuromodulation independently determines correlated channel expression and conductance levels in motor neurons of the stomatogastric ganglion.Cognition in MS correlates with resting-state oscillatory brain activity: An explorative MEG source-space studyNeuronal activity in the hub of extrasynaptic Schwann cell-axon interactions.Explaining pathological changes in axonal excitability through dynamical analysis of conductance-based modelsCooperativity between remote sites of ectopic spiking allows afterdischarge to be initiated and maintained at different locationsLeft-shifted nav channels in injured bilayer: primary targets for neuroprotective nav antagonists?Dependence of Nociceptive Detection Thresholds on Physiological Parameters and Capsaicin-Induced Neuroplasticity: A Computational StudyFocal axonal swellings and associated ultrastructural changes attenuate conduction velocity in central nervous system axons: a computer modeling study.Transitions between classes of neuronal excitability and bifurcations induced by autapse.A computational model coupling mechanics and electrophysiology in spinal cord injury.Identification and continuity of the distributions of burst-length and interspike intervals in the stochastic Morris-Lecar neuron.Complex symptoms of demyelination and nerve damage explained by nonlinear dynamical analysis of conductance-based models.Computer modeling of mild axonal injury: implications for axonal signal transmission.Perinodal glial swelling mitigates axonal degradation in a model of axonal injury.Primary paranode demyelination modulates slowly developing axonal depolarization in a model of axonal injury.Coupled left-shift of Nav channels: modeling the Na⁺-loading and dysfunctional excitability of damaged axons.Calculating the Consequences of Left-Shifted Nav Channel Activity in Sick Excitable Cells.Spike-Conducting Integrate-and-Fire ModelA Process for Digitizing and Simulating Biologically Realistic Oligocellular Networks Demonstrated for the Neuro-Glio-Vascular Ensemble
P2860
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P2860
Imbalance of ionic conductances contributes to diverse symptoms of demyelination.
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2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
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2010年の論文
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2010年論文
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2010年論文
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2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
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Imbalance of ionic conductances contributes to diverse symptoms of demyelination.
@ast
Imbalance of ionic conductances contributes to diverse symptoms of demyelination.
@en
Imbalance of ionic conductances contributes to diverse symptoms of demyelination.
@nl
type
label
Imbalance of ionic conductances contributes to diverse symptoms of demyelination.
@ast
Imbalance of ionic conductances contributes to diverse symptoms of demyelination.
@en
Imbalance of ionic conductances contributes to diverse symptoms of demyelination.
@nl
prefLabel
Imbalance of ionic conductances contributes to diverse symptoms of demyelination.
@ast
Imbalance of ionic conductances contributes to diverse symptoms of demyelination.
@en
Imbalance of ionic conductances contributes to diverse symptoms of demyelination.
@nl
P2860
P356
P1476
Imbalance of ionic conductances contributes to diverse symptoms of demyelination.
@en
P2093
Steven A Prescott
Thomas M Bartol
P2860
P304
20602-20609
P356
10.1073/PNAS.1013798107
P407
P577
2010-10-25T00:00:00Z