bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS. - Wikidata - wikimedia - TriplyDB

bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

http://www.wikidata.org/entity/Q34377321

about

Aberrant DNA methylation of OLIG1, a novel prognostic factor in non-small cell lung cancer An Id-like molecule, HHM, is a synexpression group-restricted regulator of TGF-beta signalling Development of NG2 neural progenitor cells requires Olig gene function Myelin transcription factor 1 (Myt1) expression in demyelinated lesions of rodent and human CNS Myelin gene regulatory factor is a critical transcriptional regulator required for CNS myelination Novel Olig 1-coding variants and susceptibility to multiple sclerosis Systematic Review of Pharmacological Properties of the Oligodendrocyte Lineage Glial development: the crossroads of regeneration and repair in the CNS Human embryonic stem cell-derived oligodendrocytes: protocols and perspectives Gain of Olig2 function in oligodendrocyte progenitors promotes remyelination.Intravital assessment of myelin molecular order with polarimetric multiphoton microscopy Role of NG2 expressing cells in addiction: a new approach for an old problem The oligodendrocyte-specific G protein-coupled receptor GPR17 is a cell-intrinsic timer of myelination In vivo analysis of quiescent adult neural stem cells responding to Sonic hedgehog Expression pattern of the transcription factor Olig2 in response to brain injuries: implications for neuronal repair.Olig2-regulated lineage-restricted pathway controls replication competence in neural stem cells and malignant glioma Paradoxical dysregulation of the neural stem cell pathway sonic hedgehog-Gli1 in autoimmune encephalomyelitis and multiple sclerosis Phosphorylation state of Olig2 regulates proliferation of neural progenitors Phosphorylation regulates OLIG2 cofactor choice and the motor neuron-oligodendrocyte fate switch A genome-wide screen for spatially restricted expression patterns identifies transcription factors that regulate glial development Hedgehog-regulated localization of Vax2 controls eye development Dysregulation of the Wnt pathway inhibits timely myelination and remyelination in the mammalian CNS Oligodendrocytes: biology and pathology Age-dependent epigenetic control of differentiation inhibitors is critical for remyelination efficiency Abnormal expression of TIP30 and arrested nucleocytoplasmic transport within oligodendrocyte precursor cells in multiple sclerosis CNS-resident glial progenitor/stem cells produce Schwann cells as well as oligodendrocytes during repair of CNS demyelination.Identification of OLIG2 as the most specific glioblastoma stem cell marker starting from comparative analysis of data from similar DNA chip microarray platforms.From fish to man: understanding endogenous remyelination in central nervous system demyelinating diseases.Correlations between Diffusion Tensor Imaging (DTI) and Magnetic Resonance Spectroscopy (1H MRS) in schizophrenic patients and normal controls.The recently identified P2Y-like receptor GPR17 is a sensor of brain damage and a new target for brain repair Effect of catalpol on remyelination through experimental autoimmune encephalomyelitis acting to promote Olig1 and Olig2 expressions in mice.Tcf7l2 is tightly controlled during myelin formation Tapping into the glial reservoir: cells committed to remaining uncommitted.Two-tier transcriptional control of oligodendrocyte differentiation.New developments in the treatment of optic neuritis.Delayed onset of experimental autoimmune encephalomyelitis in Olig1 deficient mice.IkappaB kinase 2 determines oligodendrocyte loss by non-cell-autonomous activation of NF-kappaB in the central nervous system The NG2 proteoglycan promotes oligodendrocyte progenitor proliferation and developmental myelination An amino terminal phosphorylation motif regulates intranuclear compartmentalization of Olig2 in neural progenitor cells.GAS6 enhances repair following cuprizone-induced demyelination.

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P2860

bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

http://www.wikidata.org/entity/Q34377321

description

2004 nî lūn-bûn

@nan

2004 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2004 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2004年の論文

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2004年論文

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2004年論文

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2004年論文

@zh-hk

2004年論文

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2004年論文

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2004年论文

@wuu

name

bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

@ast

bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

@en

bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

@nl

type

label

bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

@ast

bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

@en

bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

@nl

prefLabel

bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

@ast

bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

@en

bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

@nl

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SCIENCE.1103709

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bHLH transcription factor Olig1 is required to repair demyelinated lesions in the CNS.

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P2093

Cedric S Raine

http://www.w3.org/2001/XMLSchema#string

Chao Zhao

http://www.w3.org/2001/XMLSchema#string

Charles D Stiles

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David H Rowitch

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Heather A Arnett

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John A Alberta

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Robin J M Franklin

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Sheila R Plant

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Sovann Kaing

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Stephen P J Fancy

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2111-2115

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scholarly article

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10.1126/SCIENCE.1103709

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5704

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306

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2004-12-01T00:00:00Z

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15604411

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