Multistage carcinogenesis and the incidence of colorectal cancer. - Wikidata - wikimedia - TriplyDB

Multistage carcinogenesis and the incidence of colorectal cancer.

Multistage carcinogenesis and the incidence of colorectal cancer.

http://www.wikidata.org/entity/Q34381339

about

Genetic progression and the waiting time to cancer Comparative biology of mammalian telomeres: hypotheses on ancestral states and the roles of telomeres in longevity determination A multistage theory of age-specific acceleration in human mortality Numbers of mutations to different types of colorectal cancer Smoking: additional burden on aging and death Age-Period-Cohort approaches to back-calculation of cancer incidence rate Non-targeted effects of ionising radiation--implications for low dose risk.Cancer evolution: mathematical models and computational inference Trends in HPV cervical and seroprevalence and associations between oral and genital infection and serum antibodies in NHANES 2003-2012 Incremental benefits of screening colonoscopy over sigmoidoscopy in average-risk populations: a model-driven analysis.Colorectal cancer prevention: choosing the most effective population strategy using bio-mathematical approaches Toward an evolutionary model of cancer: Considering the mechanisms that govern the fate of somatic mutations Genomic instability and radiation risk in molecular pathways to colon cancer POPULATION GENETICS OF NEUTRAL MUTATIONS IN EXPONENTIALLY GROWING CANCER CELL POPULATIONS Age-specific incidence of cancer: Phases, transitions, and biological implications Calibrating disease progression models using population data: a critical precursor to policy development in cancer control Age-specific incidence data indicate four mutations are required for human testicular cancers.Parallel routes of human carcinoma development: implications of the age-specific incidence data Rate, molecular spectrum, and consequences of human mutation Does folic acid supplementation prevent or promote colorectal cancer? Results from model-based predictions.Exploring the recent trend in esophageal adenocarcinoma incidence and mortality using comparative simulation modeling.Effects of dual modified resistant indica rice starch on azoxymethane-induced incipient colon cancer in mice.Age-specific incidence of inherited versus sporadic cancers: a test of the multistage theory of carcinogenesis.Heuristic modeling of carcinogenesis for the population with dichotomous susceptibility to cancer: a pancreatic cancer example.Estimation of hazard functions in the log-linear age-period-cohort model: application to lung cancer risk associated with geographical area.Application of biomarkers in cancer risk management: evaluation from stochastic clonal evolutionary and dynamic system optimization points of view.Evolution in health and medicine Sackler colloquium: Somatic evolutionary genomics: mutations during development cause highly variable genetic mosaicism with risk of cancer and neurodegeneration.Cancer models, genomic instability and somatic cellular Darwinian evolution Noise-driven heterogeneity in the rate of genetic-variant generation as a basis for evolvability.A novel approach for analysis of the log-linear age-period-cohort model: application to lung cancer incidence.Risk of proximal colonic neoplasms in asymptomatic adults older than 50 years found to have distal hyperplastic polyps on routine colorectal cancer screening.Colorectal cancer incidence trends in the United States and United kingdom: evidence of right- to left-sided biological gradients with implications for screening.Number and size distribution of colorectal adenomas under the multistage clonal expansion model of cancer Weibull-like Model of Cancer Development in Aging.A heuristic solution of the identifiability problem of the age-period-cohort analysis of cancer occurrence: lung cancer example.Evolution of ibrutinib resistance in chronic lymphocytic leukemia (CLL)Evaluating the number of stages in development of squamous cell and adenocarcinomas across cancer sites using human population-based cancer modeling.Natural resistance to cancers: a Darwinian hypothesis to explain Peto's paradox.Chemical compounds from anthropogenic environment and immune evasion mechanisms: potential interactions Breast cancer incidence in black and white women stratified by estrogen and progesterone receptor statuses.

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P2860

Multistage carcinogenesis and the incidence of colorectal cancer.

http://www.wikidata.org/entity/Q34381339

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2002 nî lūn-bûn

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Multistage carcinogenesis and the incidence of colorectal cancer.

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Multistage carcinogenesis and the incidence of colorectal cancer.

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Multistage carcinogenesis and the incidence of colorectal cancer.

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Multistage carcinogenesis and the incidence of colorectal cancer.

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Multistage carcinogenesis and the incidence of colorectal cancer.

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Multistage carcinogenesis and the incidence of colorectal cancer.

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Multistage carcinogenesis and the incidence of colorectal cancer.

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Multistage carcinogenesis and the incidence of colorectal cancer.

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Multistage carcinogenesis and the incidence of colorectal cancer.

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Proceedings of the National Academy of Sciences of the United States of America

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Multistage carcinogenesis and the incidence of colorectal cancer.

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E Georg Luebeck

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Suresh H Moolgavkar

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P2860

The Hallmarks of Cancer

A new theory on cancer-inducing mechanism

The age distribution of cancer and a multi-stage theory of carcinogenesis

Mutation and cancer: statistical study of retinoblastoma

A genetic model for colorectal tumorigenesis

Cancer-susceptibility genes. Gatekeepers and caretakers

Genetic mapping of a locus predisposing to human colorectal cancer

Landscaping the cancer terrain

Non-steroidal anti-inflammatory drugs with activity against either cyclooxygenase 1 or cyclooxygenase 2 inhibit colorectal cancer in a DMH rodent model by inducing apoptosis and inhibiting cell proliferation

Stem cells: attributes, cycles, spirals, pitfalls and uncertainties. Lessons for and from the crypt

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15095-15100

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10.1073/PNAS.222118199

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colorectal cancer

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