Hypoxia-reoxygenation induces premature senescence in FA bone marrow hematopoietic cells.
about
Defective mitochondrial peroxiredoxin-3 results in sensitivity to oxidative stress in Fanconi anemiaSequencing of 50 human exomes reveals adaptation to high altitudeOxidative stress, bone marrow failure, and genome instability in hematopoietic stem cellsPathways for repairing and tolerating the spectrum of oxidative DNA lesionsMouse models of Fanconi anemiaCytokinesis failure occurs in Fanconi anemia pathway-deficient murine and human bone marrow hematopoietic cellsHypoxia reoxygenation induces premature senescence in neonatal SD rat cardiomyocytes.Senescent cardiac fibroblast is critical for cardiac fibrosis after myocardial infarction.Bone marrow failure in Fanconi anemia is triggered by an exacerbated p53/p21 DNA damage response that impairs hematopoietic stem and progenitor cells.TNF-alpha induces leukemic clonal evolution ex vivo in Fanconi anemia group C murine stem cells.TSC-mTOR maintains quiescence and function of hematopoietic stem cells by repressing mitochondrial biogenesis and reactive oxygen speciesContributions of DNA interstrand cross-links to aging of cells and organisms.Reactive Oxygen Species Limit the Ability of Bone Marrow Stromal Cells to Support Hematopoietic Reconstitution in Aging Mice.Oxidative stress in Fanconi anemia hematopoiesis and disease progression.Fanconi anaemia: genetics, molecular biology, and cancer – implications for clinical management in children and adults.How I manage patients with Fanconi anaemia.Inflammatory ROS promote and cooperate with the Fanconi anemia mutation for hematopoietic senescence.Nucleophosmin regulates cell cycle progression and stress response in hematopoietic stem/progenitor cells.Positive correlation between insulin resistance and iron overload-induced oxidative stress in patients with fanconi anemia (FA)- and non-FA-related bone marrow failure: The results of a multicenter study.Sulfated Polysaccharide Isolated from the Sea Cucumber Stichopus japonicus Against PC12 Hypoxia/Reoxygenation Injury by Inhibition of the MAPK Signaling Pathway.
P2860
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P2860
Hypoxia-reoxygenation induces premature senescence in FA bone marrow hematopoietic cells.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մարտին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Hypoxia-reoxygenation induces premature senescence in FA bone marrow hematopoietic cells.
@ast
Hypoxia-reoxygenation induces premature senescence in FA bone marrow hematopoietic cells.
@en
Hypoxia-reoxygenation induces premature senescence in FA bone marrow hematopoietic cells.
@nl
type
label
Hypoxia-reoxygenation induces premature senescence in FA bone marrow hematopoietic cells.
@ast
Hypoxia-reoxygenation induces premature senescence in FA bone marrow hematopoietic cells.
@en
Hypoxia-reoxygenation induces premature senescence in FA bone marrow hematopoietic cells.
@nl
prefLabel
Hypoxia-reoxygenation induces premature senescence in FA bone marrow hematopoietic cells.
@ast
Hypoxia-reoxygenation induces premature senescence in FA bone marrow hematopoietic cells.
@en
Hypoxia-reoxygenation induces premature senescence in FA bone marrow hematopoietic cells.
@nl
P2093
P1433
P1476
Hypoxia-reoxygenation induces premature senescence in FA bone marrow hematopoietic cells.
@en
P2093
Daniel P Sejas
Qishen Pang
Xiaoling Zhang
P356
10.1182/BLOOD-2004-08-3033
P407
P577
2005-03-15T00:00:00Z