Response of base excision repair enzymes to complex DNA lesions.
about
NEIL1 excises 3' end proximal oxidative DNA lesions resistant to cleavage by NTH1 and OGG1.NMR Solution Structures of Bistranded Abasic Site Lesions in DNA † ‡NMR Solution Structures of Clustered Abasic Site Lesions in DNA: Structural Differences between 3′-Staggered (−3) and 5′-Staggered (+3) Bistranded LesionsA network of enzymes involved in repair of oxidative DNA damage in Neisseria meningitidisApex1 can cleave complex clustered DNA lesions in cells.Quantification of the 2-deoxyribonolactone and nucleoside 5'-aldehyde products of 2-deoxyribose oxidation in DNA and cells by isotope-dilution gas chromatography mass spectrometry: differential effects of gamma-radiation and Fe2+-EDTAHigh efficiency detection of bi-stranded abasic clusters in gamma-irradiated DNA by putrescineOne-electron oxidation of DNA by ionizing radiation: competition between base-to-base hole-transfer and hole-trapping.DEMETER DNA glycosylase establishes MEDEA polycomb gene self-imprinting by allele-specific demethylation.Molecular analysis of base damage clustering associated with a site-specific radiation-induced DNA double-strand break.The Werner syndrome protein stimulates DNA polymerase beta strand displacement synthesis via its helicase activity.Oxidative DNA damage induced by copper and hydrogen peroxide promotes CG-->TT tandem mutations at methylated CpG dinucleotides in nucleotide excision repair-deficient cells.Accumulation of abasic sites induces genomic instability in normal human gastric epithelial cells during Helicobacter pylori infection.DNA polymerase delta-dependent repair of DNA single strand breaks containing 3'-end proximal lesions.Interplay between DNA N-glycosylases/AP lyases at multiply damaged sites and biological consequences.Base excision repair processing of abasic site/single-strand break lesions within clustered damage sites associated with XRCC1 deficiency.Analysis of gene expression in normal and cancer cells exposed to gamma-radiation.Elucidating DNA damage and repair processes by independently generating reactive and metastable intermediates.DNA tandem lesion repair by strand displacement synthesis and nucleotide excision repair.Which DNA damage is likely to be relevant in hormetic responses?DNA repair of clustered lesions in mammalian cells: involvement of non-homologous end-joining.On the chemical yield of base lesions, strand breaks, and clustered damage generated in plasmid DNA by the direct effect of X rays.Closely opposed apurinic/apyrimidinic sites are converted to double strand breaks in Escherichia coli even in the absence of exonuclease III, endonuclease IV, nucleotide excision repair and AP lyase cleavage.Hierarchy of lesion processing governs the repair, double-strand break formation and mutability of three-lesion clustered DNA damage.Repair of clustered uracil DNA damages in Escherichia coli.Two clustered 8-oxo-7,8-dihydroguanine (8-oxodG) lesions increase the point mutation frequency of 8-oxodG, but do not result in double strand breaks or deletions in Escherichia coli.Enhanced mutagenic potential of 8-oxo-7,8-dihydroguanine when present within a clustered DNA damage siteProcessing of thymine glycol in a clustered DNA damage site: mutagenic or cytotoxic.Role of Ape1 and base excision repair in the radioresponse and heat-radiosensitization of HeLa Cells.Energetic coupling between clustered lesions modulated by intervening triplet repeat bulge loops: allosteric implications for DNA repair and triplet repeat expansion.Energy crosstalk between DNA lesions: implications for allosteric coupling of DNA repair and triplet repeat expansion pathways.GC/MS methods to quantify the 2-deoxypentos-4-ulose and 3'-phosphoglycolate pathways of 4' oxidation of 2-deoxyribose in DNA: application to DNA damage produced by gamma radiation and bleomycin.Repair of clustered DNA lesions. Sequence-specific inhibition of long-patch base excision repair be 8-oxoguanine.Mode of inhibition of short-patch base excision repair by thymine glycol within clustered DNA lesions.Induction of single strand breaks, and base lesions in plasmid DNA films induced by carbon, nitrogen, and oxygen KLL Auger process.Characterization of a complex 125I-induced DNA double-strand break: implications for repair.Enhanced Cleavage at Abasic Sites within Clustered Lesions in Nucleosome Core ParticlesChemistry of Inflammation and DNA Damage: Biological Impact of Reactive Nitrogen Species
P2860
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P2860
Response of base excision repair enzymes to complex DNA lesions.
description
2001 nî lūn-bûn
@nan
2001 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Response of base excision repair enzymes to complex DNA lesions.
@ast
Response of base excision repair enzymes to complex DNA lesions.
@en
Response of base excision repair enzymes to complex DNA lesions.
@nl
type
label
Response of base excision repair enzymes to complex DNA lesions.
@ast
Response of base excision repair enzymes to complex DNA lesions.
@en
Response of base excision repair enzymes to complex DNA lesions.
@nl
prefLabel
Response of base excision repair enzymes to complex DNA lesions.
@ast
Response of base excision repair enzymes to complex DNA lesions.
@en
Response of base excision repair enzymes to complex DNA lesions.
@nl
P2093
P1433
P1476
Response of base excision repair enzymes to complex DNA lesions.
@en
P2093
Britten RA
Chaudhry MA
Rasouli-Nia A
Weinfeld M
P304
P356
10.1667/0033-7587(2001)156[0584:ROBERE]2.0.CO;2
P433
P577
2001-11-01T00:00:00Z