Gap junctions play a role in the 'bystander effect' of the herpes simplex virus thymidine kinase/ganciclovir system in vitro.
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Therapeutic efficacy of human hepatocyte transplantation in a SCID/uPA mouse model with inducible liver diseaseTanshinone IIA increases the bystander effect of herpes simplex virus thymidine kinase/ganciclovir gene therapy via enhanced gap junctional intercellular communicationTetramethylpyrazine suppresses transient oxygen-glucose deprivation-induced connexin32 expression and cell apoptosis via the ERK1/2 and p38 MAPK pathway in cultured hippocampal neuronsOsteocyte apoptosis is required for production of osteoclastogenic signals following bone fatigue in vivo.Neighbor effects of neurons bearing protective transgenesCross-talk between pulmonary injury, oxidant stress, and gap junctional communication.Experimental approaches for the treatment of malignant gliomas.Gene therapy and targeted toxins for glioma.The Anti-Tumor Effects of Adipose Tissue Mesenchymal Stem Cell Transduced with HSV-Tk Gene on U-87-Driven Brain Tumor.Gap junction- and hemichannel-independent actions of connexinsEnforced effect of tk-MCP-1 fusion gene in ovarian cancerThe roles of macrophages and nitric oxide in interleukin-3-enhanced HSV-Sr39tk-mediated prodrug therapyHighly efficient electro-gene therapy of solid tumor by using an expression plasmid for the herpes simplex virus thymidine kinase gene.Gap junctions and cancer: communicating for 50 years.Enzymes to die for: exploiting nucleotide metabolizing enzymes for cancer gene therapy.Chapter seven--Cancer treatment with gene therapy and radiation therapy.Gap junctions and Bystander Effects: Good Samaritans and executioners.Exceptionally potent anti-tumor bystander activity of an scFv:sTRAIL fusion protein with specificity for EGP2 toward target antigen-negative tumor cells.The Microvascular Gap Junction Channel: A Route to Deliver MicroRNAs for Neurological Disease TreatmentThe promise and reality of cancer gene therapy.Sequential histopathological changes in vivo after suicide gene therapy of gastric cancer induced by N-methyl-N'-nitro-N-nitrosoguanidine in rats.The impact of γ-irradiation on the induction of bystander killing by genetically engineered ovarian tumor cells: implications for clinical use.Gene therapy and targeted toxins for glioma
P2860
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P2860
Gap junctions play a role in the 'bystander effect' of the herpes simplex virus thymidine kinase/ganciclovir system in vitro.
description
1996 nî lūn-bûn
@nan
1996 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
name
Gap junctions play a role in t ...... e/ganciclovir system in vitro.
@ast
Gap junctions play a role in t ...... e/ganciclovir system in vitro.
@en
Gap junctions play a role in t ...... e/ganciclovir system in vitro.
@nl
type
label
Gap junctions play a role in t ...... e/ganciclovir system in vitro.
@ast
Gap junctions play a role in t ...... e/ganciclovir system in vitro.
@en
Gap junctions play a role in t ...... e/ganciclovir system in vitro.
@nl
prefLabel
Gap junctions play a role in t ...... e/ganciclovir system in vitro.
@ast
Gap junctions play a role in t ...... e/ganciclovir system in vitro.
@en
Gap junctions play a role in t ...... e/ganciclovir system in vitro.
@nl
P2093
P1433
P1476
Gap junctions play a role in t ...... e/ganciclovir system in vitro.
@en
P2093
Albelda SM
Elshami AA
Fishman GI
Kucharczuk JC
Saavedra A
P577
1996-01-01T00:00:00Z