about
Viruses and neurodegenerationNetwork analysis reveals common host protein/s modulating pathogenesis of neurotropic virusesZika virus infection reprograms global transcription of host cells to allow sustained infectionEnergetic cost of building a virus.Quantitative proteomics of Spodoptera frugiperda cells during growth and baculovirus infection.Proteomics analysis of differentially expressed proteins in chicken trachea and kidney after infection with the highly virulent and attenuated coronavirus infectious bronchitis virus in vivo.Mitochondrial bioenergetic alterations in mouse neuroblastoma cells infected with Sindbis virus: implications to viral replication and neuronal death.Determining host metabolic limitations on viral replication via integrated modeling and experimental perturbation.HIV-1 pathogenicity and virion production are dependent on the metabolic phenotype of activated CD4+ T cellsDichloroacetate blocks aerobic glycolytic adaptation to attenuated measles virus and promotes viral replication leading to enhanced oncolysis in glioblastoma.Population dynamics of a Salmonella lytic phage and its host: implications of the host bacterial growth rate in modellingMeta-Analysis of Dengue Severity during Infection by Different Dengue Virus Serotypes in Primary and Secondary InfectionsBioenergetics of murine lungs infected with respiratory syncytial virus.Activity increase in respiratory chain complexes by rubella virus with marginal induction of oxidative stress.Perspectives in metabolic engineering: understanding cellular regulation towards the control of metabolic routes.The role of host cell physiology in the productivity of the baculovirus-insect cell system: Fluxome analysis of Trichoplusia ni and Spodoptera frugiperda cell lines.Mitochondrial dysfunction in rabies virus infection of neurons.Metabolic profiling of insect cell lines: Unveiling cell line determinants behind system's productivity.Pseudomonas predators: understanding and exploiting phage-host interactions.Development and evaluation of a host-targeted antiviral that abrogates herpes simplex virus replication through modulation of arginine-associated metabolic pathways.Regulation of the Type I-F CRISPR-Cas system by CRP-cAMP and GalM controls spacer acquisition and interference.Toward system-level understanding of baculovirus-host cell interactions: from molecular fundamental studies to large-scale proteomics approaches.Elucidation of complexity and prediction of interactions in microbial communities.Integrated human-virus metabolic stoichiometric modelling predicts host-based antiviral targets against Chikungunya, Dengue and Zika viruses
P2860
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P2860
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
The virus as metabolic engineer.
@ast
The virus as metabolic engineer.
@en
The virus as metabolic engineer.
@nl
type
label
The virus as metabolic engineer.
@ast
The virus as metabolic engineer.
@en
The virus as metabolic engineer.
@nl
prefLabel
The virus as metabolic engineer.
@ast
The virus as metabolic engineer.
@en
The virus as metabolic engineer.
@nl
P2093
P2860
P356
P1476
The virus as metabolic engineer.
@en
P2093
Elsa W Birch
Markus W Covert
Miriam V Gutschow
Nathaniel D Maynard
P2860
P304
P356
10.1002/BIOT.201000080
P577
2010-07-01T00:00:00Z