Pinocembrin protects against β-amyloid-induced toxicity in neurons through inhibiting receptor for advanced glycation end products (RAGE)-independent signaling pathways and regulating mitochondrion-mediated apoptosis.
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Cerebrospinal fluid biomarkers of neurovascular dysfunction in mild dementia and Alzheimer's diseaseThe Natural Flavonoid Pinocembrin: Molecular Targets and Potential Therapeutic ApplicationsPinocembrin protects hemorrhagic brain primarily by inhibiting toll-like receptor 4 and reducing M1 phenotype microglia.Methyl salicylate lactoside inhibits inflammatory response of fibroblast-like synoviocytes and joint destruction in collagen-induced arthritis in micePinocembrin protects human brain microvascular endothelial cells against fibrillar amyloid-β(1-40) injury by suppressing the MAPK/NF-κB inflammatory pathwaysRadix Achyranthis Bidentatae improves learning and memory capabilities in ovariectomized rats.A specific RAGE-binding peptide biopanning from phage display random peptide library that ameliorates symptoms in amyloid β peptide-mediated neuronal disorder.Quercetin protects human brain microvascular endothelial cells from fibrillar β-amyloid1-40-induced toxicityDL0410, a novel dual cholinesterase inhibitor, protects mouse brains against Aβ-induced neuronal damage via the Akt/JNK signaling pathway.RAGE regulation and signaling in inflammation and beyond.Molecular strategies to prevent, inhibit, and degrade advanced glycoxidation and advanced lipoxidation end products.Flavonoids in modulation of cell survival signalling pathways.Investigation of phosphoproteome in RAGE signaling.Prevention of protein glycation by natural compounds.Pinocembrin Attenuates Mitochondrial Dysfunction in Human Neuroblastoma SH-SY5Y Cells Exposed to Methylglyoxal: Role for the Erk1/2-Nrf2 Signaling Pathway.Biological Properties and Therapeutic Applications of Propolis.Pinocembrin attenuates 6-OHDA-induced neuronal cell death through Nrf2/ARE pathway in SH-SY5Y cells.Pinocembrin protects SH-SY5Y cells against MPP+-induced neurotoxicity through the mitochondrial apoptotic pathway.Can Co-Activation of Nrf2 and Neurotrophic Signaling Pathway Slow Alzheimer's Disease?Mechanistic comparison of current pharmacological treatments and novel phytochemicals to target amyloid peptides in Alzheimer's and neurodegenerative diseases.Chemical composition and disruption of quorum sensing signaling in geographically diverse United States propolis.Pinocembrin suppresses TGF-β1-induced epithelial-mesenchymal transition and metastasis of human Y-79 retinoblastoma cells through inactivating αvβ3 integrin/FAK/p38α signaling pathway.Scutellaria baicalensis stem-leaf total flavonoid reduces neuronal apoptosis induced by amyloid beta-peptide (25-35).Pinocembrin Suppresses H2O2-Induced Mitochondrial Dysfunction by a Mechanism Dependent on the Nrf2/HO-1 Axis in SH-SY5Y Cells.l-3-n-Butylphthalide attenuates β-amyloid-induced toxicity in neuroblastoma SH-SY5Y cells through regulating mitochondrion-mediated apoptosis and MAPK signaling.Protection of mice against lipopolysaccharide-induced endotoxic shock by pinocembrin is correlated with regulation of cytokine secretion.Recent Advances in the Inhibition of p38 MAPK as a Potential Strategy for the Treatment of Alzheimer's Disease.New propolis type from north-east Brazil: chemical composition, antioxidant activity and botanical origin.p38 MAPK-MK2 pathway regulates the heat-stress-induced accumulation of reactive oxygen species that mediates apoptotic cell death in glial cells.Hydroalcoholic extract of red propolis promotes functional recovery and axon repair after sciatic nerve injury in rats.Methyl Salicylate Lactoside Protects Neurons Ameliorating Cognitive Disorder Through Inhibiting Amyloid Beta-Induced Neuroinflammatory Response in Alzheimer's Disease.Validation of RetroPath, a computer-aided design tool for metabolic pathway engineering.No effects without causes: the Iron Dysregulation and Dormant Microbes hypothesis for chronic, inflammatory diseases.Inhibitory effect of propolis on the development of AA amyloidosis.The role of neutrophil granule proteins in neuroinflammation and Alzheimer's disease
P2860
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P2860
Pinocembrin protects against β-amyloid-induced toxicity in neurons through inhibiting receptor for advanced glycation end products (RAGE)-independent signaling pathways and regulating mitochondrion-mediated apoptosis.
description
2012 nî lūn-bûn
@nan
2012 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Pinocembrin protects against β ...... ochondrion-mediated apoptosis.
@ast
Pinocembrin protects against β ...... ochondrion-mediated apoptosis.
@en
Pinocembrin protects against β ...... dvanced glycation end products
@nl
type
label
Pinocembrin protects against β ...... ochondrion-mediated apoptosis.
@ast
Pinocembrin protects against β ...... ochondrion-mediated apoptosis.
@en
Pinocembrin protects against β ...... dvanced glycation end products
@nl
prefLabel
Pinocembrin protects against β ...... ochondrion-mediated apoptosis.
@ast
Pinocembrin protects against β ...... ochondrion-mediated apoptosis.
@en
Pinocembrin protects against β ...... dvanced glycation end products
@nl
P2093
P2860
P356
P1433
P1476
Pinocembrin protects against β ...... ochondrion-mediated apoptosis.
@en
P2093
Cai-Xia Wu
Guan-Hua Du
Tian-Tai Zhang
P2860
P2888
P356
10.1186/1741-7015-10-105
P407
P577
2012-09-18T00:00:00Z
P5875
P6179
1017434576