Dural afferents express acid-sensing ion channels: a role for decreased meningeal pH in migraine headache. - Wikidata - wikimedia - TriplyDB

Dural afferents express acid-sensing ion channels: a role for decreased meningeal pH in migraine headache.

Dural afferents express acid-sensing ion channels: a role for decreased meningeal pH in migraine headache.

http://www.wikidata.org/entity/Q34419829

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Acid-sensing ion channels in pain and disease.ASICs as therapeutic targets for migraine Intravital imaging of a massive lymphocyte response in the cortical dura of mice after peripheral infection by trypanosomes Meningeal norepinephrine produces headache behaviors in rats via actions both on dural afferents and fibroblasts Muscle IL1β Drives Ischemic Myalgia via ASIC3-Mediated Sensory Neuron Sensitization Extracranial injections of botulinum neurotoxin type A inhibit intracranial meningeal nociceptors' responses to stimulation of TRPV1 and TRPA1 channels: Are we getting closer to solving this puzzle?Dural fibroblasts play a potential role in headache pathophysiology.The sensory innervation of the calvarial periosteum is nociceptive and contributes to headache-like behavior Selective inhibition of meningeal nociceptors by botulinum neurotoxin type A: therapeutic implications for migraine and other pains.Targeting TRP channels for novel migraine therapeutics.Comprehensive RNA-Seq expression analysis of sensory ganglia with a focus on ion channels and GPCRs in Trigeminal ganglia Sensitization of dural afferents underlies migraine-related behavior following meningeal application of interleukin-6 (IL-6).Sumatriptan inhibits TRPV1 channels in trigeminal neurons.ASIC3 channels in multimodal sensory perception Activation of TRPA1 on dural afferents: a potential mechanism of headache pain.Meningeal transient receptor potential channel M8 activation causes cutaneous facial and hindpaw allodynia in a preclinical rodent model of headache Protective Effects of Alisma orientale Extract against Hepatic Steatosis via Inhibition of Endoplasmic Reticulum Stress.Expression of the transient receptor potential channels TRPV1, TRPA1 and TRPM8 in mouse trigeminal primary afferent neurons innervating the dura.Mast Cell-Mediated Mechanisms of Nociception.Two mechanisms involved in trigeminal CGRP release: implications for migraine treatment.Activation of TRPV4 on dural afferents produces headache-related behavior in a preclinical rat model.Cortical spreading depression as a target for anti-migraine agents.Functional analysis of a migraine-associated TRESK K+ channel mutation.pH-evoked dural afferent signaling is mediated by ASIC3 and is sensitized by mast cell mediators.CGRP and migraine: could PACAP play a role too?Gastrodin protects against chronic inflammatory pain by inhibiting spinal synaptic potentiation.Ion channels and migraine ASICs and neuropeptides.Diverse Physiological Roles of Calcitonin Gene-Related Peptide in Migraine Pathology: Modulation of Neuronal-Glial-Immune Cells to Promote Peripheral and Central Sensitization.Dural stimulation in rats causes brain-derived neurotrophic factor-dependent priming to subthreshold stimuli including a migraine trigger.Acid-sensing ion channels contribute to the increase in vesicular release from SH-SY5Y cells stimulated by extracellular protons.Gene-based pleiotropy across migraine with aura and migraine without aura patient groups.The effects of acute and preventive migraine therapies in a mouse model of chronic migraine.Characterization of a mouse model of headache Parthenolide inhibits nociception and neurogenic vasodilatation in the trigeminovascular system by targeting the TRPA1 channel.Acid-Sensing Ion Channels as Potential Therapeutic Targets in Neurodegeneration and Neuroinflammation.Cranial dural permeability of inflammatory nociceptive mediators: Potential implications for animal models of migraine.From Mechanism to Cure: Renewing the Goal to Eliminate the Disease of Pain.Acid-sensing ion channels promote the inflammation and migration of cultured rat microglia.NS383 Selectively Inhibits Acid-Sensing Ion Channels Containing 1a and 3 Subunits to Reverse Inflammatory and Neuropathic Hyperalgesia in Rats.

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Dural afferents express acid-sensing ion channels: a role for decreased meningeal pH in migraine headache.

http://www.wikidata.org/entity/Q34419829

description

2010 nî lūn-bûn

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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Dural afferents express acid-s ...... ngeal pH in migraine headache.

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Dural afferents express acid-s ...... ngeal pH in migraine headache.

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Dural afferents express acid-s ...... ngeal pH in migraine headache.

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type

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Dural afferents express acid-s ...... ngeal pH in migraine headache.

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Dural afferents express acid-s ...... ngeal pH in migraine headache.

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Dural afferents express acid-s ...... ngeal pH in migraine headache.

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Dural afferents express acid-s ...... ngeal pH in migraine headache.

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Dural afferents express acid-s ...... ngeal pH in migraine headache.

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Dural afferents express acid-s ...... ngeal pH in migraine headache.

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Dural afferents express acid-s ...... ngeal pH in migraine headache.

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Frank Porreca

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Gregory Dussor

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Jin Yan

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Milena De Felice

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Rebecca M Edelmayer

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Xiaomei Wei

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P2860

Acid-sensing ion channel 3 matches the acid-gated current in cardiac ischemia-sensing neurons

Contrasting phenotypes of putative proprioceptive and nociceptive trigeminal neurons innervating jaw muscle in rat

ASIC3, an acid-sensing ion channel, is expressed in metaboreceptive sensory neurons

Heteromultimers of DEG/ENaC subunits form H+-gated channels in mouse sensory neurons

Deconstructing migraine headache into peripheral and central sensitization.

Sensitization of meningeal sensory neurons and the origin of headaches.

Migraine--current understanding and treatment.

Migraine pain, meningeal inflammation, and mast cells.

Different contributions of ASIC channels 1a, 2, and 3 in gastrointestinal mechanosensory function

Mast cell degranulation activates a pain pathway underlying migraine headache.

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migraine disorder

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