An inflammation loop orchestrated by S100A9 and calprotectin is critical for development of arthritis. - Wikidata - wikimedia - TriplyDB

An inflammation loop orchestrated by S100A9 and calprotectin is critical for development of arthritis.

An inflammation loop orchestrated by S100A9 and calprotectin is critical for development of arthritis.

http://www.wikidata.org/entity/Q34429165

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CD16 is indispensable for antibody-dependent cellular cytotoxicity by human monocytes Circulating Autoantibodies in Age-Related Macular Degeneration Recognize Human Macular Tissue Antigens Implicated in Autophagy, Immunomodulation, and Protection from Oxidative Stress and Apoptosis S100A8/A9 and S100A9 reduce acute lung injury.Proinflammatory effects of S100A8/A9 via TLR4 and RAGE signaling pathways in BV-2 microglial cells Alarmin S100A8/S100A9 as a biomarker for molecular imaging of local inflammatory activity.The pro-apoptotic and pro-inflammatory effects of calprotectin on human periodontal ligament cells.S100A8 and S100A9 induce cytokine expression and regulate the NLRP3 inflammasome via ROS-dependent activation of NF-κB(1.).DAMP molecule S100A9 acts as a molecular pattern to enhance inflammation during influenza A virus infection: role of DDX21-TRIF-TLR4-MyD88 pathway.S100A9 induced inflammatory responses are mediated by distinct damage associated molecular patterns (DAMP) receptors in vitro and in vivo.Lymphocyte gene expression signatures from patients and mouse models of hereditary hemochromatosis reveal a function of HFE as a negative regulator of CD8+ T-lymphocyte activation and differentiation in vivo S100A8/A9 (calprotectin) is critical for development of glomerulonephritis and promotes inflammatory leukocyte-renal cell interactions.Calprotectin Increases the Activity of the SaeRS Two Component System and Murine Mortality during Staphylococcus aureus Infections.CD14 Is a Co-Receptor for TLR4 in the S100A9-Induced Pro-Inflammatory Response in Monocytes Elevated S100A9 expression in tumor stroma functions as an early recurrence marker for early-stage oral cancer patients through increased tumor cell invasion, angiogenesis, macrophage recruitment and interleukin-6 production.TLR4 Endogenous Ligand S100A8/A9 Levels in Adult-Onset Still's Disease and Their Association with Disease Activity and Clinical Manifestations S100A8/A9 proteins mediate neutrophilic inflammation and lung pathology during tuberculosis Role of neutrophils in systemic autoimmune diseases Matrix metalloproteinase processing of signaling molecules to regulate inflammation.Calprotectin levels in patients with rheumatoid arthritis to assess and association with exercise treatment.S100-alarmins: potential therapeutic targets for arthritis.Calprotectin Induces IL-6 and MCP-1 Production via Toll-Like Receptor 4 Signaling in Human Gingival Fibroblasts.Myeloid Populations in Systemic Autoimmune Diseases.Calprotectin in rheumatic diseases.A specific anti-citrullinated protein antibody profile identifies a group of rheumatoid arthritis patients with a toll-like receptor 4-mediated disease.Proinflammatory effects and mechanisms of calprotectin on human gingival fibroblasts.Serum Calprotectin Versus Acute-Phase Reactants in the Discrimination of Inflammatory Disease Activity in Rheumatoid Arthritis Patients Receiving Tumor Necrosis Factor Inhibitors.Extracellular Matrix Metalloproteinase Inducer EMMPRIN (CD147) in Cardiovascular Disease.Discovery and confirmation of a protein biomarker panel with potential to predict response to biological therapy in psoriatic arthritis.Inflammatory Changes in Bone Marrow Microenvironment Associated with Declining B Lymphopoiesis.Coevolution of the Toll-Like Receptor 4 Complex with Calgranulins and Lipopolysaccharide.Secretion of the Phosphorylated Form of S100A9 from Neutrophils Is Essential for the Proinflammatory Functions of Extracellular S100A8/A9.Paquinimod prevents development of diabetes in the non-obese diabetic (NOD) mouse.S100A8/A9 in Inflammation.Neutrophils and the S100A9 protein critically regulate granuloma formation

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An inflammation loop orchestrated by S100A9 and calprotectin is critical for development of arthritis.

http://www.wikidata.org/entity/Q34429165

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2012 nî lūn-bûn

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2012 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2012 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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An inflammation loop orchestra ...... for development of arthritis.

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An inflammation loop orchestra ...... for development of arthritis.

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An inflammation loop orchestra ...... for development of arthritis.

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An inflammation loop orchestra ...... for development of arthritis.

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An inflammation loop orchestra ...... for development of arthritis.

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An inflammation loop orchestra ...... for development of arthritis.

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Annabelle Cesaro

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Audrey Plante

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Mélanie R Tardif

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Nadia Anceriz

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Nathalie Pagé

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P2860

Proinflammatory activities of S100: proteins S100A8, S100A9, and S100A8/A9 induce neutrophil chemotaxis and adhesion

Two calcium-binding proteins in infiltrate macrophages of rheumatoid arthritis

Neutrophil derived human S100A12 (EN-RAGE) is strongly expressed during chronic active inflammatory bowel disease

Fibroblast biology. Signals targeting the synovial fibroblast in arthritis.

S100A9/S100A8: Myeloid representatives of the S100 protein family as prominent players in innate immunity

The human S100 protein MRP-14 is a novel activator of the beta 2 integrin Mac-1 on neutrophils

S100A8 and S100A9 activate MAP kinase and NF-kappaB signaling pathways and trigger translocation of RAGE in human prostate cancer cells

Synovial fluid proteomic fingerprint: S100A8, S100A9 and S100A12 proteins discriminate rheumatoid arthritis from other inflammatory joint diseases.

Proinflammatory S100 proteins in arthritis and autoimmune disease.

The S100A8/A9 heterodimer amplifies proinflammatory cytokine production by macrophages via activation of nuclear factor kappa B and p38 mitogen-activated protein kinase in rheumatoid arthritis.

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