Critical role of macrophages and their activation via MyD88-NFκB signaling in lung innate immunity to Mycoplasma pneumoniae - Wikidata - wikimedia - TriplyDB

Critical role of macrophages and their activation via MyD88-NFκB signaling in lung innate immunity to Mycoplasma pneumoniae

Critical role of macrophages and their activation via MyD88-NFκB signaling in lung innate immunity to Mycoplasma pneumoniae

http://www.wikidata.org/entity/Q34437813

about

CD11c(+)/CD11b(+) cells are critical for organic dust-elicited murine lung inflammation Housing conditions modulate the severity of Mycoplasma pulmonis infection in mice deficient in class A scavenger receptor Heat shock factor 1 protects against lung mycoplasma pneumoniae infection in mice.Vaccination of BALB/c mice with an avirulent Mycoplasma pneumoniae P30 mutant results in disease exacerbation upon challenge with a virulent strain.Role of the Mycoplasma pneumoniae/Interleukin-8/Neutrophil Axis in the Pathogenesis of Pneumonia Nitric oxide suppresses LPS-induced inflammation in a mouse asthma model by attenuating the interaction of IKK and Hsp90.Experimental and natural infections in MyD88- and IRAK-4-deficient mice and humans Insights into the pathogenesis of Mycoplasma pneumoniae (Review).Mycoplasma pneumoniae from the Respiratory Tract and Beyond.Calpastatin upregulation in Mycoplasma hyorhinis-infected cells is promoted by the mycoplasma lipoproteins via the NF-κB pathway.Inhaled corticosteroid influence toll like receptor 2 expression in induced sputum from patients with COPD.ATF3 inhibits the inflammation induced by Mycoplasma pneumonia in vitro and in vivo.Sulfur compounds block MCP-1 production by Mycoplasma fermentans-infected macrophages through NF-κB inhibition.Mpn491, a secreted nuclease of Mycoplasma pneumoniae, plays a critical role in evading killing by neutrophil extracellular traps.Characteristics of alveolar macrophages from murine models of OVA-induced allergic airway inflammation and LPS-induced acute airway inflammation.Interleukin 17A as a good predictor of the severity of Mycoplasma pneumoniae pneumonia in children.TIPE2 negatively regulates mycoplasma pneumonia-triggered immune response via MAPK signaling pathway.NLRP3 Is a Critical Regulator of Inflammation and Innate Immune Cell Response during Mycoplasma pneumoniae Infection.Post-injury and resolution response to repetitive inhalation exposure to agricultural organic dust in mice.

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Critical role of macrophages and their activation via MyD88-NFκB signaling in lung innate immunity to Mycoplasma pneumoniae

http://www.wikidata.org/entity/Q34437813

description

2010 nî lūn-bûn

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2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Carlene L Zindl

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David D Chaplin

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Duncan C Krause

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Jen-Feng Lai

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Ken B Waites

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Lynn B Duffy

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T Prescott Atkinson

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Yong Woo Jung

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P2860

RAG-1-deficient mice have no mature B and T lymphocytes

The family of five: TIR-domain-containing adaptors in Toll-like receptor signalling

Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function

Terminal organelle development in the cell wall-less bacterium Mycoplasma pneumoniae.

Toll-like receptor heterodimer variants protect from childhood asthma.

Mycoplasma pneumoniae protein P30 is required for cytadherence and associated with proper cell development.

A trial of clarithromycin for the treatment of suboptimally controlled asthma.

Synthesis, stability, and function of cytadhesin P1 and accessory protein B/C complex of Mycoplasma pneumoniae

Chronic respiratory mycoplasmosis in C3H/HeN and C57BL/6N mice: lesion severity and antibody response.

Biofunctional domains of the Mycoplasma pneumoniae P30 adhesin

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Nico van Rooijen

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2010-12-23T00:00:00Z

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