Interleukin-13 is the key effector Th2 cytokine in ulcerative colitis that affects epithelial tight junctions, apoptosis, and cell restitution.
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Role of the gut microbiota in defining human healthBlocking IL-25 signalling protects against gut inflammation in a type-2 model of colitis by suppressing nuocyte and NKT derived IL-13Intestinal permeability defects: is it time to treat?Roles of infection, inflammation, and the immune system in cholesterol gallstone formationTight junctions in inflammatory bowel diseases and inflammatory bowel disease associated colorectal cancerBreaking down the barriers: the gut microbiome, intestinal permeability and stress-related psychiatric disordersCurrent stage in inflammatory bowel disease: What is next?Mitochondrial dysfunction in inflammatory bowel diseaseEvaluating the Oxidative Stress in Inflammation: Role of MelatoninControl of intestinal homeostasis through crosstalk between natural killer T cells and the intestinal microbiotaConceptual barriers to understanding physical barriersInnate and adaptive immunity in inflammatory bowel diseaseMolecular aspects of tight junction barrier functionClaudins in intestines: Distribution and functional significance in health and diseasesRole of Rho kinase signal pathway in inflammatory bowel diseaseIntestinal permeability--a new target for disease prevention and therapyIntestinal epithelium in inflammatory bowel diseaseIntestinal barrier in inflammatory bowel diseaseImmunopathology of inflammatory bowel diseaseCryptosporidium Priming Is More Effective than Vaccine for Protection against Cryptosporidiosis in a Murine Protein Malnutrition Model.Interleukin 13 and serotonin: linking the immune and endocrine systems in murine models of intestinal inflammationRho-A prenylation and signaling link epithelial homeostasis to intestinal inflammation.ClC-2 regulation of intestinal barrier function: Translation of basic science to therapeutic targetMolecular mechanism of action of anti-tumor necrosis factor antibodies in inflammatory bowel diseasesDysregulation of Group 3 Innate Lymphoid Cells in the Pathogenesis of Inflammatory Bowel DiseaseCrosstalk between Inflammation and ROCK/MLCK Signaling Pathways in Gastrointestinal Disorders with Intestinal HyperpermeabilityWhen Insult Is Added to Injury: Cross Talk between ILCs and Intestinal Epithelium in IBDIntestinal Permeability in Inflammatory Bowel Disease: Pathogenesis, Clinical Evaluation, and Therapy of Leaky GutInterleukin-12 and Th1 immune response in Crohn's disease: pathogenetic relevance and therapeutic implicationThe intestinal barrier as an emerging target in the toxicological assessment of mycotoxinsAberrant mucin assembly in mice causes endoplasmic reticulum stress and spontaneous inflammation resembling ulcerative colitisClaudin-2 Expression Levels in Ulcerative Colitis: Development and Validation of an In-Situ Hybridisation Assay for Therapeutic StudiesApplication of the adverse outcome pathway (AOP) concept to structure the available in vivo and in vitro mechanistic data for allergic sensitization to food proteinsRole of Corticotropin-releasing Factor in Gastrointestinal Permeability.SAMP1/YitFc mouse strain: a spontaneous model of Crohn's disease-like ileitis.Probiotic bacteria regulate intestinal epithelial permeability in experimental ileitis by a TNF-dependent mechanismMicrotubules are required for efficient epithelial tight junction homeostasis and restoration.Glycoprotein A33 deficiency: a new mouse model of impaired intestinal epithelial barrier function and inflammatory disease.Changes in expression and distribution of claudin 2, 5 and 8 lead to discontinuous tight junctions and barrier dysfunction in active Crohn's disease.Proteasome-mediated degradation of IkappaBalpha and processing of p105 in Crohn disease and ulcerative colitis.
P2860
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P2860
Interleukin-13 is the key effector Th2 cytokine in ulcerative colitis that affects epithelial tight junctions, apoptosis, and cell restitution.
description
2005 nî lūn-bûn
@nan
2005 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Interleukin-13 is the key effe ...... optosis, and cell restitution.
@ast
Interleukin-13 is the key effe ...... optosis, and cell restitution.
@en
Interleukin-13 is the key effe ...... optosis, and cell restitution.
@nl
type
label
Interleukin-13 is the key effe ...... optosis, and cell restitution.
@ast
Interleukin-13 is the key effe ...... optosis, and cell restitution.
@en
Interleukin-13 is the key effe ...... optosis, and cell restitution.
@nl
prefLabel
Interleukin-13 is the key effe ...... optosis, and cell restitution.
@ast
Interleukin-13 is the key effe ...... optosis, and cell restitution.
@en
Interleukin-13 is the key effe ...... optosis, and cell restitution.
@nl
P2093
P1433
P1476
Interleukin-13 is the key effe ...... optosis, and cell restitution.
@en
P2093
Alfred H Gitter
Bernd Hillenbrand
Christian Bojarski
Frank Heller
Jan Richter
Joachim Mankertz
Jörg D Schulzke
Martin Zeitz
Melanie Christ
P304
P356
10.1053/J.GASTRO.2005.05.002
P407
P577
2005-08-01T00:00:00Z