about
The Role of ACTH and Corticosteroids for Sepsis and Septic Shock: An UpdateEarly impairment of intracranial conduction time predicts mortality in deeply sedated critically ill patients: a prospective observational pilot studyPharmacokinetics of oral fludrocortisone in septic shock.Early Standard Electroencephalogram Abnormalities Predict Mortality in Septic Intensive Care Unit PatientsDesign and conduct of the activated protein C and corticosteroids for human septic shock (APROCCHSS) trialLentiviral-Mediated Overexpression of the 18 kDa Translocator Protein (TSPO) in the Hippocampal Dentate Gyrus Ameliorates LPS-Induced Cognitive Impairment in Mice.Neuroanatomy of sepsis-associated encephalopathyRecent developments in severe sepsis research: from bench to bedside and back.Cerebral Blood Flow Autoregulation in Sepsis for the Intensivist: Why Its Monitoring May Be the Future of Individualized Care.Neuroinflammation in liver disease: sessional talks from ISHEN.Potentially modifiable factors contributing to sepsis-associated encephalopathy.Cerebral perfusion alterations and cognitive decline in critically ill sepsis survivors.Melatonin attenuates sepsis-induced cardiac dysfunction via a PI3K/Akt-dependent mechanism.Septicemia is associated with increased risk for dementia: a population-based longitudinal study.Activation of the Akt/mTOR signaling pathway: A potential response to long-term neuronal loss in the hippocampus after sepsis.Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment.Enhancing Recovery From Sepsis: A Review.Unraveling the myriad contributors to persistent diminished exercise capacity after critical illness.Forebrain Cholinergic Dysfunction and Systemic and Brain Inflammation in Murine Sepsis Survivors.Endotoxemia induces lung-brain coupling and multi-organ injury following cerebral ischemia-reperfusion.Brain-Defective Insulin Signaling Is Associated to Late Cognitive Impairment in Post-Septic Mice.Neuroinflammation-Induced Downregulation of Hippocampacal Neuregulin 1-ErbB4 Signaling in the Parvalbumin Interneurons Might Contribute to Cognitive Impairment in a Mouse Model of Sepsis-Associated Encephalopathy.S100A8/A9 Drives Neuroinflammatory Priming and Protects against Anxiety-like Behavior after Sepsis.
P2860
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P2860
description
2014 nî lūn-bûn
@nan
2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Cognitive decline after sepsis.
@ast
Cognitive decline after sepsis.
@en
Cognitive decline after sepsis.
@nl
type
label
Cognitive decline after sepsis.
@ast
Cognitive decline after sepsis.
@en
Cognitive decline after sepsis.
@nl
prefLabel
Cognitive decline after sepsis.
@ast
Cognitive decline after sepsis.
@en
Cognitive decline after sepsis.
@nl
P1476
Cognitive decline after sepsis.
@en
P2093
Tarek Sharshar
P356
10.1016/S2213-2600(14)70246-2
P577
2014-11-28T00:00:00Z