Interleukin-22 regulates the complement system to promote resistance against pathobionts after pathogen-induced intestinal damage. - Wikidata - wikimedia - TriplyDB

Interleukin-22 regulates the complement system to promote resistance against pathobionts after pathogen-induced intestinal damage.

Interleukin-22 regulates the complement system to promote resistance against pathobionts after pathogen-induced intestinal damage.

http://www.wikidata.org/entity/Q34453038

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Interleukin-22 Signaling in the Regulation of Intestinal Health and Disease Interleukin-22 in kidney injury and regeneration.The binary toxin CDT enhances Clostridium difficile virulence by suppressing protective colonic eosinophilia.Microbiota-Regulated IL-25 Increases Eosinophil Number to Provide Protection during Clostridium difficile Infection.The microbiota and immune response during Clostridium difficile infection.Gut Microbiota-Induced Immunoglobulin G Controls Systemic Infection by Symbiotic Bacteria and Pathogens Innate Immune Defenses Mediated by Two ILC Subsets Are Critical for Protection against Acute Clostridium difficile Infection.Understanding the mechanisms of faecal microbiota transplantation.Clostridium difficile colitis: pathogenesis and host defence Short bowel syndrome results in increased gene expression associated with proliferation, inflammation, bile acid synthesis and immune system activation: RNA sequencing a zebrafish SBS model Innate lymphoid cells: new insights into function and development.Emerging roles for antigen presentation in establishing host-microbiome symbiosis Influence of orally fed a select mixture of Bacillus probiotics on intestinal T-cell migration in weaned MUC4 resistant pigs following Escherichia coli challenge.IL-23 induced in keratinocytes by endogenous TLR4 ligands polarizes dendritic cells to drive IL-22 responses to skin immunization IL-22 Controls Iron-Dependent Nutritional Immunity Against Systemic Bacterial Infections.Interactions between intestinal pathogens, enteropathy and malnutrition in developing countries Neutrophil-mediated inflammation in the pathogenesis of Clostridium difficile infections.Colonization and effector functions of innate lymphoid cells in mucosal tissues Mechanisms of inflammation-driven bacterial dysbiosis in the gut.The Role of the Microbiota in Shaping Infectious Immunity Linking the Microbiota, Chronic Disease, and the Immune System.Severe and Rapid Progression in Very Early-Onset Chronic Granulomatous Disease-Associated Colitis.Cytokines IL-17 and IL-22 in the host response to infection.Role of interferon-γ and inflammatory monocytes in driving colonic inflammation during acute Clostridium difficile infection in mice.Citrobacter rodentium: a model enteropathogen for understanding the interplay of innate and adaptive components of type 3 immunity.Amelioration of Clostridium difficile Infection in Mice by Dietary Supplementation With Indole-3-carbinol.Gut microbiota: Role in pathogen colonization, immune responses, and inflammatory disease.Is Clostridium difficile infection a risk factor for subsequent bloodstream infection?Novel Resolvin D2 Receptor Axis in Infectious Inflammation.Interleukin-23 (IL-23), independent of IL-17 and IL-22, drives neutrophil recruitment and innate inflammation during Clostridium difficile colitis in mice.Critical Role of IL-22/IL22-RA1 Signaling in Pneumococcal Pneumonia Infectious disease: IL-22 complements protection.In Silico Screening of the Human Gut Metaproteome Identifies Th17-Promoting Peptides Encrypted in Proteins of Commensal Bacteria.Elevated levels of CCR6+ T helper 22 cells correlate with skin and renal impairment in systemic lupus erythematosus.Innate Lymphoid Cells in Intestinal Inflammation Bacillus anthracis lethal toxin negatively modulates ILC3 function through perturbation of IL-23-mediated MAPK signaling.Novel mechanisms and functions of complement.IL-27/IL-27 Receptor Signaling Provides Protection in Clostridium difficile-Induced Colitis.Interleukin-22 Prevents Microbial Dysbiosis and Promotes Intestinal Barrier Regeneration Following Acute Injury.Interleukin-22 immunotherapy during severe influenza enhances lung tissue integrity and reduces secondary bacterial systemic invasion.

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Interleukin-22 regulates the complement system to promote resistance against pathobionts after pathogen-induced intestinal damage.

http://www.wikidata.org/entity/Q34453038

description

2014 nî lūn-bûn

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2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Interleukin-22 regulates the c ...... gen-induced intestinal damage.

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Interleukin-22 regulates the c ...... gen-induced intestinal damage.

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Interleukin-22 regulates the c ...... gen-induced intestinal damage.

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Interleukin-22 regulates the c ...... gen-induced intestinal damage.

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Interleukin-22 regulates the c ...... gen-induced intestinal damage.

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J.IMMUNI.2014.09.010

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Interleukin-22 regulates the c ...... ogen-induced intestinal damage

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Gabriel Núñez

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Meng Zhen Liu

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Mizuho Hasegawa

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Naohiro Inohara

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Nhu Do

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Raúl Muñoz-Planillo

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Shoko Yada

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P2860

Introducing mothur: open-source, platform-independent, community-supported software for describing and comparing microbial communities

Faecal microbiota transplantation for the treatment of recurrent Clostridium difficile infection: current promise and future needs

Emerging insights on intestinal dysbiosis during bacterial infections

IL-22 increases the innate immunity of tissues

MyD88-mediated signals induce the bactericidal lectin RegIII gamma and protect mice against intestinal Listeria monocytogenes infection

The gut microbiota shapes intestinal immune responses during health and disease

Clostridium difficile infection: new developments in epidemiology and pathogenesis

Interleukin-22 mediates early host defense against attaching and effacing bacterial pathogens

In vivo physiological and transcriptional profiling reveals host responses to Clostridium difficile toxin A and toxin B

Development of a dual-index sequencing strategy and curation pipeline for analyzing amplicon sequence data on the MiSeq Illumina sequencing platform.

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620-632

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10.1016/J.IMMUNI.2014.09.010

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4

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41

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Nobuhiko Kamada

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267814349

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25367575

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4220303

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