Recovery of arrested replication forks by homologous recombination is error-prone.
about
Mechanisms underlying structural variant formation in genomic disordersThe FHIT gene product: tumor suppressor and genome "caretaker"Genetic instability in budding and fission yeast-sources and mechanismsPolymerase δ replicates both strands after homologous recombination-dependent fork restartFission yeast Rad52 phosphorylation restrains error prone recombination pathwaysIs homologous recombination really an error-free process?The extent of error-prone replication restart by homologous recombination is controlled by Exo1 and checkpoint proteins.Mutation frequency dynamics in HPRT locus in culture-adapted human embryonic stem cells and induced pluripotent stem cells correspond to their differentiated counterpartsThe chromatin assembly factor 1 promotes Rad51-dependent template switches at replication forks by counteracting D-loop disassembly by the RecQ-type helicase Rqh1Size of gene specific inverted repeat--dependent gene deletion In Saccharomyces cerevisiae.Regulation of recombination at yeast nuclear pores controls repair and triplet repeat stability.Repeat instability during DNA repair: Insights from model systemsMassive interstitial copy-neutral loss-of-heterozygosity as evidence for cancer being a disease of the DNA-damage response.Role of the double-strand break repair pathway in the maintenance of genomic stability.Loss of heterozygosity preferentially occurs in early replicating regions in cancer genomes.Replication fork collapse is a major cause of the high mutation frequency at three-base lesion clustersReplicative mechanisms for CNV formation are error prone.Loss of Caenorhabditis elegans BRCA1 promotes genome stability during replication in smc-5 mutantsChromosome replication origins: do we really need them?Quality control of homologous recombination.Homologous recombination as a replication fork escort: fork-protection and recovery.Stalled replication forks generate a distinct mutational signature in yeast.Systematic Identification of Determinants for Single Strand Annealing Mediated Deletion Formation in Saccharomyces cerevisiaeRecombination-restarted replication makes inverted chromosome fusions at inverted repeats.Break-induced replication links microsatellite expansion to complex genome rearrangements.Role of recombination and replication fork restart in repeat instability.Chronic p53-independent p21 expression causes genomic instability by deregulating replication licensing.Recombination occurs within minutes of replication blockage by RTS1 producing restarted forks that are prone to collapseCharacterization of 26 deletion CNVs reveals the frequent occurrence of micro-mutations within the breakpoint-flanking regions and frequent repair of double-strand breaks by templated insertions derived from remote genomic regions.Hyperactive Cdc2 kinase interferes with the response to broken replication forks by trapping S.pombe Crb2 in its mitotic T215 phosphorylated state.Host factors that promote retrotransposon integration are similar in distantly related eukaryotes.The end-joining factor Ku acts in the end-resection of double strand break-free arrested replication forks.Cdc7-Dbf4-mediated phosphorylation of HSP90-S164 stabilizes HSP90-HCLK2-MRN complex to enhance ATR/ATM signaling that overcomes replication stress in cancer.A Molecular Toolbox to Engineer Site-Specific DNA Replication Perturbation.Increased activity of both CDK1 and CDK2 is necessary for the combinatorial activity of WEE1 inhibition and cytarabine.Preserving replication fork integrity and competence via the homologous recombination pathway
P2860
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P2860
Recovery of arrested replication forks by homologous recombination is error-prone.
description
2012 nî lūn-bûn
@nan
2012 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Recovery of arrested replication forks by homologous recombination is error-prone.
@ast
Recovery of arrested replication forks by homologous recombination is error-prone.
@en
Recovery of arrested replication forks by homologous recombination is error-prone.
@nl
type
label
Recovery of arrested replication forks by homologous recombination is error-prone.
@ast
Recovery of arrested replication forks by homologous recombination is error-prone.
@en
Recovery of arrested replication forks by homologous recombination is error-prone.
@nl
prefLabel
Recovery of arrested replication forks by homologous recombination is error-prone.
@ast
Recovery of arrested replication forks by homologous recombination is error-prone.
@en
Recovery of arrested replication forks by homologous recombination is error-prone.
@nl
P2093
P2860
P921
P1433
P1476
Recovery of arrested replication forks by homologous recombination is error-prone.
@en
P2093
Audrey Costes
Ismail Iraqui
Karine Fréon
Nada Jmari
Sarah A E Lambert
Violena Pietrobon
Yasmina Chekkal
P2860
P304
P356
10.1371/JOURNAL.PGEN.1002976
P577
2012-10-18T00:00:00Z