Modeling K,ATP--dependent excitability in pancreatic islets.
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Decreases in Gap Junction Coupling Recovers Ca2+ and Insulin Secretion in Neonatal Diabetes Mellitus, Dependent on Beta Cell Heterogeneity and NoiseConcerted Trafficking Regulation of Kv2.1 and KATP Channels by Leptin in Pancreatic β-Cells.A computationally efficient algorithm for fitting ion channel parameters.NMDA receptors mediate leptin signaling and regulate potassium channel trafficking in pancreatic β-cells.
P2860
Modeling K,ATP--dependent excitability in pancreatic islets.
description
2014 nî lūn-bûn
@nan
2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Modeling K,ATP--dependent excitability in pancreatic islets.
@ast
Modeling K,ATP--dependent excitability in pancreatic islets.
@en
Modeling K,ATP--dependent excitability in pancreatic islets.
@nl
type
label
Modeling K,ATP--dependent excitability in pancreatic islets.
@ast
Modeling K,ATP--dependent excitability in pancreatic islets.
@en
Modeling K,ATP--dependent excitability in pancreatic islets.
@nl
prefLabel
Modeling K,ATP--dependent excitability in pancreatic islets.
@ast
Modeling K,ATP--dependent excitability in pancreatic islets.
@en
Modeling K,ATP--dependent excitability in pancreatic islets.
@nl
P2860
P1433
P1476
Modeling K,ATP--dependent excitability in pancreatic islets
@en
P2093
Colin G Nichols
P2860
P304
P356
10.1016/J.BPJ.2014.09.037
P407
P577
2014-11-01T00:00:00Z