A mathematical model of CR3/TLR2 crosstalk in the context of Francisella tularensis infection. - Wikidata - wikimedia - TriplyDB

A mathematical model of CR3/TLR2 crosstalk in the context of Francisella tularensis infection.

A mathematical model of CR3/TLR2 crosstalk in the context of Francisella tularensis infection.

http://www.wikidata.org/entity/Q34468902

about

Fine tuning inflammation at the front door: macrophage complement receptor 3-mediates phagocytosis and immune suppression for Francisella tularensis Adherence and uptake of Francisella into host cells Inflammasome priming is similar for francisella species that differentially induce inflammasome activation.Dynamics of a Tularemia Outbreak in a Closely Monitored Free-Roaming Population of Wild House Mice.Non-canonical Activation of Akt in Serum-Stimulated Fibroblasts, Revealed by Comparative Modeling of Pathway Dynamics Exosomal miRs in Lung Cancer: A Mathematical Model.Lyn mitigates mouse airway remodeling by downregulating the TGF-β3 isoform in house dust mite models.Serving the new masters - dendritic cells as hosts for stealth intracellular bacteria.Mathematical Models for Immunology: Current State of the Art and Future Research Directions.Complement Receptor 3-Mediated Inhibition of Inflammasome Priming by Ras GTPase-Activating Protein During Francisella tularensis Phagocytosis by Human Mononuclear Phagocytes.

P2860

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P2860

A mathematical model of CR3/TLR2 crosstalk in the context of Francisella tularensis infection.

http://www.wikidata.org/entity/Q34468902

description

2012 nî lūn-bûn

@nan

2012 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

A mathematical model of CR3/TL ...... ncisella tularensis infection.

@ast

A mathematical model of CR3/TL ...... ncisella tularensis infection.

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A mathematical model of CR3/TL ...... ncisella tularensis infection.

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type

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A mathematical model of CR3/TL ...... ncisella tularensis infection.

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A mathematical model of CR3/TL ...... ncisella tularensis infection.

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A mathematical model of CR3/TL ...... ncisella tularensis infection.

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prefLabel

A mathematical model of CR3/TL ...... ncisella tularensis infection.

@ast

A mathematical model of CR3/TL ...... ncisella tularensis infection.

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A mathematical model of CR3/TL ...... ncisella tularensis infection.

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PLOS Computational Biology

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A mathematical model of CR3/TL ...... ncisella tularensis infection.

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Avner Friedman

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Larry S Schlesinger

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Rachel Leander

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Shipan Dai

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P2860

Identification of ARAP3, a novel PI3K effector regulating both Arf and Rho GTPases, by selective capture on phosphoinositide affinity matrices

Mechanistic basis of differential cellular responses of phosphatidylinositol 3,4-bisphosphate- and phosphatidylinositol 3,4,5-trisphosphate-binding pleckstrin homology domains

Protein-tyrosine kinase and GTPase signals cooperate to phosphorylate and activate Wiskott-Aldrich syndrome protein (WASP)/neuronal WASP

ArhGAP15, a novel human RacGAP protein with GTPase binding property

Membrane sorting of toll-like receptor (TLR)-2/6 and TLR2/1 heterodimers at the cell surface determines heterotypic associations with CD36 and intracellular targeting

Kinetic mechanism of AKT/PKB enzyme family

ARAP3 is transiently tyrosine phosphorylated in cells attaching to fibronectin and inhibits cell spreading in a RhoGAP-dependent manner

gC1q receptor ligation selectively down-regulates human IL-12 production through activation of the phosphoinositide 3-kinase pathway

Cell size of alveolar macrophages: an interspecies comparison

Regulatory and signaling properties of the Vav family

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10.1371/JOURNAL.PCBI.1002757

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