Efficient marker-free recovery of custom genetic modifications with CRISPR/Cas9 in Caenorhabditis elegans.
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Genome Editing in C. elegans and Other Nematode SpeciesNuclease Target Site Selection for Maximizing On-target Activity and Minimizing Off-target Effects in Genome EditingRendering the Intractable More Tractable: Tools from Caenorhabditis elegans Ripe for Import into Parasitic NematodesThe application of CRISPR-Cas9 genome editing in Caenorhabditis elegansMultisite Phosphorylation of NuMA-Related LIN-5 Controls Mitotic Spindle Positioning in C. elegansEvolution of New cis-Regulatory Motifs Required for Cell-Specific Gene Expression in CaenorhabditisSelection on a Subunit of the NURF Chromatin Remodeler Modifies Life History Traits in a Domesticated Strain of Caenorhabditis elegansSynapse-Assembly Proteins Maintain Synaptic Vesicle Cluster Stability and Regulate Synaptic Vesicle Transport in Caenorhabditis elegans.Prospects and challenges of CRISPR/Cas genome editing for the study and control of neglected vector-borne nematode diseasesTargeted genome engineering in Caenorhabditis elegansMutations in Nonessential eIF3k and eIF3l Genes Confer Lifespan Extension and Enhanced Resistance to ER Stress in Caenorhabditis elegansIn Vivo Modelling of ATP1A3 G316S-Induced Ataxia in C. elegans Using CRISPR/Cas9-Mediated Homologous Recombination Reveals Dominant Loss of Function DefectsBalancing selection shapes density-dependent foraging behaviourLocal inhibition of microtubule dynamics by dynein is required for neuronal cargo distribution.Reliable CRISPR/Cas9 Genome Engineering in Caenorhabditis elegans Using a Single Efficient sgRNA and an Easily Recognizable Phenotype.Autoinhibition of a Neuronal Kinesin UNC-104/KIF1A Regulates the Size and Density of Synapses.Localized TWIST1 and TWIST2 basic domain substitutions cause four distinct human diseases that can be modeled in Caenorhabditis elegansSapTrap, a Toolkit for High-Throughput CRISPR/Cas9 Gene Modification in Caenorhabditis elegansScalable and versatile genome editing using linear DNAs with microhomology to Cas9 Sites in Caenorhabditis elegansDual sgRNA-directed gene knockout using CRISPR/Cas9 technology in Caenorhabditis elegans.Rapid and precise engineering of the Caenorhabditis elegans genome with lethal mutation co-conversion and inactivation of NHEJ repair.Dramatic enhancement of genome editing by CRISPR/Cas9 through improved guide RNA design.Rapid and Efficient Identification of Caenorhabditis elegans Legacy Mutations Using Hawaiian SNP-Based Mapping and Whole-Genome Sequencing.CRISPR/Cas9 Genome Editing in Caenorhabditis elegans: Evaluation of Templates for Homology-Mediated Repair and Knock-Ins by Homology-Independent DNA RepairStaufen Negatively Modulates MicroRNA Activity in Caenorhabditis elegans.Distinct patterns of Cas9 mismatch tolerance in vitro and in vivoHigh Efficiency, Homology-Directed Genome Editing in Caenorhabditis elegans Using CRISPR-Cas9 Ribonucleoprotein Complexes.Streamlined Genome Engineering with a Self-Excising Drug Selection CassetteEfficient Genome Editing in Caenorhabditis elegans with a Toolkit of Dual-Marker Selection Cassettes.Functional relevance of "seed" and "non-seed" sequences in microRNA-mediated promotion of C. elegans developmental progression.A conserved family of proteins facilitates nascent lipid droplet budding from the ER.Coordinated inhibition of C/EBP by Tribbles in multiple tissues is essential for Caenorhabditis elegans development.Two Paralogous Tetraspanins TSP-12 and TSP-14 Function with the ADAM10 Metalloprotease SUP-17 to Promote BMP Signaling in Caenorhabditis elegansβ-Integrin de-phosphorylation by the Density-Enhanced Phosphatase DEP-1 attenuates EGFR signaling in C. elegans.Targeted Chromosomal Translocations and Essential Gene Knockout Using CRISPR/Cas9 Technology in Caenorhabditis elegans.The auxin-inducible degradation (AID) system enables versatile conditional protein depletion in C. elegansPGL germ granule assembly protein is a base-specific, single-stranded RNase.The E2F-DP1 Transcription Factor Complex Regulates Centriole Duplication in Caenorhabditis elegans.Enriching CRISPR-Cas9 targeted cells by co-targeting the HPRT gene.CRISPR-Based Methods for Caenorhabditis elegans Genome Engineering.
P2860
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P2860
Efficient marker-free recovery of custom genetic modifications with CRISPR/Cas9 in Caenorhabditis elegans.
description
2014 nî lūn-bûn
@nan
2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Efficient marker-free recovery ...... as9 in Caenorhabditis elegans.
@ast
Efficient marker-free recovery ...... as9 in Caenorhabditis elegans.
@en
Efficient marker-free recovery ...... as9 in Caenorhabditis elegans.
@nl
type
label
Efficient marker-free recovery ...... as9 in Caenorhabditis elegans.
@ast
Efficient marker-free recovery ...... as9 in Caenorhabditis elegans.
@en
Efficient marker-free recovery ...... as9 in Caenorhabditis elegans.
@nl
prefLabel
Efficient marker-free recovery ...... as9 in Caenorhabditis elegans.
@ast
Efficient marker-free recovery ...... as9 in Caenorhabditis elegans.
@en
Efficient marker-free recovery ...... as9 in Caenorhabditis elegans.
@nl
P2093
P2860
P1433
P1476
Efficient marker-free recovery ...... as9 in Caenorhabditis elegans.
@en
P2093
Andrew Z Fire
Becky X H Fu
Joshua A Arribere
Karen L Artiles
Phil S Hartman
Ryan T Bell
P2860
P304
P356
10.1534/GENETICS.114.169730
P407
P577
2014-08-26T00:00:00Z