Recruited inflammatory monocytes stimulate antiviral Th1 immunity in infected tissue.
about
CCR2 deficiency promotes exacerbated chronic erosive neutrophil-dominated chikungunya virus arthritisThe role of dendritic cells in immunity against primary herpes simplex virus infectionsInflammatory monocytes and the pathogenesis of viral encephalitisMolecular control of monocyte developmentDistinct APC subtypes drive spatially segregated CD4+ and CD8+ T-cell effector activity during skin infection with HSV-1IL-22 and IDO1 affect immunity and tolerance to murine and human vaginal candidiasisInflammatory monocytes hinder antiviral B cell responses.Differential roles of lung dendritic cell subsets against respiratory virus infectionMDR1A deficiency restrains tumor growth in murine colitis-associated carcinogenesisTannic acid modified silver nanoparticles show antiviral activity in herpes simplex virus type 2 infectionHerpes simplex virus type-2 stimulates HIV-1 replication in cervical tissues: implications for HIV-1 transmission and efficacy of anti-HIV-1 microbicidesToll-like receptor 4 and MAIR-II/CLM-4/LMIR2 immunoreceptor regulate VLA-4-mediated inflammatory monocyte migrationProphylactic and therapeutic modulation of innate and adaptive immunity against mucosal infection of herpes simplex virusThe pathological effects of CCR2+ inflammatory monocytes are amplified by an IFNAR1-triggered chemokine feedback loop in highly pathogenic influenza infection.Mice transgenic for CD4-specific human CD4, CCR5 and cyclin T1 expression: a new model for investigating HIV-1 transmission and treatment efficacy.Cornea-infiltrating and lymph node dendritic cells contribute to CD4+ T cell expansion after herpes simplex virus-1 ocular infectionHSV-2 regulates monocyte inflammatory response via the Fas/FasL pathwayAlveolar macrophage-derived type I interferons orchestrate innate immunity to RSV through recruitment of antiviral monocytesCervicovaginal bacteria are a major modulator of host inflammatory responses in the female genital tractInflammatory monocyte effector mechanisms.Distinct Upstream Role of Type I IFN Signaling in Hematopoietic Stem Cell-Derived and Epithelial Resident Cells for Concerted Recruitment of Ly-6Chi Monocytes and NK Cells via CCL2-CCL3 CascadeDysbiosis-induced IL-33 contributes to impaired antiviral immunity in the genital mucosaThe Vaginal Acquisition and Dissemination of HIV-1 Infection in a Novel Transgenic Mouse Model Is Facilitated by Coinfection with Herpes Simplex Virus 2 and Is Inhibited by Microbicide Treatment.Modulation of Female Genital Tract-Derived Dendritic Cell Migration and Activation in Response to Inflammatory Cytokines and Toll-Like Receptor AgonistsCooperation between Monocyte-Derived Cells and Lymphoid Cells in the Acute Response to a Bacterial Lung PathogenNK cell-derived interferon-γ orchestrates cellular dynamics and the differentiation of monocytes into dendritic cells at the site of infectionEffect of Mucosal Cytokine Administration on Selective Expansion of Vaginal Dendritic Cells to Support Nanoparticle Transport.Unique features of antiviral immune system of the vaginal mucosaBoth Cerebral and Hematopoietic Deficiencies in CCR2 Result in Uncontrolled Herpes Simplex Virus Infection of the Central Nervous System in Mice.Amelioration of Japanese encephalitis by blockage of 4-1BB signaling is coupled to divergent enhancement of type I/II IFN responses and Ly-6C(hi) monocyte differentiation.CD11c(hi) Dendritic Cells Regulate Ly-6C(hi) Monocyte Differentiation to Preserve Immune-privileged CNS in Lethal Neuroinflammation.CD11b+Ly6G- myeloid cells mediate mechanical inflammatory pain hypersensitivity.OASL1 deficiency promotes antiviral protection against genital herpes simplex virus type 2 infection by enhancing type I interferon productionBlockage of indoleamine 2,3-dioxygenase regulates Japanese encephalitis via enhancement of type I/II IFN innate and adaptive T-cell responses.Use of transcriptional profiling to delineate the initial response of mice to intravaginal herpes simplex virus type 2 infection.Access of protective antiviral antibody to neuronal tissues requires CD4 T-cell helpInflammatory monocytes regulate pathologic responses to commensals during acute gastrointestinal infection.Inflammatory monocytes and NK cells play a crucial role in DNAM-1-dependent control of cytomegalovirus infection.Regulatory T cells are essential to promote proper CD4 T-cell priming upon mucosal infection.Minimal differentiation of classical monocytes as they survey steady-state tissues and transport antigen to lymph nodes
P2860
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P2860
Recruited inflammatory monocytes stimulate antiviral Th1 immunity in infected tissue.
description
2010 nî lūn-bûn
@nan
2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Recruited inflammatory monocytes stimulate antiviral Th1 immunity in infected tissue.
@ast
Recruited inflammatory monocytes stimulate antiviral Th1 immunity in infected tissue.
@en
Recruited inflammatory monocytes stimulate antiviral Th1 immunity in infected tissue.
@nl
type
label
Recruited inflammatory monocytes stimulate antiviral Th1 immunity in infected tissue.
@ast
Recruited inflammatory monocytes stimulate antiviral Th1 immunity in infected tissue.
@en
Recruited inflammatory monocytes stimulate antiviral Th1 immunity in infected tissue.
@nl
prefLabel
Recruited inflammatory monocytes stimulate antiviral Th1 immunity in infected tissue.
@ast
Recruited inflammatory monocytes stimulate antiviral Th1 immunity in infected tissue.
@en
Recruited inflammatory monocytes stimulate antiviral Th1 immunity in infected tissue.
@nl
P2093
P2860
P356
P1476
Recruited inflammatory monocytes stimulate antiviral Th1 immunity in infected tissue.
@en
P2093
Akiko Iwasaki
Lisa M Mattei
Norifumi Iijima
P2860
P304
P356
10.1073/PNAS.1005201108
P407
P577
2010-12-20T00:00:00Z