Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease - Wikidata - wikimedia - TriplyDB

Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

http://www.wikidata.org/entity/Q34479228

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Beneficial effects of autologous bone marrow-derived mesenchymal stem cells in naturally occurring tendinopathy Proteomic analysis reveals age-related changes in tendon matrix composition, with age- and injury-specific matrix fragmentation Aging delays resolution of acute inflammation in mice: reprogramming the host response with novel nano-proresolving medicines Transcriptome analysis of ageing in uninjured human Achilles tendon.Inflammation activation and resolution in human tendon disease.Proteomic analysis of tendon extracellular matrix reveals disease stage-specific fragmentation and differential cleavage of COMP (cartilage oligomeric matrix protein).Resolving an inflammatory concept: the importance of inflammation and resolution in tendinopathy.An overview of structure, mechanical properties, and treatment for age-related tendinopathy.Are inflammatory cells increased in painful human tendinopathy? A systematic review.MicroRNA29a Treatment Improves Early Tendon Injury.Influence of Ageing on Tendon Homeostasis.Selenium Deficiency Influences the Expression of Selenoproteins and Inflammatory Cytokines in Chicken Aorta Vessels.Tendon overload results in alterations in cell shape and increased markers of inflammation and matrix degradation.Ageing does not result in a decline in cell synthetic activity in an injury prone tendon.Differential expression of alarmins-S100A9, IL-33, HMGB1 and HIF-1α in supraspinatus tendinopathy before and after treatment.Tendon cell ciliary length as a biomarker of in situ cytoskeletal tensional homeostasis.

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P2860

Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

http://www.wikidata.org/entity/Q34479228

description

2012 nî lūn-bûn

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2012 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2012 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

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Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

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Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

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Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

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Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

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Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

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Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

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Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

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Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

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Inflamm-aging and arachadonic acid metabolite differences with stage of tendon disease

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Dilkush Robert Ephrem Abayasekara

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Roger Kenneth Whealands Smith

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Stephanie Georgina Dakin

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P2860

Aspirin-triggered 15-epi-lipoxin A4 (LXA4) and LXA4 stable analogues are potent inhibitors of acute inflammation: evidence for anti-inflammatory receptors

Mixed messages: modulation of inflammation and immune responses by prostaglandins and thromboxanes

A new mathematical model for relative quantification in real-time RT-PCR

Inflamm-aging. An evolutionary perspective on immunosenescence

Lipid-derived mediators in endogenous anti-inflammation and resolution: lipoxins and aspirin-triggered 15-epi-lipoxins

The lipoxin receptor ALX: potent ligand-specific and stereoselective actions in vivo

Lipoxins: novel series of biologically active compounds formed from arachidonic acid in human leukocytes

Macrophage sub-populations and the lipoxin A4 receptor implicate active inflammation during equine tendon repair

Double immunolabeling of neuropeptides in the human hypothalamus as analyzed by confocal laser scanning fluorescence microscopy.

Gap junction protein expression and cellularity: comparison of immature and adult equine digital tendons.

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Natalie Jayne Werling

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2012-11-14T00:00:00Z

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