Bone cancer induces a unique central sensitization through synaptic changes in a wide area of the spinal cord
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The Physiology of Bone Pain. How Much Do We Really Know?Bone cancer pain: from mechanism to therapyPalliation of bone cancer pain by antagonists of platelet-activating factor receptorsDissociation between the relief of skeletal pain behaviors and skin hypersensitivity in a model of bone cancer pain.Alleviation of behavioral hypersensitivity in mouse models of inflammatory pain with two structurally different casein kinase 1 (CK1) inhibitors.PAR2-mediated upregulation of BDNF contributes to central sensitization in bone cancer pain.The neurobiology of cancer pain.Enhanced GABAergic synaptic transmission at VLPAG neurons and potent modulation by oxycodone in a bone cancer pain model.Preventive or late administration of anti-NGF therapy attenuates tumor-induced nerve sprouting, neuroma formation, and cancer pain.Activation of c-jun N-terminal kinase in spinal cord contributes to breast cancer induced bone pain in rats.Activation of spinal MrgC-Gi-NR2B-nNOS signaling pathway by Mas oncogene-related gene C receptor agonist bovine adrenal medulla 8-22 attenuates bone cancer pain in mice.Minocycline attenuates bone cancer pain in rats by inhibiting NF-κB in spinal astrocytes.Pathobiology and management of prostate cancer-induced bone pain: recent insights and future treatments.Dorsal horn interneuron-derived Netrin-4 contributes to spinal sensitization in chronic pain via Unc5B.Differences in electrophysiological properties of functionally identified nociceptive sensory neurons in an animal model of cancer-induced bone painMediation of Movement-Induced Breakthrough Cancer Pain by IB4-Binding Nociceptors in Rats.Metastatic human breast cancer to the spine produces mechanical hyperalgesia and gait deficits in rodents.Brain-derived neurotrophic factor modulates N-methyl-D-aspartate receptor activation in a rat model of cancer-induced bone pain.Rat model of cancer-induced bone pain: changes in nonnociceptive sensory neurons in vivo.Mice with cancer-induced bone pain show a marked decline in day/night activity.Activation of PI3Kγ/Akt pathway mediates bone cancer pain in rats.Analgesic Effect of Intrathecal Administration of Chemokine Receptor CCR2 Antagonist is Related to Change in Spinal NR2B, nNOS, and SIGIRR Expression in Rat with Bone Cancer Pain.
P2860
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P2860
Bone cancer induces a unique central sensitization through synaptic changes in a wide area of the spinal cord
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
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2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
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2010年论文
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name
Bone cancer induces a unique c ...... a wide area of the spinal cord
@ast
Bone cancer induces a unique c ...... a wide area of the spinal cord
@en
Bone cancer induces a unique c ...... a wide area of the spinal cord
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type
label
Bone cancer induces a unique c ...... a wide area of the spinal cord
@ast
Bone cancer induces a unique c ...... a wide area of the spinal cord
@en
Bone cancer induces a unique c ...... a wide area of the spinal cord
@nl
prefLabel
Bone cancer induces a unique c ...... a wide area of the spinal cord
@ast
Bone cancer induces a unique c ...... a wide area of the spinal cord
@en
Bone cancer induces a unique c ...... a wide area of the spinal cord
@nl
P2093
P2860
P356
P1433
P1476
Bone cancer induces a unique c ...... a wide area of the spinal cord
@en
P2093
Daisuke Uta
Hidemasa Furue
Jun Yamamoto
Keiji Imoto
Megumu Yoshimura
Shingo Furuse
Tomoyuki Kawamata
Toshihiko Katafuchi
Yoshikazu Yanagisawa
Yukihide Iwamoto
P2860
P2888
P356
10.1186/1744-8069-6-38
P577
2010-07-05T00:00:00Z
P5875
P6179
1024463402