Identification of genes that elicit disuse muscle atrophy via the transcription factors p50 and Bcl-3. - Wikidata - wikimedia - TriplyDB

Identification of genes that elicit disuse muscle atrophy via the transcription factors p50 and Bcl-3.

Identification of genes that elicit disuse muscle atrophy via the transcription factors p50 and Bcl-3.

http://www.wikidata.org/entity/Q34490064

about

The TWEAK-Fn14 pathway: a potent regulator of skeletal muscle biology in health and disease Human Skeletal Muscle Disuse Atrophy: Effects on Muscle Protein Synthesis, Breakdown, and Insulin Resistance-A Qualitative Review Radiation protection following nuclear power accidents: a survey of putative mechanisms involved in the radioprotective actions of taurine during and after radiation exposure Regulatory circuitry of TWEAK-Fn14 system and PGC-1α in skeletal muscle atrophy program.Multi-tasking role of the mechanosensing protein Ankrd2 in the signaling network of striated muscle.Long-lasting effect of obesity on skeletal muscle transcriptome.RNA expression analysis of passive transfer myasthenia supports extraocular muscle as a unique immunological environment.DNA methyltransferase 3a and mitogen-activated protein kinase signaling regulate the expression of fibroblast growth factor-inducible 14 (Fn14) during denervation-induced skeletal muscle atrophy.Skeletal muscle atrophy and the E3 ubiquitin ligases MuRF1 and MAFbx/atrogin-1 The ChIP-seq-defined networks of Bcl-3 gene binding support its required role in skeletal muscle atrophy.p300 Acetyltransferase activity differentially regulates the localization and activity of the FOXO homologues in skeletal muscle.C26 cancer-induced muscle wasting is IKKβ-dependent and NF-kappaB-independent.Genome-wide mRNA expression profiling in vastus lateralis of COPD patients with low and normal fat free mass index and healthy controls.TWEAK-Fn14 pathway activation after exercise in human skeletal muscle: insights from two exercise modes and a time course investigation.The orphan nuclear receptor Nur77 is a determinant of myofiber size and muscle mass in mice.The sirtuin SIRT6 blocks IGF-Akt signaling and development of cardiac hypertrophy by targeting c-Jun.The TWEAK-Fn14 system: breaking the silence of cytokine-induced skeletal muscle wasting.The E3 ubiquitin ligase TRAF6 intercedes in starvation-induced skeletal muscle atrophy through multiple mechanisms Transcriptome signature of resistance exercise adaptations: mixed muscle and fiber type specific profiles in young and old adults.TWEAK and TRAF6 regulate skeletal muscle atrophy.Rel A/p65 is required for cytokine-induced myotube atrophy.Nuclear factor-κB signaling contributes to mechanical ventilation-induced diaphragm weakness*Disuse-induced muscle wasting.Differential RNA Expression Profile of Skeletal Muscle Induced by Experimental Autoimmune Myasthenia Gravis in Rats.TWEAK/Fn14 Signaling Axis Mediates Skeletal Muscle Atrophy and Metabolic Dysfunction.NF-κB but not FoxO sites in the MuRF1 promoter are required for transcriptional activation in disuse muscle atrophy.It takes two to tango: IκBs, the multifunctional partners of NF-κB.Nuclear factor-κB signalling and transcriptional regulation in skeletal muscle atrophy.Muscle-specific and age-related changes in protein synthesis and protein degradation in response to hindlimb unloading in rats.Transcriptomics, NF-κB Pathway, and Their Potential Spaceflight-Related Health Consequences.Endophilin-A Deficiency Induces the Foxo3a-Fbxo32 Network in the Brain and Causes Dysregulation of Autophagy and the Ubiquitin-Proteasome System.Paradoxical effect of IKKβ inhibition on the expression of E3 ubiquitin ligases and unloading-induced skeletal muscle atrophy.Effect of irradiation on Akt signaling in atrophying skeletal muscle.Chromatin structure analysis enables detection of DNA insertions into the mammalian nuclear genome.lncRNA Chronos is an aging-induced inhibitor of muscle hypertrophy.In utero LPS exposure impairs preterm diaphragm contractility.Recent advances in mitochondrial turnover during chronic muscle disuse.

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Identification of genes that elicit disuse muscle atrophy via the transcription factors p50 and Bcl-3.

http://www.wikidata.org/entity/Q34490064

description

2011 nî lūn-bûn

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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հունվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Identification of genes that e ...... ription factors p50 and Bcl-3.

@ast

Identification of genes that e ...... ription factors p50 and Bcl-3.

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Identification of genes that e ...... ription factors p50 and Bcl-3.

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type

label

Identification of genes that e ...... ription factors p50 and Bcl-3.

@ast

Identification of genes that e ...... ription factors p50 and Bcl-3.

@en

Identification of genes that e ...... ription factors p50 and Bcl-3.

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prefLabel

Identification of genes that e ...... ription factors p50 and Bcl-3.

@ast

Identification of genes that e ...... ription factors p50 and Bcl-3.

@en

Identification of genes that e ...... ription factors p50 and Bcl-3.

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JOURNAL.PONE.0016171

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Identification of genes that e ...... ription factors p50 and Bcl-3.

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Chia-Ling Wu

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Robert W Jackman

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Susan C Kandarian

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P2860

NF-kappaB p105 is a target of IkappaB kinases and controls signal induction of Bcl-3-p50 complexes

Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy

Statistical significance for genomewide studies

Gene expression profiling of potential peroxisome proliferator-activated receptor (PPAR) target genes in human hepatoblastoma cell lines inducibly expressing different PPAR isoforms

MURF-1 and MURF-2 target a specific subset of myofibrillar proteins redundantly: towards understanding MURF-dependent muscle ubiquitination

The candidate proto-oncogene bcl-3 encodes a transcriptional coactivator that activates through NF-kappa B p50 homodimers

Introduction to NF-kappaB: players, pathways, perspectives

Integrating cell-signalling pathways with NF-kappaB and IKK function

Identification of ubiquitin ligases required for skeletal muscle atrophy

Detection of functional DNA motifs via statistical over-representation

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scholarly article

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10.1371/JOURNAL.PONE.0016171

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