E-cadherin transcriptional downregulation by promoter methylation but not mutation is related to epithelial-to-mesenchymal transition in breast cancer cell lines. - Wikidata - wikimedia - TriplyDB

E-cadherin transcriptional downregulation by promoter methylation but not mutation is related to epithelial-to-mesenchymal transition in breast cancer cell lines.

E-cadherin transcriptional downregulation by promoter methylation but not mutation is related to epithelial-to-mesenchymal transition in breast cancer cell lines.

http://www.wikidata.org/entity/Q34497064

about

An expression signature of syndecan-1 (CD138), E-cadherin and c-met is associated with factors of angiogenesis and lymphangiogenesis in ductal breast carcinoma in situ Loss of FOXA1/2 is essential for the epithelial-to-mesenchymal transition in pancreatic cancer The SNAG domain of Snail1 functions as a molecular hook for recruiting lysine-specific demethylase 1 Estrogen-dependent sushi domain containing 3 regulates cytoskeleton organization and migration in breast cancer cells EMT in Breast Carcinoma-A Review Trachoma and Ocular Chlamydial Infection in the Era of Genomics Breast cancer epithelial-to-mesenchymal transition: examining the functional consequences of plasticity Epigenetic plasticity: a central regulator of epithelial-to-mesenchymal transition in cancer Polarized E-cadherin endocytosis directs actomyosin remodeling during embryonic wound repair.Microenvironment promotes tumor cell reprogramming in human breast cancer cell lines Regulation of epithelial-mesenchymal transition through epigenetic and post-translational modifications Examining the pathogenesis of breast cancer using a novel agent-based model of mammary ductal epithelium dynamics Resveratrol inhibits cisplatin-induced epithelial-to-mesenchymal transition in ovarian cancer cell lines Epigenetic coordination of signaling pathways during the epithelial-mesenchymal transition Down-regulation of epithelial cadherin is required to initiate metastatic outgrowth of breast cancer.ATBF1 and NQO1 as candidate targets for allelic loss at chromosome arm 16q in breast cancer: absence of somatic ATBF1 mutations and no role for the C609T NQO1 polymorphism Genome-wide analysis of aberrant methylation in human breast cancer cells using methyl-DNA immunoprecipitation combined with high-throughput sequencing Role of epithelial-mesenchymal transition in gastric cancer initiation and progression Role of DNA methylation in miR-200c/141 cluster silencing in invasive breast cancer cells Interplay between neural-cadherin and vascular endothelial-cadherin in breast cancer progression.Epigenetic regulation of epithelial-mesenchymal transition Annexin A1 regulates TGF-beta signaling and promotes metastasis formation of basal-like breast cancer cells.Dysregulation of Fra1 expression by Wnt/β-catenin signalling promotes glioma aggressiveness through epithelial-mesenchymal transition.Chlamydia trachomatis Infection Is Associated with E-Cadherin Promoter Methylation, Downregulation of E-Cadherin Expression, and Increased Expression of Fibronectin and α-SMA-Implications for Epithelial-Mesenchymal Transition.Mechanisms of the epithelial-mesenchymal transition by TGF-beta Tudor Domain Containing Protein 3 Promotes Tumorigenesis and Invasive Capacity of Breast Cancer Cells Involvement of members of the cadherin superfamily in cancer Whole DNA methylome profiling in lung cancer cells before and after epithelial-to-mesenchymal transition.Interference with Sin3 function induces epigenetic reprogramming and differentiation in breast cancer cells.RIN1 is a breast tumor suppressor gene.Disseminated breast cancer cells acquire a highly malignant and aggressive metastatic phenotype during metastatic latency in the bone.Intracellular modifiers of integrin alpha 6p production in aggressive prostate and breast cancer cell lines Reduced expression of miR-200 family members contributes to antiestrogen resistance in LY2 human breast cancer cells Genome-wide epigenetic modifications in cancer.Estrogen-related receptor gamma promotes mesenchymal-to-epithelial transition and suppresses breast tumor growth EMT and stem cell-like properties associated with miR-205 and miR-200 epigenetic silencing are early manifestations during carcinogen-induced transformation of human lung epithelial cells.The high mobility group A2 protein epigenetically silences the Cdh1 gene during epithelial-to-mesenchymal transition Is upregulation of BCL2 a determinant of tumor development driven by inactivation of CDH1/E-cadherin?Genes suppressed by DNA methylation in non-small cell lung cancer reveal the epigenetics of epithelial-mesenchymal transition Methylated genes in breast cancer: associations with clinical and histopathological features in a familial breast cancer cohort.

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P2860

E-cadherin transcriptional downregulation by promoter methylation but not mutation is related to epithelial-to-mesenchymal transition in breast cancer cell lines.

http://www.wikidata.org/entity/Q34497064

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2006 nî lūn-bûn

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A-M Cleton-Jansen

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B van de Water

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C J Cornelisse

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J W F Dierssen

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M Lombaerts

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P H Eilers

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R M E Zimmerman

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R van Eijk

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P2860

The use of cystatin C to inhibit epithelial-mesenchymal transition and morphological transformation stimulated by transforming growth factor-beta.

The SLUG zinc-finger protein represses E-cadherin in breast cancer

E-cadherin gene mutations provide clues to diffuse type gastric carcinomas

Accurate normalization of real-time quantitative RT-PCR data by geometric averaging of multiple internal control genes

E-cadherin is a tumour/invasion suppressor gene mutated in human lobular breast cancers

Methylation-specific PCR: a novel PCR assay for methylation status of CpG islands

Cluster analysis and display of genome-wide expression patterns

Matriptase and HAI-1 are expressed by normal and malignant epithelial cells in vitro and in vivo

Expression analysis of candidate breast tumour suppressor genes on chromosome 16q

Epithelial-mesenchymal transitions in tumour progression

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